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181 Cards in this Set

  • Front
  • Back
Ontogeny
Growth and development reflects the series of changes by which an individual embryo becomes a mature organism

-change in siz shape and structure
-addition loss and substitution of materials
-includes effects of aging and senescence
Human Auxology
the study of human physical growth and development
Greulich and Pyle Hand Againg
forensic anthro, way to chart age by the bone growth and fusion in the hand
Comparative Method
Using other living species to illuminate fuction (think bio 212)
Body Fat peaks at 6 months
parents begin weening the child, solitude is more present, babies starts using the fat storage to develop brain
Secular Trend
change over generational time
stature
increase in nearly all human populations in last century
Developmental Plasticity is pronounced in humans
nutrition, exposure to infectious disease, physical environment, social environment and child rearing practices
Medical Perspective on Human Growth and Development
individual children in clinical context
Social perspective
public health groups of children
Mesopotamia view (3500 years ago)
-variation in HGD recorded birth weight and defects
-links between hight, social status and health (positive correlation)
Egyptians, Greeks and Romans
lifestyle and HGD links -> diet, activity and education

goals to breed the most physically fit
Middle Ages
shift shown in art:
children were more like mini adults, given scaled down adult body proportions, given adult responsibilities early on
Renaissance
14th-16th Centuries
-> rediscovery of greek and roman approach
-roots of modern scientific approach, humans are part of nature and change with nature
-> Passionate pursuit of the scientific study of the human body
Leonardo Da Vinci
anatomical studies including accurate drawing of a fetus (vs. homunculus - "little man")
Andreas Vesalius (1514-1564)
flemish anatomist, On the workings of the human body
Bergmuller's Anthrometria (1723)
depicted childrens growth over life time span
Comparative Anatomy
Edward Tyson (1699): saw the similarity between chimp anatomy and human anatomy -> between species -> common ancestry
Studying Children Growth
Christian Jampert (1758) study boys height (looking at correlation between height and light?)
-Good Quality measurement procedures
- didn't see individual patterns of growth -> sampling issue
George LeClerc de Buffon
First longitudinal growth study, used his son Montbeillard from birth to 18years old measuring every 6 months

-> continuous gain in height, usually ends around 18-19, skeletal growth ends around 25

-> Seasonal Variation in height present (accelerated in the summer months, decelerated in the winter months)

-> Taller in the morning than in the afternoon (fluctuation)
height velocity
Looking at the height gained per year, distinct stages:
infancy(rapid)
childhood(steady)
adolescence (rapid)
approaching adulthood (very slow)
Karl Ernst von Baer
-Founder of embryology
described cellular fertilizationa nd embryonic development: endoderm, mesoderm, and ectoderm
-> Comparative Embryonology
recognizing the similarities in embryos of all vertebrates, shared ancestry
Auxiological Epidemiology
the study of the distribution of patterns of human growth in relation to environmental factors

monitoring groups of children rather than indvls in order to change social conditions through public health measures and political reform

-> pointed out the differences in conditions between classes (lower class had poor conditions meaning improper growth)

-housing, work and social conditions as variables
Franz Boas
"Father of American Anthropology"

-recognized variation in tempo of growth
-developed american standard of growth using 90,000 children 5-18
-incorporated other studies: critical of cross sectional data
- looked at adult height and timing of growth spurts -> no difference between early and late
- developmental plasticity (see card)
Longitudinal
Repeated measurements of same individuals at different ages
Cross Sectional
Single set of measurements taken once on subjects of different ages
Francis Galton
"Eugenics": attempted improvement of the human species through controlled selective breeding

American Eugenics Movement -> Sterilization of those not deemed fit to reproduce

racist
Samuel Morton
thought white caucasian males have larger head and are therefor smarter

corrupt, flawed data
Migration Study Design
european born immigrants vs american born children
-measurements focus on environment
-critique on race concept but still US anti immigration movement
Margaret Mead
Study development of sexual identity and adolescent behavior among Somoans.

-> sexuality as purely culturally constructed
Developmental Plasticity (Boas)
recognized secular trend, change over generational time as results of proximate environmental factors ignoring genetic changes

Secular trends can be positive or negative
World Bank: Economics and HGD
height as a proxy for well being, criterion for economic need
Robert Fogel
Uses data from African American slave children and shows the impact on adverse social conditions and health
Phylogeny
the evolutionary history of a species of higher taxonomic group
Ontogeny
ernst haeckel,
each successive developmental stage represents one of the adult forms that appeared in evolutionary history
Stephen Jay Gould
Ontogeny as an evolutionary trait acted on by natural selection

importance of heterochrony in evolutionary change
Heterochrony
Neoteny: slowdown of the developmental rates resulting in retention of juvenile stage into adult stage (Mickey Mouse, Betty Boop -> juvinile is more desireable)

Hypermorhposis: prolongation of the time of development of a feature in a descendant so that he descendant has features that are hypermature compared to the ancestor
Mammals
mammals have expensive lives, need lots of energy and consumption

can inhabit many different environment types, flexible
Dietary Adaptations
heterodont detntion

must meet relatively high continuous energy demands
Locomotor Adaptations
more dependance on an active life

epipheseal fusion

bone romodeling
Unique Reproductive Biology
high reproductive costs: placenta maternal nourishment, lactation high quality nutrition to poorly developed toothless infant

-> increased mother infant connection and bonding interaction important relationship
= social creatures
Sexual Dimorphism
typically divergence in male and female anatomy, physiology and behavior
Z-Score
Indicates how many standard deviations a value is above or below the mean
Stunting
Assessed by height for age index

does not indicate past nutrition or health risks in the future
Wasting
Assessed by weight for height index

indicates present nutritional status and is associated with higher risk of disease and death
Hormonal Regulation (Ovarian)
Menstral Phase (5days), Follicular Phase(9days), Luteal Phase (13 days)
Ovulation
2 weeks after last period, growth of follicles containing oocyte
Capacitation
conditioning of the sperm, altered sperm Cap
LOOK AT BIO 212 FERTILIZATION NOTES
LOOK AT BIO 212 FERTILIZATION NOTES
Monozygotic Twins
identical twins, ovum divides into two identical zygotes, random, always same sex and genetically identical

not hereditary
Dizygotic Twins
Fraternal twins, two oocytes are fertilized by two different sperm, genetically no more similar than other siblings, can be same or different

Can be hereditary, determined by maternal lineage
Conceptus
Zygote->Morula-> Blastocyst

weeks 0-2
Embryo
weeks 3-8
formation of body plan and major organ systems
Fetal
9-38 weeks
resembles human
growth in size
post fetal
growth
Trimesters
weeks 1-12
weeks 13-27
weeks 28-40

variation of menstruation to fertilization
Trophoblast
outer layer of blastocyst, produces hCG, Human Chorionic Gonadotropin

prevents menstration during pregnancy
Corpus Luteum
Temporary endocrine structure derived from follicle
hCG
produced by the fetus, low = something wrong->miscarriage

-tests viability of embryo
Birth Weight
trade off between maternal conditiona nd offspring size
-> lower would reduce probability of offspring servival, increased maternal condition
-> higher birth weight, increased probability of offspring survival, increased adverse maternal conditions, higher IQ in heavier twins tho
NVP
protects mothers and developming embryos from dietary mutagens and pathogens that could cause malformation or spontaneous abortion
1st trimester energetics
minimal demand, weight gain occurs in mother
2nd trimester energetics
growing fetus as more of a metabolic load
Nutritional Needs
more focus on energy instead of specifics
Fetal Fat Gain
1% by 24 weeks
11-16% by week 40
most energy in a late pregnancy goes to baby fat

up until 6 months after birth to 25% (PEAK)
Birth Defects/Congenital Malformations
most suseptable during first trimester (environmental factors)
Teratogen
agent capable of disrupting normal development of an embryo or fetus: pregancy complications, miscarriage or birth defects

timing is biggest effect
Psychosocial Stress
decreased conception rate
-increased risk of miscarriage
Anencephaly (no head)
failure for the head end of the neural tube to close.

->fetus who lacks a major portion of the brain, forehead, scalp and skull
(occurs between 23-26th day)
Spina Bifida
Condition where neural tube does not completely form aroudn the spinal cord. Mild to Severe and can be associated with loss of bladder and bowel function, paralysis and sometimes death.
Cleft Lip/Palate
occurs early in fetal development, gap in the tissues of the mouth or lip, easily fixed with surgery

-if not fixed baby can starve and die
Club Foot
structural defect to the foot the top is where the bottom should be

easily fixed (developmental plasticity)
Fetus in Fetus
fetus within a fetus, one fetus is absorbed by its twin like a tumor
Bloody Show
sign that labor is hours or days away
Labor Stages
1st stage: Dilation and thinning of cervix, more frequent contractions

2nd Stage: Pushing and Birth Stage, up to 50 minutes, gets easier after first child, mother feels natural urge to push

3rd Stage: Expulsion of the placenta within 5-10 minutes of the birth
Parturition
Timing of Birth
-> used to be thought to be determined by mother
Postnatal Nutrition
every 100 calories a woman takes in 90 calories go to infant
Timing of Birth
The Obstetric Dilemma: evolutionary baggage, Karen Rosenburg, evolved to be bipedal defining hominids, creates different pelvis with smaller birth canal, secondary altriciality - born developmentally young
Obligate Midwifery
assisted childbirth required in humans

need health and social support
Potential "Solutions"
Smaller brain size of fetus at birth
-> shorter gestation period? CONSTRAINED
or
Larger maternal pelvis
-> already really big, less curved sacrum? more ounded birth canal?
Apgar Score
test of newborns physical conditionat 1 min and 5 min to assess need for medical intervention
preterm delivery
under 37 weeks
low birth weight
anything under 2500 grams at birth
Cranial Deformation
malformation of the skull that results from intentional or unintentional pressures applied to infant skull
Synaptogenesis
dendritic growth in neurons after birth (no more are made)
Synaptic Pruning
rapid pruning of electrically inactive synapses (at around 1-2 years old)

use it or lose it
Myelination
coating the nerve fibers with a fatty substance (myelin) that seres as an electrical insulator
Colostrum
breasts produce yellowish liquid during the first few days after birth: high in protein and low in fat and sugar, lots of immune value, helps establish bacterial gut flora and growth in gut maturation
Kwashiorkor
inadequate intake of protein that often occurs at weaning its fluid swelling of the stomach
Marasmus
severe malnutrition resulting from diet in all essential nutrients
Juvenile
age 7-10 or 12 puberty
independent cognitively advanced enough to survive on their own, can make logical inferences and increased ability to recognize cause and effect relationships and less egocentric

Concrete Operational stage: less able to deal with hypothetical tasks

lots of social learning
Juvenile Growth
stable, slowest rate of growth since birth

stable and predictable growth
Adolescence
growth rate declines
sexual maturity
secondary sexual characteristics
-transition from independent subadult to fully functional adult

Menarche in females begins
Puberty
"to grow hairy" in latin

onset of adult reproductive capacity

Gonadarche
Gonadarche
reactivation of the hypothalamic-pituitary-gonadal axis which regulates sexual development and function -> regs sex hormones
Puberty Initiation
activation of hypothalamus: releases Gonadotropin - Releasing - Hormone (GnRH) its the growth hormone so it is always being released but more frequently at the stage and stimulates pituitary gland to release hormones as well
HPT Axis
Testicular: steroid hormone produced by testes: Testosterone

Ovarian: steroid hormones produced by ovaries (Estradiol & Progesterone)-> produce oocyte
Estradiol
Endometrial proliferation and secondary sexual characteristics also pelvic deveolpment and boobs

(Progesterone maintains endometrium)
Puberty in Females
pubic hair
AGS
menarche when done growing: resources that produce growth can now focus on being reproductively effective

about 12 or 13 in US and Euro populations, variability internationally by life resources
Adolescent Growth Spurt (AGS)
dramatic growth of most body measures
long bone lenght
vertebrae
facial bones change
heart & lungs expand

rate is asymmetrical: slow ascent and rapid halt
Brain Development
Size doesnt change but content does

neuron to neuron processing is increased: better memory ability and concentration, better self control
Emergence of Sex Difference
body fat: women increase adiposity at 8 yeasr old and then reach a peak during puberty, men have late weight gain
Tempo of Growth
mostly controlled by genes and then environment flexibility

same with speed of growth
Frisch
Fat accumulation hypothesis
women must have a minimum % of body fat for menstration to begin -> actually has to do with HPG axis maturation
Tanner Ellison skeletal maturaity hypoth
appropriate age of skeletal deveolopment to reproduce

close correlation between menarche and attainment of adult pelvic dimension

coordination to ensure successful reproduction

weight accounts for 18% height 50% of variance in age of menarche

pelvic size doesnt reach adult until 17-18

birthing issues increase <17 years old
Subfecundity
unable to get pregnant, "sterility"

55-85% of menstrual cycles are ANOVULATORY for first two years
Chromosomes
Autosomes & 23 Sex Chromosomes
Turner Syndrome
Monosomy, only an X chromosome, no ovaries function, webbed neck, drooping lids, broad flat chest

concurrent health problems: heart disease, hypothyroidism, diabetes, vision problems

1/3000 births

treats GH to increase height
Klinfelter Syndrome
Trisomy, extra X chormosome
XXY: disjunction in the sperm or egg with two xs and a y

1/650 males
males grow breasts, small testes, infertile, treatments testosterone to grow body hair and improve muscle appearance
Edward Syndrome
Trisomy: third copy of chromosome 18, affects all biological systems and causes distinct facial features: dont survive long, clenched fist, seizures, small head/eyes, heart abnormalities kidney malfomations and other organ disorders

1/3000 live births, most die before birth.
Down Syndrome
Trisomy (autosome) extra copy of chromosome 21
impaired physical and cognitive development

small chin round face protruding oversized tongue poor muscle tone cognitive impairment neotenization of the brain and body to fetal state

mothers over age 45 1/19 risk fathers over 42 increased risk

1/733 births
Prader Willi Syndrome
missing portion of chrom 15 from father that results in voracious appetite and lack of full snesation after eating. -> Obesity

often when born SGA, kicks in around adolescence

decreased muscle tone, failure to grow impaired mental capacity and gonad disfunction: little to no sex hormones

slower metabolism, chronically hungry

Growth Therapy: HGH and Behavioral Therapy: teaching them how to eat
Tay Sachs
rare terminal heritable disease of nervous system

mutation on chrom 15, body lacks a protein necessary to break down a chemical in nerve tissue called gangliosides (build nerve cells)

symptoms appear at 6 months

infant becomes blind, deaf, unable to swallow,

life expectancy 4 years old

most common among Ashkenazi Jews (some Cajuns too) -> Founder Effect Hypoth
Founder Effect
Few indiciduals from a larger pop establish a new pop increasing frequency of certain genes and mutation in the new group
Proteus Syndrome
"Elephant Man Condition"
mutation on chrom 16

overgrowth of skin bones muscled fatty tissues and blood and lymphatic vessels

Cells divide and expand unchecked resulting in benine tumorous growths throughout body-> Severe disfiguring

average or above average intelligence

Primordial Dwarfism
rare genetic mutation: born SGA and have intrauterine growth retardation (IUGR) <3lbs at birth

disruption in utero, lack of O2

growth stays slow after birth, <2.5 feet tall

head growth is even slower than body "microencephaly"

high voices, prominent nose and eyes, scoliosis
Bone Growth Disorders
organic protein collagen & inorganic mineral calcium phosphate

changes in response to activity, growth, lifestyle
Osteoblasts
cells respnsible for bone formation
Osteoclasts
cells responsible for bone removal
Osteogenesis Imperfecta
literally imperfect formation of bone, brittle bone syndrome- glass man in unbreakable, lack of collagen strengthening the bone(cement of bone)

-> Blue eyes in the white part
Achondroplasia Dwarfism
literally: without cartilage formation

genetic condition effects long bone growth and nasal bridge

sporadic mutation in DNA

avg 4ft2in height and large head megalencephaly, sleep apnea from small nose

1/15,000-40,000 birhts

associated with fathers age
Osteopetrosis
bones like rocks

disorder in osteoclasts that results in denser bones

abnormally dense skull impinges on cranial nerves: vision hearing loss and facial paralysis

long bones continue to grow and grow into marrow cavity where white blood cells are stored-> children die from infections
Fibrodysplasia Ossificans Progressiva (FOP)
rare mutation affecting body's ability to repair tissue when damaged

bruise and sprains cause new bones are formed instead of muscles tendons and ligaments

overproduction of osteoblasts

no way to treat this because anything will cause bone growth

mutation
Endocrine Disorders
3 types: Hyposecretion, Hypersecretion, tumors of endocrine glands:

Glands: pineal, thymus, adrenal, gonads, pancreas, thyroid, pituitary
pineal
melatonin prod reg wake and sleep
thymus
immune system
adrenal
stress hormones cotisol and androgens
gonads
sex hormones
thyroid
metabolism, energy
pituitary
master gland, regulates secretion of hormones throughout the body involved in controlling various somatic processes
Precocious Pubery
development of secondary sex characteristics before age 8

genetic problem or tumors

stop growing earlier, shorter

psychosocial issues

girl could conceive earlier than she should, youngest mother 5 years old, started menstrual cycles at 8months

later normal function

treatment inhibit release of sex hormones or tumor
Growth Hormone Deficiency (GH)
dwarfism

hypopituitary disorder, can come from lack of O2 in utero, genetic mutation, pituitary gland disease, brain and liver tumors

look young for whole life, adult in child body

normal life span

Gary Coleman, Andy Milonakis

slower muscle devel, moderate chubiness, cherubic facial features, masillary hypoplasia, forehead prominence

GH injections

women birth Csection
Growth Hormone Excess
gigantism, acromegaly(mutation, tumor pit gland)

andre the giant

over production of GH before epiphyses closes or after they close

gap in between teeth, jaw outgrows teeth,
Cushing Syndrome
overproduction of cortisol by adrenal gland, TUMORS in pit gland or adrenal

cortisol stimulates glucose production, fat pronounced

fat, excess hair growth, acne, easy bruising, high blood pressure

common response to corticosteroid medication (Prednisone)

treatin asthma and rheumatoid arthritis
Thyroid Gland
largest endocrine gland
regs metabolism, sensitivity to other hormones

Thyroid uses iodine to make thyroid hormone
Hypothyroidism
thyroid gland fails to make enough thyroid hormone, slow down metabolism, weight gain, sensitivity to cold.

mother has under secretion: Cretinism in infants: severe physical and developmental stunting
Hyperthyroidism
Goiters in both hyper and hypo

thyroid gland overproduces thyroid hormone

swelling of eyes

too much iodine

major weight loss, anxiety, hair loss, intolerance to heat, fatigue, weakness, muscle aches, intellectual disability

infants: lack of appetite, jaundice and stunted growth
Assessing Deveolpomental Disruption in the Skeletal Record
Skeletal markers in disruption of growth

Enamel

Harris Lines

Cribra Orbitalia and Porotic Hyperostosis
Skeletal markers in disruption of growth
length of long bones in relation to population,

cortical bone maintenance: patterns of growth, thicknes, cross section, nutitional status and activity patterns, look at deficiencies, malnutrition
Linear Enamel Hypoplasias (LEH)
lines, pits or grooves on the enamel indicating a severe disruption during development

proxy for health in living pops and skeletal pops: economic development

# of lines and size of the line indicate quantity and duration of insult, respectively
Harris Lines
estimating based on growth lines on long bones

cross sections of bones
Cribra Orbitalia and Porotic Hyperostosis
indicates iron deficiency anemia

iron binds o2 to red blood cells, marrow cavities expand, makes bones thinner, porous like consistency

high pathogen exposure

nutritional stress
Iron Deficiency Anemia
>50% of women and children in developing countries, females more susceptible: PREGNANCY

infants over 6months if weaning foods are inadequate in iron

Maize Agriculture: corn, weat, inhibit ability to absorb iron

Iron: meat, beans, spinach

low birth weight, fetal death decreased resistance to infections
What drives neuron migration and how do the neurons know where to go?
Neurons migrate along glial cells, or supporting cells. Young neurons have an idea about where they are
migrating to and can recognize their future position in the brain. By the time a neuron begins its
migration it has already received instructions about what to become. These are genetically
predetermined instructions about what its position will be
How does brain wiring occur? Why is the period of infancy and early childhood often called
the “exuberant” period of brain development
Early in fetal development, neurons reach out to each other and connect via synapses. This process of
brain wiring follows a specified genetic blueprint which ensures that connections are made between
appropriate neurons and that these connections are working properly. Billions of connections are made
during infancy and early childhood and this overproduction of connections is referred to as the
“exuberant” period of brain development. If connections are being used, they will be maintained. If they
are not used, those connections are lost
How does premature birth affect brain development and cognitive functioning
The brain is faced with more challenges outside the womb than inside because the bodily functions of
the neonate (e.g., digestion, respiration) are no longer performed by the mother. Furthermore, the
brain, which regulates these critical functions, is not as developed to the extent that it should be. As a
result, premature babies will be hypersensitive and more easily stimulated. When they grow up, these
infants will have trouble planning, prioritizing, paying attention, and imposing structure.
According to the video, what can be done for premature babies to help minimize future
cognitive problems?
One way to help minimize cognitive problems is to reduce the noise of the intensive care unit and create
a carefully controlled environment tuned to the needs of the premature baby; this will help the baby
better manage the world around them as they grow. This controlled environment would emulate the
womb, be quiet and dark, and the mother would be encouraged to have as much skin-to-skin contact
with the baby. This is the opposite of treatment most premature babies have in a standard hospital
setting
What do Dr. Mriganka Sur’s studies of the ferret tell us about brain plasticity and the
interaction of “nature” and “nurture” in brain development?
In a series of experiments, Dr. Sur rewired the brains of newborn ferrets and hooked up the visual
cortex to the auditory cortex. Sur’s study posed the question: If the visual inputs of the eye are
redirected to the auditory cortex, would the hearing cortex become like a visual cortex? If the hearing
cortex does not become like the visual cortex, then this suggests that “nature,” or the genetic
endowment, is important to brain development. That is, brain wiring is genetically predisposed and there is little that can change the connectivity established in fetal life. If the hearing cortex does become
like the visual cortex, then the external inputs, or “nurture” components, are important; the animal,
then, would see from its auditory cortex. Sur’s studies demonstrate hat environment shapes the brain
but can’t completely change genetic destiny. Genes do impose some structure on how the brain
develops but these are further influenced by the environment that the brain is developing in.
How do structural problems in the eye (e.g., cataracts) in newborns affect brain development?
Are there permanent consequences if these defects are repaired?
If a baby has cataracts, the visual images that are important for the brain to develop its vision are not
entering the eye. The brain will not get the opportunity to experience vision and go through its normal
development. Because the infant’s brain is still being wired, limited visual stimulus will mean that the
baby could lose that connection (i.e., use it or lose it). Even one or two months of missing visual stimulus
can have permanent effects on how the brain is wired. With cataracts, the good eye must be covered to
strengthen the weak eye for a number of years following surgery. However, visually, even within a short
period after surgery, the baby can see as well as a normal newborn. Vision improves quickly in babies
who have had cataracts removed.
What factor determines the sex of a baby?
Determined by the father, 23rd pair of chromosome → Mother only gives a type X but Father can give an X or a Y
Which organ is the first to form during embryological development? What is the function of this organ in the embryo?
The Heart, set the beat of all the cells to contract and relax, pumping the blood and delivering food and oxygen to all the cells
At what point during development does the embryo begin to resemble a human? Why does this period represent a milestone for the fetus?
8 weeks, embryo is now known as a human. Until now the embryo was dependant on the nutrients from the yoke sack, by 2 months the yoke sack goes away and the placenta begins to provide food and nutrients and filters waste, connected by the umbilical chord. Fetus life support, takes stuff from the mothers blood stream
-placenta also produces hormones (progesterone: stops production of more eggs) some cause morning sickness and an adverse to potentially harmful foods
What information regarding fetal growth can an obstetrician gain from 4-D scans?
They can look at how the fetus grows, behaves and reacts to stimulus. Looks at the fetus in real time, helpful for checking health of baby and seeing if there are multiple babies
What is the significance of the stepping reflex in the growing fetus?
Start after just 11 weeks in the womb, babies kicking and pushing out legs, reflex actions/biological impulse, nervous system is working, Fetus can bounce and leap around like in a trampoline, the more this happens the more the baby develops walking abilities→ can be seen in newborns when legs are dangling
Why are miscarriages most likely to occur during the first three months of fetal development?
New cells developing imbalance of hormones stress or previously given births to a boy
What is meant by proprioception and when is this ability developed?
Unconscious sense of bodies and space that helps the baby understand its place in environment
What developmental landmarks can be expected in a 6 month-old fetus?
Senses organs begin to function: now can respond to touch and smell and sight and sound and taste causing the brain to have to learn to interpret these sensations
What is a 3-month old fetus’ most developed sense?
Hearing
In addition to swallowing, describe another reflex that is critical for survival after birth and has been observed in utero.
Startled reflex to louder sounds and stimuli, adaptive to when we were hunted?
How can a mother’s emotional state influence the physiology of her gestating infant?
If the mother is calm her blood pressure goes down and heart rate slows, passes through the placenta to the baby, mother is in recovery from stress while baby is feeling the effects -> Can lead to low birth rate or long term can be passed on and cause health problems like heart disease and mental issues later on in life
According to the film, what determines the timing of birth? What is the function of oxytocin during delivery?
Initiates the contractions of the uterine wall. Also helps the mom forget the pain of labor and bond with baby
What is the function of adrenaline during the birthing process?
To keep the babies heart pumping fast enough, also helps prepare the lungs for the lifetime of work
Adulthood
attainment of full reproductive maturity, bio and social demensions
Elderly
Over 65 years old

fastest segment of the population of the US and most developed nations

decreased childhood mortality
Scenescence
biological decline in function, leads to reduced ability to deal with environmental stressors

Decreased ability to maintain homeostasis in the face of environmental challenges

age is based on the passage of time

must be: cumulative, universal, progressive, intrinsic, deleterious
Life Span
average length of life of a particular type of organism in a particular environment under specified circumstances

-women live longer than men, men are more susceptable to disease because of increased testosterone levels
Life Expectancy at Birth
measure of the average life span of a population or group

expected (mean) survival based
Facial WRinklings
decline sin collagen, skin becomes thinner, paperlike
Male Pattern Baldness
result of thinner diameter of hair and distribution, strong genetic control
Weight Gain (Middle Age Spread)
age related declines in BMR changes in activity patterns, hormone changes
Reduced Stature
due to compression of vertebral column and reduction in bone quality and quantity measured as bone mineral density

could result in KYPHOSIS or DOWAGER's hump
Osteoporosis
Low bone mineral density (BMD) reduces integrity of bone and increasing risk of fractures

Osteopenia low BMD

Osteoporosis EXTREMELY low BMD

Femals have greater risk: cessation of ovarian function at menopause stops production of estrogen which is th ekey hormone in maintainging bone integrity

physical activity prevents
Menopause
permanent cessation of menstruation and monthly cycling, after 12 months = post menopausal

around age 50

only species that have menopause

men dont really have it, just a social menopause

general loss of sex drive
Rural Mayans in Yucatan (Beyene, 1986)
Menopause in Yucatan

women with numerous pregnancies extended lactational amenorrhea, malnutrition reported no hot flashes or sweats
Japanese Women (Lock 1998)
less likely to report experiencing menopausal symptoms, related to dietary and activity differences, cultural differencces with US and european women in how menopause is viewed

"KEONKI"
Adaptation or Epiphenomenon
why do women live after menopause?

prolonged offspring dependency after?

Grandmothers? Kin Selection, Inclusive Fitness
Andropause
Male Menopause, male reproductive output does not stay constant throughout life even tho they can still produce sperm

-> Low Libedo
-> decline in stamina
-> Declines in spermogenesis
Antagonistic Pleiotropy Disposable Stoma
Genes that have good effects early in life... see lecture slides and ADDD!!!!
Adaptation
changes by which organisms surmount challenges to life
Bergmann's Rule
body size within a species larger at lower temps

mass to surface area

higher body masss= lower SA and greater heat retention

lower body mass= less heat retention
Allen's Rule
appendages (arms and legs & ears) shape and body mass to sa

longer = less heat retention
shorter = greater heat retention