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143 Cards in this Set

  • Front
  • Back

Sternum

3 parts: manubrium, body, xiphoid process

Suprasternal notch

Ridged top of manubrium


Most superior

Manubriosternal angle

Also called sternal angle of Angle of Louis


Bony ridge that is the articulation of manubrium and body of sternum


Continuous with 2nd rib


Site of tracheal bifurcation


Corresponds with upper border of atria

ICS

Intercostal spaces


Numbered by rib above

Ribs

12 pairs


Costochondral junction: where rib attaches to cartilage

Costochondritis

Inflammed costochondral junction

Floating ribs

11 and 12


Attached to spinal column only

Costal angle

Normally equal to or less than 90 degrees


Abnormal: angle increases or flattens with hyperinflation of lungs, ie. emphysema

Verterbral Prominens

C7 - palpated when head is flexed


If there are 2 bumps, it is C7 and T1

Thoracic vertebrae

12


Spinous process - knobs on vertebrae, some palpable

Scapula

Lower tip/inferior border at 7-8th rib


May be difficult to palpate r/t muscle mass

Reference lines

Verticle lines used to document findings

Anterior reference lines

Midsternal


Midclavicular (MCL)

Posterior reference lines

Vertebral (midspinal)


Scapular

Lateral reference lines

Anterior axillary line (AAL): anterior axillary fold


Midaxillary line (MAL): midway between AAL/PAL


Posterior axillary line (PAL): at post axillary fold

Supraclavicular

Above clavical

Intraclavicular

Below clavicle

Infrascapular

Below tip of scapula

Mediastinum

Cavity that holds:


Heart


Great vessels


Esophagus


Trachea

Pleaural Cavities

Contain lungs

Diaphragm

Floor of thorax


Major muscle of respiration

Lung Borders: Anterior

Apex: 3-4 cm above 1st rib, listen on top of clavicle


Base: Rest on diaphragm


- Right: at 5th ICS, MCS (liver pushes it up)


- Left: at 6th ICS, MCL

Lung borders: lateral

From apex of axilla to 7-8th ribs

Lung borders: posterior

C7 to T10 or T12 when inspiring


Upper lobes: T1-T3 or T4


Lower lobes: T3-T10 (expiration) or T12 (inspiration)

Lobes: right

3 lobes


RUL, RML, RLL


Shorter r/t liver


RML extends into axilla

Lobes: left

2 lobes


LUL, LLL


Narrower r/t heart border

Horizontal Fissure

Right side only


4th rib right sternal border to 5th rib MAL


Separates RUL and RML

Anterior oblique fissures

Bilateral


5th rib MAL to 6th rib MCL


Separates RML and RLL


Separates LUL and LLL

Pleura

Visceral: lines lung


Parietal: lines chest wall and diaphragm


Pleural cavity: negative pressure holds lung against chest wall with sucking pressure


Costodiaphragmatic recess: pleura exends 3 cm below level of lung

Costodiaphragmatic recess

Potential space for fluid/air collection that compresses lung


Inflammatory fluid collected may be infected

Bleb

Ruptured weakened alveoli


Atmospheric pressure (+) is introduced and leaked into pleural cavity (-)


Seen in young, male smokers

Anterior chest

Mostly upper and middle lobes

Posterior chest

Mostly lower lobes

Trachea

Anterior to esophagus


Starts at cricoid


10-11 cm long


Bifurcates at manubriosternal angle/T4

Bronchial tree

Right main stem bronchus is shorter and straighter, greater risk of aspiration


Lined with goblet cells and cilia

Dead space

Trachea and bronchi


Filled with air, but no gas exchange takes place

Goblet cells

Secretes mucus that entraps particles


Found in bronchial tree

Cilia

Sweeps particles in airways upward


Smoking paralyzes cilia and results in mucus pooling

Acinus

Functional respiratory unit


Bronchioles, alveolar ducts, alveolar sacs, and alveoli

Alveoli

300 million


Where gas exchange takes place

Mechanism of respiration

Supplies O2 and eliminates CO2


Helps maintain acid/base balance

Respiratory acidosis

Retained CO2

Respiratory alkalosis

Excessive excretion of CO2 through respirations

Respiratory center

Brain stem - pons and medulla


Increased CO2 is normal stiumuls to breath


Chonic hypoxia desensitizes CO2 receptors and low O2 begomces the trigger to breath


Delivery of high O2 concentrations = apnea

Ventilation

Air in and out

Perfusion

Diffusion of gases

Cough

Note timing: may indication cause


Continuous (resp infection)


Night, when recumbent (post nasal drip, sinusitis, GERD)


Morning, upon wakening (chronic bronchitis, smokers cough)


Specific settings (allergies)

Cough character

Hacking: mycoplasm pneumonia/walking pna


Dry/nonproductive: early CHF, allergies, meds (ACEI)


Barking: croup, whooping cough


Congested: bronchitis, PNA

Sputum

Amt, color, odor


Clear/white: viral bronchitis, PNA


Transluscent white/gray: noninfectious, chronic bronchitis, smoker


Green/yellow: bacterial bronchitis, PNA


Rust: pneumococcal PNA, blood and yellow sputum mixing


Pink, frothy: pulmonary edema


Hemoptysis: cancer, TB


Foul odor: bacterial

SOB and Dyspnea

Difficult, labored breathing

Orthopnea

Difficulty breathing when supine


Heart failure


Measure pillows used for sleeping or degree of incline

Paroxysmal nocturnal dyspnea

PND


Starts and stops suddenly


Awakens from sleep with SOB and resolves afer 10 min or so


r/t heart failure

Dyspnea on Exertion

DOE


Walking up incline

Allergies

dust, pollen, animals, mold

Mold allergies

seen anywhere there is moisture:


Water pipes, A/C, rain/dampness, plant mold


Plant mold blows in with Santa Anna winds from desert exacerbating allergies/asthma

Diaphoresis/Night sweats

TB, HIV, other infection

F/C/S

Infection

Unintentional weight loss

Cancer

Dependent edema, PND, orthopnea

Heart failure

Confusion, restlessness

Hypoxia

Pleurisy

Chest pain with breathing


Inflammation of pleura

Smoking history

Cigarettes, cigars, pipes, marijuana


Indicate never, past, current


Pack yr history, 2nd hand smoke

Family history


Allergies, asthma, TB (sick contacts), CF (genetic), lung cancer, emphysema (often r/t smoking or environmental exposure), antitripsin deficiency (genetic)

Environmental exposure

How often, do they wear a mask


Grain/pesticide inhalation (farmers)


Histoplasmosis (midwest)


Coccidioidomycosis (San Joaquin valley)


Pneumoconiosis (coal miners)


Silicosis (stone cutters, miners, potters)


Asbestos (plumbers, navy ships)

Histoplasmosis

Inhaled fungus


Primarily in midwest


r/t bird droppings or pigeon excrement

Coccidiodomycosis

Valley fever


Inhaled fungus


San Joaquin Valley


Causes cavitating lung lesions

Asbestos

Exposure and smoking increases lung cancer more than 10x


Old pipes were insulated with asbestos


Plumbers and old navy ships

PPD

Purified protein derivative


TB screening

Influenza immunization

Everyone > 50 yrs or at risk

Pneumococcal vaccine

Over 65 yrs or at risk

Children

Frequent URIs, 4-6/yr, lower immunities


Asthma: may be grown out of as bronchial tubes enlarge


Accidental aspiration: child-proof home

Aging Adult

SOB and fatigue with ADLs


Decreased vital capacity


Lung disease


Chest pain with breathing, rib fx r/t trauma, abuse, falls, severe coughing, osteoporosis

Vital capacity

Exhaled air after maximum inspiration


Measured by spirometry


Will decrease with bronchoconstriction, assesses the degree of bronchoconstriction

Inspect

Rate, rhythm, effort


Normal shape and symmetry


Spine: curvature may impair cardiopulmonary


Body position: professorial or tripod

AP:TV ration

1:2 to 5:7, increases with age


May be closer to 1:1 in chronic lung conditions, ie. barrel chested COPD

Scoliosis

Lateral curve


Entire spine affected


More common in adolescent girls


Assess uneven shoulders, scapulas, hips


Severe curvature (>45 degrees) may decrease lung volumes

Kyphosis

Hump back


T-Spine

Lordosis

Sway back


L-spine

Tripod position

Abnormal


Aids expiration


COPD

Palpate

Entire chest wall


Note tenderness, lumps, masses

Symmetric chest expansion

At level of T9 or T10


Uneven with atelectasis, pneumothorax, pleural effusion, phrenic nerve damage

Tactile fremitus

Vocal fremitus


Palpate vibrasions


Most prominent between scapulae and sternum, progressively decreases down thorax


Greater in thin people

Increased fremitus

Consolidation extending to lung surface


Lung tissue filled with fluid vs air


PNA

Decreased fremitus

Transmission of vibration blocked


Bronchial obstruction, pneumothorax, pleural effusion, COPD

Crepitus

Subcutaneous emphysema


Course crackling sensation


Air entering subcutaneous tissue via open thoracic injury, chest surgery, tracheostomy, CT placement

Percussed sounds

Percuss ICS


Resonant: normal


Hyperresonant: emphysema, pneumothorax, more hollow


Dull: increased density, atelectasis, PNA, pleural effusion

Diaphragmatic excursion

Non-routine, assess movement of diaphragm


Diaphragm should change 3-5 cm in inhalation vs exhalation


If no change, may be r/t paralysis, phrenic nerve damage, pleural effusion, atelectasis of lower lobes

CVA tenderness

Sign of kidney infection

Auscultation

Use diaphragm of stethoscope


Top to bottom and side to side

Bronchial sounds

Loud, harsh


Normally over neck, trachea, larynx


Abnormal if heard over peripheral lung field, indicates consolidation

Bronchovesicular

Moderately loud/harsh


Normally over midsternum and between scapula


Major bronchi

Vesicular

Low, soft


Normally over peripheral lung fields


If absent, report immediately


r/t mucus plug, collapsed lung

Adventitious sounds

Added sounds, not normally present


Crackles, rhonchi, wheezes

Crackles

Rales, sound like velcro opening


Produced when fluid inside bronchus causing a collapse of distal airways and alveoli.


Occur when there is sudden equalization of pressure causing some airways to pop open


Heard on inspiration doesn't clear with coughing


May be caused by atelectasis, PNA, fibrosis, heart failure, pulmonary edema

Fine crackles

High-pitched, short duration, cracking and popping sound

Coarse crackles

Low-pitched, longer duration, bubbling and gurgling sounds

Rhonchi

Deeper than crackles


Clears with coughing


Airflow through airway obstructed by thick secretions, spasm, or tumor - secretions pooling in bronchial tree


Loud, low, coarse sounds, like a snore or rumble


Heard continuously during expiration and inspiration


Bronchitis, decreased cough reflex

Wheeze

Airflow through constricted airway


High-pitched squeaking, like a whistle


Primarily heard on expiration, but may be on inspiration


Assess breath sounds with forced expiration in an asthma pt to check for bronchoconstriction


Bronchospasm r/t asthma, acute or chronic bronchitis

Stridor

Sign of respiratory distress


Partially obstructed airway


Characterized by inspiratory wheeze


Louder in neck than chest

Pleural friction rub

Caused by inflammation of pleural spaces


Coarse, rubbing or grating sound during inspiration or expiration that disappears when breath is held


Pleurisy

Voice sounds

Assess if breath sounds are abnormal

Bronchophony

Listen to "99" on back


Abnormal: clear "99"


Increased lung density, PNA, atelectasis

Egophony

Repeat "E" sound, listen on back


Abnormal: "E" changes to "A"


Consolidation

Whispered pectoriloquy

Whisper 1, 2, 3


Abnormal: sounds clear and distinct


Consolidation

Inspect anterior thorax

Skin: pallor, cyanosis


Nails: clubbing, r/t fibrotic lung changes


Pursed lips: obstructive disease


Spinting: shallow breaths to control pain


Quality of respiration: quiet, easy, nonlabored


Tracheal position: midline


Chest


Costal Angle: <90 degrees


LOC: drowsiness r/t cerebral hypoxia


Retraction or bulging ICS: unilateral vs bilateral


Accessory muscle use

Pursed lips

Seen in obstructive disease


Prolongs expiration cycle to allow for exhalation of trapped air

Tension pneumothorax

Trachea shifts, tracheal tug


To opposite side of lung collapse

Pectus excavatum

Sunken sternum, funnel chest


Can impair respiration

Pectus Carinatum

Forward protrusion


Pigeon chest

Barrel chest

Increased AP diameter


Associated with aging, emphysema, asthma


Costal angle > 90 degrees

Retraction of ICS

Obstruction or increased respiratory effort

Bulging of ICS

Trapped air, emphysema


Leads to barrel chest

Accessory muscle use

Neck muscles used to lift sternum and rib cage


SCM, Scaleni (below SCM), and trapezius


Neck muscles may be overdeveloped with chronic respiratory disease

Normal respiratory rate

10-20 breaths/min with occasional sigh


Sighs expand alveoli

Tachypnea

Rapid, shallow breathing


>20 breaths/min


Fever, fear, anxiety, exercise, respiratory insufficiency, PNA, alkalosis, pleurisy, lesions in the pons

Hyperventilation

Rapid, deep breathing


Extreme exertion, fear, anxiety, DKA


CO2 is excreted through respirations increasing the alkalinity of the blood

Bradypnea

Regular, slow breathing


< 10 breaths/min


Depressant drugs, increased ICP, diabetic coma

Hypoventilation

Irregular, shallow breaths


Narcotic OD, anesthetics, prolonged bedrest, splinting with pain


CO2 is retained, may cause acidosis

Cheyne-stokes

Regular, cyclic


Breathing 30-40 seconds followed by 20 sec apnea


CHF and other causes

Biots

Ataxic


Irregular, deep breaths


Slow rate with periods of apnea


Precedes Cheyne-stokes

Stertorous

Snoring

Stridor

Croup, foreign body, growth on vocal cords, high-pitched on inspiration

PEFR

Peak Expiratory flow rate


Maximum flow of air during forced expiration


Useful surrogate to measure vital capacity


Compare results to pt's personal best (% of pt's personal best) or to norms (% of predicted)

Atelectasis

Collapsed alveoli


Predisposes to PNA

PNA

Pneumonia: infection of alveoli, interstitial lung tissue, and/or bronchioles

Viral PNA

More common in children

CAP

Community acquired PNA


usually strep or H. Flu

Atypical PNA

Mycoplasm


Walking PNA

Nosocomial PNA

Hospital acquired


Usually gram (-), ie. pseudomonos


staph if gram (+)

Aspiration Pneumonia

Aspiration of gastric contents


Usualy r/t poor gag refluex

Lobar PNA

Large area of lung parenchyma involved


Very serious type of PNA

Bronchitis

Inflammation of bronchi


Acute or Chronic

Emphysema

Destruction of alveoli


Decreased gas exchange

Asthma

Intermittent bronchospasm, constriction


May lead to chonic lung disease

Pleural effusion

Fluid in pleural space

Pneumothorax

Air in pleura space


Collapsed lung

Hemothorax

Blood in pleural space

Empyema

Purulent exudate in pleural space

Cystic Fibrosis

Autosomal recessive disorder of exocrine glands affecting lungs, pancreas, sweat glands

Bronchiectasis

Chronic dilation of bronchi and bronchioles


Caused by repeated infections


Very serious condition

Corpulmonale

Lung disease that causes hypertrophy of right ventricle and progresses to heart failure

Pre-term infants

Surfactant is produced at 32 weeks gestation


Collapse of alveoli if pre-term labor before 32 weeks


Assessing infants/children

Count RR for 60 seconds, irregular rate


Less than 3 mo: obligatory nose breathers, nasal obstruction can cause death because soft palate obstructs oral airway - suction out nares


Less than 5-6 yrs: bronchovesicular breath sounds normal in lung fields