Antiarrhythmic Drugs

Front 1

Drugs used in Arrhythmia

Back 1

1. Class 1a=Na+ channel blockers that prolong AP
2. Class 1b=Na+ channel blockers that shorten AP
3. Class 1c=Na+ channel blocks w/no effect on AP
4. Class 2=B-blockers
5. Class 3=K+ channel blockers
6. Class 4=Ca2+ channel blockers
7. Adenosine
8. Magnesium
9. Potassium
10. Vasopressors

Front 2

Bradyarrhythmia Treatment

Back 2

1. Atropine
2. Dopamine
3. Sympathomimetics (any + ionotropic)

Front 3

Causes of Arrhythmias

Back 3

1. Abnormal Impulse Formation
a. Increased automaticity
b. Afterdepolarization
2. Abnormal Impulse Conduction
a. Reentry in ventricle
b. Reentry in AV node

Front 4

Type Ia Anti-arrhythmics

Back 4

Na+ channel blockers that prolong AP
1. Procainamide-oral, IM, IV
2. Disopyramide
3. Quinidine- oral IM

Front 5

Procainamide

Back 5

class 1a antiarrhythmic
sodium channel blocker
prolongs AP
Drug-induced Lupus Erythromatosis (just like Hydralazine)

Front 6

Disopyramide

Back 6

class 1a antiarrhythmic
sodium channel blocker
prolongs AP
Greatest tendency to produce CHF./AV Block

Front 7

Quinidine

Back 7

class 1a antiarrhythmic
sodium channel blocker
prolongs AP
Increases toxicity of Digoxin and Verapamil (i/a at level of kidney)
SE: tinnitis, blurred vision, N/V (think of how quinidine is like quinine, which is in tonic water...too many Gin and tonic---gives you these symptoms

Front 8

SE of all Ia anti-arrhythmic drugs

Back 8

1. Ventricular tachycardia
2. AV block
3. Decreased contractility
4. DECREASED BP

Front 9

Use of all Ia anti-arrhythmic drugs

Back 9

1. Prophylaxis of SVT arrhythmias
2. Treat/Prophylaxis of Ventricular Arrythmia