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35 Cards in this Set

  • Front
  • Back
Angina
Imbalance between oxygen supply and oxygen demand
Normal coronary arteries
supply the myocardium with blood to meet metabolic needs
Dilate when the myocardium needs more blood
CAD: coronary arteries occluded with atherosclerosis
can't supply the myocardium with blood for normal workload
Usually 75% occlusion before angina results or symptoms are noticed
Other causes of angina
Coronary artery spasm
Coronary arteritis
Hypotension
-spinal anesthesia
-antihypertensives
-blood loss
Aortic valve stenosis
Blood disorders
-anemia, polycythemia
Ischemia
1. exercise or stress
2. increased oxygen demand
3. coronary arteries can't accomodate (can't dilate)
4. Anaerobic metabolism
5. formation of lactic acid
6. pain occurs
Anaerobic metabolism causes lactic acid formation
Lactic acid irritates myocardial fibers
Pain transmitted cardiac nerve and upper thoracic posterior nerve roots
Pain can be referred
Pain called ANGINA
Symptoms of Angina
Not felt the same way by everyone
-tightening, squeezing
-indigestion
Women mostly feel anxious
Any kind of pain from the umbilicus up could be MI
Symptoms include
Tightening, pressure, squeezing, or aching sensation in the chest, arms, back, shoulders, neck, or jaw
Indigestion
An ache that begins or spreads to the neck, jaw, throat, shoulder, back or arms
Associated symptoms include:
SOB
diaphoresis
weakness
dizziness
nausea
vomiting uneasiness (anxiety)
Precipitating factors:
exertion (increased oxygen demand & workload on heart, increased HR)
Emotions (release of catecholamines
Heavy meal
Hot or cold environment
Onset
Sudden pain
Duration (Stable Angina)
3-5 minutes
Rarely longer than 20 minutes
Relieved by rest or nitrates
SL nitroglycerine (vasodilates) coronary artery, BP can plummet, they can get a HA
Intensity (Stable Angina)
Rated on a scale from 1-10
Usually less than 5
Varies by individual
If angina persists, an MI, or damage to the heart muscle can occur
Any pain >20 minutes need to call 911 and get to the hospital
Types of Angina
Stable (very predictable)
Unstable
Variant
Stable Angina (Pain is always consistent)
Syndrome of at least several weeks duration provoked by predictable activities that increase oxygen demands
should not be increasing in severity
Should be relieved by rest, NTG, or oxygen
Management of Stable Angina
Precipitating factors
Education
Medication
PTCI (cardiac cath for extent of CAD)
Unstable Angina (Pain is worse, comes at rest, need to go right to the hospital to be stabilized)
Placque formation may be starting to rupture, chest pain persists
Recurring progressive episodes of angina pectoris lasting longer than 15 minutes and poorly relieved by rest or NTG
Deteriorating chronic angina
ST or T wave changes associated with ischemia
No change in cardiac enzymes or EKG evidence of acute MI
Management for Unstable Angina
Need immediate hospitalization
Stabilize
PTCI (Cardiac cath)
Medication
Stable vs Unstable Angina
Characteristics
Duration
Precipitating event
severity
relief
management
Variant or Prinzmetal's Angina
Coronary arteries spasm
Occurs at rest
Elevated STs
Can be associated with cocaine
Usually happens in women with small coronary arteries
Hx of migraines or raynauds disease is more susceptible to prinzmetal's angina
Treated with Calcium Channel Blockers
A note about women: Anterior MI
Women under 50 are twice as likely to die after an AMI than men in the same age group
Women are 55% less likely than men to participate in cardiac rehab
Women present with significant fatigue, anxiety. Thought they were having an anxiety attack.
Management of Variant Angina
NTG
Ca Channel Blockers
Vasodilate bring more blood to myocardium
Snapshot of the EKG
the EKG wave that reflects the movement of electricity through the myocardium
Myocardial tissue that is ischemic or injured will transmit the electricity differently
What can you see on an EKG? (12 lead)
P: begins with the firing of the SA node
-represents depolarization of the fibers in the atria
QRS: represents depolarization from the AV node through the ventricles
What can you see on an EKG? (12 lead)
PR Interval: Delay of transmission through the AV node, accounts for the time sequence from P to QRS
Width of the QRS represents time it takes ventricles to depolarize
T: represents repolarization of the ventricles
Ischemia and ST Segment Depression
Ischemia causes a change in repolarization (T wave area)
You will see changes in the ST segment and T-wave if the myocardium is ischemic
ST Segment depression
EKG changes: ST depression, flipped T's
Ischemic
ST elevation
Injury
Angina Case Study
30-year old man with a hx of smoking, alcohol abuse, and cocaine use
Presents with frequent episodes of chest pain, shortness of breath and diaphoresis while at rest
Episodes of chest pain usually awaken him early in the morning and lasted a few minutes
Tox screens negative for alcohol and controlled substances at time of admission
Prinzmetal's Angina
Cardiac cath revealed hyperventilation-induced total occlusion of the proximal left circumflex artery
Resolved with administration of intracoronary nitroglycerine and Diltiazem
Symptoms controlled with oral nitrates and calcium-channel blockade during follow-up of two years
Goals of therapy
Increase coronary artery blood supply
Reduce demands on the heart
Relief of pain
Modify risk factors
Health promotion activities
Take medications as prescribed
Know what activities bring on angina and modify or avoid them
Dress warmly in cold weather, avoid physical exertion in the cold air
Allow plenty of time to accomplish a task
If you become tired, sleep or rest
Avoid eating large meals
Dietary changes
Health promotion activities
Limit caffeine
Avoid situations that trigger angina (outbursts of temper or excitement)
Stop smoking
Control diabetes
Health maintenance
-lose weight, get enough sleep, balanced diet, regular exercise, avoid exposure to infectious diseases
Perform activities at a moderate pace
Viagra (Sildenafil)
Enhances effects of nitric oxide released during sexual stimulation
-enhanced blood flow to the corpus cavernosum and erection
Contraindicated with concurrent nitrate therapy: risk for serious and potentially fatal hypotension
Caution with serious underlying cardiovascular disease
Nursing diagnosis with interventions
Pain r/t myocardial ischemia
Activity intolerance r/t development of chest pain with exertion
Knowledge deficit r/t disease process, medications, reduction of risk factors, lifestyle changes, symptoms to report