2- Abnormalities Of Teeth

Front 1

deciduous dentition develops from __ to __

Back 1

14th week of gestation through one year after birth

Front 2

permanent dentition develops from __ to __

Back 2

6 months of age through age 15

Front 3

abnormalities vary from slight (___) to huge defects

Back 3

neo-natal ring

Front 4

if you look hard enough, ___% of children will have some degree of defect.

___ affected teeth per individual

Back 4

68%

3.6

Front 5

high fever:

abnormalities present as ___ rows of pits, __ symmetrical and correspond to the stage of development at the time of insult.

0-2 years of age affets __ and __ teeth

4-5 years of age affects ___, ___, and ___ teeth

Back 5

horizontal

bilaterally

anterior teeth and 1st molars

cuspids, bicuspids, and 2nd molars

Front 6

___ is enamel defect in the permanent teeth due to periapical infections of the overlying deciduous tooth

Back 6

turner's tooth

Front 7

Turner's tooth:

___ depends on timing, duration and severity of insult

if infection, most commonly found in ___ teeth followed by ___ teeth

if trauma related, most commonly found in ___ teeth

Back 7

hypoplasia (of enamel)

bicuspids, anterior teeth

maxillary central incisors

Front 8

___ is the ingestion of excess amounts of fluoride in drinking water (>___ppm)

defects are ___ dependent

Back 8

dental fluorosis

>4ppm

Front 9

congenital syphilis (AKA ____) is the transmission of the ___ oragnisms across the placenta to infect the developing fetus.

__ and __ are affected

Back 9

syphilitic hypoplasia

treponema

incisors an 1st molars

Front 10

congenital syphilis = ______ triad

it includes.....

Back 10

hutchenson's triad

hytchenson's teeth, interstitial keratitis, 8th nerve defect

Front 11

environmental structural defects can be restored with: (3)

Back 11

acid-etch composite resin

labial veneers

full crowns

Front 12

the loss of tooth structure caused by tooth-to-tooth contact

Back 12

attrition

Front 13

tooth wear that shows wear facets, pulp exposure, and sensitive dentin

Back 13

attrition

Front 14

the pathogenic loss of tooth structure secondary to external abrasive agents

Back 14

abrasion

Front 15

abrasion creates a horizontal cervical notch on the ___ surface

Back 15

buccal

Front 16

the pathologic loss of tooth structure due to a chemical process, generally and acid

Back 16

erosion

Front 17

___ is erosion secondary to gastric secretions

Back 17

perimolysis

Front 18

erosion destruction is proportional to the __ and __ of the insult

Back 18

frequency and duration

Front 19

the loss of tooth structure caused by the repeated tooth flexure and occlusal stresses causing the enamel to chip away at the ____

Back 19

abfraction

CEJ (wedge shaped lesion)

Front 20

internal resorption may follow ___ to the tooth of pulpitis (pink tooth of mummery).

it starts within the pulp and proceeds to the ___ destroying ___ along the wway

can only be detected how?

Back 20

trauma

PDL

dentin

radiographically

Front 21

____ resorption is very common, even a normal process of deciduous tooth resorption

Back 21

external

Front 22

____ shows bright yellow fluorescence under UV light

Back 22

tetracycline tooth discoloration

Front 23

tetracycline tooth discoloration severity is dependent on __ and ___

to prevent the staining do not give tetracycline during pregnancy and up to ___ years of age

treatment?

Back 23

dose, duration

8 years

full crowns or veneers

Front 24

___ tooth discoloration is prescribed for acne.

can discolor tooth, ___ and ___.

causes a ___ discoloration

Back 24

minocycline

bone, soft tissue

blue-gray

Front 25

___ are teeth that have failed to erupt on schedule.

___ are most common, followed by __ and __

Back 25

impacted teeth

3rd molars

cuspids then bicuspids

Front 26

2 factors that cause impacted teeth

Back 26

1. blcoked in their eruption path

2. lost their eruptive forces

Front 27

treatment choices for impacted teeth

Back 27

long-term observation

orthodontic eruption

transplantation

removal

Front 28

____ is abrupt cessation of eruption due to the fusion of cementum or dentin to the ____

Back 28

ankylosis

alveolar bone

Front 29

ankylosis is most commonly found in ____ and with the ____ tooth.

will cause ___

clinically, will provide a sharp, solid sound of ___

Back 29

children, primary first molar

infraocclusion ("submerged")

percussion

Front 30

___ (too few teeth) OR ___ (zero teeth)

Back 30

hypodontia

adontia

Front 31

hypodontia is common in the permanent dentition in ____ teeth, followed by ___ then ___ teeth/

it is more common in ___ than ___

Back 31

3rd molars

2nd premolars

lateral incisors

females than males

Front 32

hyperdontia is most common in ___ teeth and ___ sex.

order of prevalence:

Back 32

maxillary dentition

males

mesiodens, 4th molars, paramolars

Front 33

___ is eruption of teeth into an abnormal position.

most common is __ and __

Back 33

dental transposition

maxillary canines and premolars

Front 34

___ are prematurely erupted decidious teeth present at or soon after birth.

Back 34

natal teeth

Front 35

the most common natal teeth are ___ and ___.

only remove these if the are extremely __ or cuasing traumatic ulcerations (___ disease)

Back 35

mandibular incisors and maxillary incisors

mobile

Riga-Fede Disease

Front 36

microdontia is more common in ___ sex.

___ is rare, ___ is common (example: ___ then ___)

Back 36

female

diffuse

isolated, peg lateral then 3rd molar

Front 37

macrodontia is more common in ___ sex.

___ and ___ are rare

Back 37

males

diffuse and localized

Front 38

____ is an attempt of a single tooth bud to divide into two

___ is the union of two normally separated tooth buds into one

___ is the union of two adjacent teeth by their cementum

Back 38

gemination (same root canal)

fusion (separate root canals)

concrescence (after development due to trauma)

Front 39

concrescence often occurs in the ____ area

Back 39

maxillary molar area

Front 40

fusion or gemination?

1. normal count of teeth

2. less than normal count

Back 40

1. gemination

2. fusion

Front 41

Accessory cusps:

1. ____= ML cusp of maxillary molar

2. ___= cingulum enlargement on maxillary incisors

3. ___= central tubercles on mandibula rpremolars

Back 41

1. cusp of carabelli

2. talon cusp

3. dens evaginatus

Front 42

most commonly affected teeth in dens invaginatus?

Back 42

permanent lateral incisors

Front 43

treatment for dense-in-dente?

Back 43

restore the opening quickly or caries/pulpal pathosis will result

-early? occlude opening with restorative material

-late? endodontic and apicoectomy

Front 44

2 ectopic enamel pathosis

Back 44

enamel pearls and cervical enamel extensions

Front 45

enamel pearls are found on the ___ surface usually on ___ teeth

Back 45

root

maxillary molars

Front 46

cervical enamel extensions are from the CEJ into the ____, usually on _____ teeth

Back 46

furcation

mandibular molars

Front 47

___ is enlargement of the pulp chamber with increased occlusal-apical dimensions. no treatment necessary

Back 47

taurodontism

Front 48

___ is non-neoplastic (non-tumor) excessive cementum on the root

Back 48

hypercementosis

Front 49

hypercementosis is usually in adulthood and may be result of __, __ or __.

PDL surrounding it or no?

Back 49

trauma, infection, lack of occlusion

PDL surrounds it

Front 50

____ is an abnormal bend in the root possibly due to injury to the tooth bud

Back 50

dilaceration

Front 51

abnormal number of roots on a tooth

Back 51

supernumerary roots

Front 52

____ is a developmental abnormalitity in enamel

Back 52

amelogenesis imperfecta

Front 53

___ different hereditary subtypes exist of AI.

The problem may manifest in different time periods therefore there are 3 different classifications:

Back 53

14

hypoplastic AI (during elaboration of enamel matrix phase)

hypocalcified AI (mineralization of the matrix phase)

hypomaturation AI (maturation of enamel phase)

Front 54

main clinical problems of AI

Back 54

aesthetics, dental sensitivity, loss of enamel and vertical dimension

Front 55

AI:

1. severity of caries depends on:

2. hardness/softness depends on:

3. color depends on:

4. radiographic appearance depends on:

Back 55

1. caries= diet and type of AI

2. hardness= type of AI

3. color= type of AI and extrinsic staining

4. radiographic= type of AI

Front 56

diagnosis of AI and DI:

treatment of AI:

Back 56

diagnosis: radiograph, clinical exam, family pedigree

treatment: full crowns, facial veneers, overdentures

Front 57

____= hereditary opalescent dentin

Back 57

dentinogenesis imperfecta

Front 58

DI is autosomal ____.

enamel does not stay attached and the exposed dentin wears away ____

Back 58

dominant

quickly

Front 59

Sheilds classification of DI:

type 1,2,3

Back 59

type I= associated with osteogenesis imperfecta

type II= known as hereditary opalescent dentin (HOD)

type III= known as shell teeth

Front 60

radiographs of DI include ___ crowns, ___ constriction, ___ roots, and early obliteration of the ___.

___ are common

Back 60

bulbous crowns, cervical constriction, thin roots, early obliteration of the root canals/pulp chambers

root fractures

Front 61

Type I dentin dysplasia- nicknames and what is it?

Back 61

crown enamel and dentin are normal but root is short and malformed

"radicular dentin dysplasia" and "rootless teeth"

Front 62

type II dentin dysplasia- nicknames and what is it?

Back 62

pulp stones in enlarged pulp chambers

"coronal dentin dysplasia"

Front 63

_____ = ghost teeth

Back 63

regional odontodysplasia

Front 64

____ is the localized nonhereditary abnormality of tooth possibly due to vascular problem during development

Back 64

regional odontodysplasia

Front 65

in regional odontodysplasia, ____ teeth are common.

the "teeth" are conglomerates of __ and __

Back 65

maxillary anterior segment

enamel and dentin