A

Front 1

Acetylcholine

Back 1

Primary transmitter of the peripheral NS.

Front 2

Dopamine (DA)

Back 2

Catecholamine (tyrosine)/Biogenic amine

Front 3

Norepinephrine (NE)

Back 3

Catecholamine (tyrosine)/Biogenic amine

Front 4

Epinephrine (E)

Back 4

Catecholamine (tyrosine)/Biogenic amine

Front 5

Serotonin

Back 5

Biogenic amine

Front 6

Histamine

Back 6

Biogenic Amine

Front 7

Glycine (Gly)

Back 7

Amino acid Transmitter

Front 8

y-aminobutyric acid (GABA)

Back 8

Amino acid Transmitter

Front 9

Glutamate (Glu)

Back 9

Amino Acid Transmitter

Front 10

Aspartate (Asp)

Back 10

Amino Acid Transmitter

Front 11

Endorphin

Back 11

Opioid/Neuropeptide

Front 12

Enkephalin

Back 12

Opioid/Neuropeptide

Front 13

Dynorphin

Back 13

Opioid/Neuropeptide

Front 14

Nitric Oxide (NO)

Back 14

Gaseous Transmitter/ Mod

Front 15

Carbon Monoxide (CO)

Back 15

Gaseous Transmitter/ Mod

Front 16

Hydrogen Sulfide

Back 16

Gaseous Transmitter/ Mod (perhaps?)

Front 17

Tetrodotoxin

Back 17

Sodium Channel Blocker

Front 18

Saxitoxin

Back 18

Sodium Channel Blocker

Front 19

Mu-conotoxin

Back 19

Sodium Channel Blocker

Front 20

Lidocaine/procaine/tetracaine

Back 20

Use Dependent Sodium Blocker

Front 21

batrachotoxin

Back 21

Sodium Channel Promoter

Front 22

pyrethrins

Back 22

Sodium Channel Promoter

Front 23

brevetoxin

Back 23

Sodium Channel Promoter

Front 24

Botulinium Toxin

Back 24

Bacterial Toxin

Front 25

Tetanus Toxin

Back 25

Bacterial Toxin

Front 26

carbamylocholine/succinylcholine

Back 26

Agonist

Front 27

curare

Back 27

Antagonist

Front 28

physostigmine/neostigmine

Back 28

Inhibitor of Ach-esterase

Front 29

organophosphorous cmpds

Back 29

Inhibitor of Ach-esterase

Front 30

Acetylcholine

Back 30

Primary transmitter of the peripheral NS.Participates in several pathways in CNS degradation of paths occur in Alzheimer's disease

Front 31

Dopamine (DA)

Back 31

Catecholamine (tyrosine)/Biogenic amine Found in mid-brain and dienchephalon. Parkinsonism involved in it's degradation. Schizophrenia is linked to disorders in DA systems

Front 32

Norepinephrine (NE)

Back 32

Catecholamine (tyrosine)/Biogenic amine

Front 33

Epinephrine (E)

Back 33

Catecholamine (tyrosine)/Biogenic amine

Front 34

Serotonin

Back 34

Biogenic amine

Front 35

Histamine

Back 35

Biogenic Amine

Front 36

Glycine (Gly)

Back 36

Amino acid Transmitter

Front 37

y-aminobutyric acid (GABA)

Back 37

Amino acid Transmitter

Front 38

Glutamate (Glu)

Back 38

Amino Acid Transmitter

Front 39

Aspartate (Asp)

Back 39

Amino Acid Transmitter

Front 40

Endorphin

Back 40

Opioid/Neuropeptide

Front 41

Enkephalin

Back 41

Opioid/Neuropeptide

Front 42

Dynorphin

Back 42

Opioid/Neuropeptide

Front 43

Nitric Oxide (NO)

Back 43

Gaseous Transmitter/ Mod

Front 44

Carbon Monoxide (CO)

Back 44

Gaseous Transmitter/ Mod

Front 45

Hydrogen Sulfide

Back 45

Gaseous Transmitter/ Mod (perhaps?)

Front 46

Tetrodotoxin

Back 46

Sodium Channel Blocker

Front 47

Saxitoxin

Back 47

Sodium Channel Blocker

Front 48

Mu-conotoxin

Back 48

Sodium Channel Blocker

Front 49

Lidocaine/procaine/tetracaine

Back 49

Use Dependent Sodium Blocker

Front 50

batrachotoxin

Back 50

Sodium Channel Promoter

Front 51

pyrethrins

Back 51

Sodium Channel Promoter

Front 52

brevetoxin

Back 52

Sodium Channel Promoter

Front 53

Botulinium Toxin

Back 53

Bacterial Toxin

Front 54

Tetanus Toxin

Back 54

Bacterial Toxin

Front 55

carbamylocholine/succinylcholine

Back 55

Agonist

Front 56

curare

Back 56

Antagonist

Front 57

physostigmine/neostigmine

Back 57

Inhibitor of Ach-esterase

Front 58

organophosphorous cmpds

Back 58

Inhibitor of Ach-esterase

Front 59

Acetylcholine

Back 59

Primary transmitter of the peripheral NS.

Front 60

Dopamine (DA)

Back 60

Catecholamine (tyrosine)/Biogenic amine

Front 61

Norepinephrine (NE)

Back 61

Catecholamine (tyrosine)/Biogenic amine

Front 62

Influences sleep

Back 62

wakefulness

Front 63

Epinephrine (E)

Back 63

Catecholamine (tyrosine)/Biogenic amine

Front 64

Serotonin

Back 64

Biogenic amine

Front 65

Made from tryptophan (5-hydroxytryptamine

Back 65

5-HT)

Front 66

Implicated in onset of sleep

Back 66

mood

Front 67

Histamine

Back 67

Biogenic Amine

Front 68

Glycine (Gly)

Back 68

Amino acid Transmitter

Front 69

y-aminobutyric acid (GABA)

Back 69

Amino acid Transmitter

Front 70

Glutamate (Glu)

Back 70

Amino Acid Transmitter

Front 71

Aspartate (Asp)

Back 71

Amino Acid Transmitter

Front 72

Endorphin

Back 72

Opioid/Neuropeptide

Front 73

Enkephalin

Back 73

Opioid/Neuropeptide

Front 74

Dynorphin

Back 74

Opioid/Neuropeptide

Front 75

Nitric Oxide (NO)

Back 75

Gaseous Transmitter/ Mod

Front 76

Carbon Monoxide (CO)

Back 76

Gaseous Transmitter/ Mod

Front 77

Hydrogen Sulfide

Back 77

Gaseous Transmitter/ Mod (perhaps?)

Front 78

Tetrodotoxin

Back 78

Sodium Channel Blocker

Front 79

Prevents influx of sodium into cell

Back 79

preventing depolarization and subsequent action potentials and release of ACh

Front 80

Saxitoxin

Back 80

Sodium Channel Blocker

Front 81

Prevents influx of sodium into cell

Back 81

preventing depolarization and subsequent action potentials and release of ACh

Front 82

Mu-conotoxin

Back 82

Sodium Channel Blocker

Front 83

Prevents influx of sodium into cell in skeletal muscles

Back 83

preventing depolarization and subsequent action potentials

Front 84

Lidocaine/procaine/tetracaine

Back 84

Use Dependent Sodium Blocker

Front 85

batrachotoxin

Back 85

Sodium Channel Promoter

Front 86

Keeps sodium channel open for a much longer period of time

Back 86

leading to extensive depolarization

Front 87

pyrethrins

Back 87

Sodium Channel Promoter

Front 88

Keeps sodium channel open for a much longer period of time

Back 88

leading to extensive depolarization

Front 89

brevetoxin

Back 89

Sodium Channel Promoter

Front 90

Keeps sodium channel open for a much longer period of time

Back 90

leading to extensive depolarization

Front 91

Botulinium Toxin

Back 91

Bacterial Toxin

Front 92

Tetanus Toxin

Back 92

Bacterial Toxin

Front 93

carbamylocholine/succinylcholine

Back 93

Agonist

Front 94

Activates the Ach receptor to open for prolonged periods of time. Leads to prolonged depolarization (tremors). Eventually

Back 94

cells become desensitized because Na Channels are inactivated after prolonged depolarizations.. (relaxation)

Front 95

curare

Back 95

Antagonist

Front 96

Competitive inhibitor of Ach.. binding of the chemical prevents binding of Ach. Reduces the amplitude of EPP's

Back 96

leading to flaccid paralysis

Front 97

physostigmine/neostigmine

Back 97

Inhibitor of Ach-esterase

Front 98

organophosphorous cmpds

Back 98

Inhibitor of Ach-esterase

Front 99

Acetylcholine

Back 99

Primary transmitter of the peripheral NS.

Front 100

degradation of paths occur in Alzheimer's disease;Dopamine (DA)

Back 100

Catecholamine (tyrosine)/Biogenic amine

Front 101

Schizophrenia is linked to disorders in DA systems;Norepinephrine (NE)

Back 101

Catecholamine (tyrosine)/Biogenic amine

Front 102

Influences sleep

Back 102

wakefulness

Front 103

unknown function in brain;Serotonin

Back 103

Biogenic amine

Front 104

Made from tryptophan (5-hydroxytryptamine

Back 104

5-HT)

Front 105

Implicated in onset of sleep

Back 105

mood

Front 106

Mediate arousal;Glycine (Gly)

Back 106

Amino acid Transmitter

Front 107

found in brainstem and spinal cord;y-aminobutyric acid (GABA)

Back 107

Amino acid Transmitter

Front 108

Deficit is implicated in Huntington's disease;Glutamate (Glu)

Back 108

Amino Acid Transmitter

Front 109

Can act as excitotoxin causing cell death if in excess;Aspartate (Asp)

Back 109

Amino Acid Transmitter

Front 110

Ubiquitous excitatory transmitter;Endorphin

Back 110

Opioid/Neuropeptide

Front 111

binds preferentially to u-receptors;Enkephalin

Back 111

Opioid/Neuropeptide

Front 112

binds to d-receptors;Dynorphin

Back 112

Opioid/Neuropeptide

Front 113

binds to K receptors;Nitric Oxide (NO)

Back 113

Gaseous Transmitter/ Mod

Front 114

Does not bind to post synaptic cell but diffuses into cell and directly interacts with secondary messengers systems;Carbon Monoxide (CO)

Back 114

Gaseous Transmitter/ Mod

Front 115

A major action is activation of guanylyl cyclase;Hydrogen Sulfide

Back 115

Gaseous Transmitter/ Mod (perhaps?);Tetrodotoxin

Front 116

Prevents influx of sodium into cell

Back 116

preventing depolarization and subsequent action potentials and release of ACh;Saxitoxin

Front 117

Prevents influx of sodium into cell

Back 117

preventing depolarization and subsequent action potentials and release of ACh;Mu-conotoxin

Front 118

Prevents influx of sodium into cell in skeletal muscles

Back 118

preventing depolarization and subsequent action potentials;Lidocaine/procaine/tetracaine

Front 119

Keeps sodium channel open for a much longer period of time

Back 119

leading to extensive depolarization;pyrethrins

Front 120

Keeps sodium channel open for a much longer period of time

Back 120

leading to extensive depolarization;brevetoxin

Front 121

Keeps sodium channel open for a much longer period of time

Back 121

leading to extensive depolarization;Botulinium Toxin

Front 122

inhibits neurotransmitter release by cleaving proteins related to normal vesicle fusion and exocytosis;Tetanus Toxin

Back 122

Bacterial Toxin

Front 123

Inhibits neurotransmitter release in the inhibitory neurons of the spinal cord. This causes the inhibitory neurons to fail when trying to inhibit contraction in the alpha-motoneurons;carbamylocholine/succinylcholine

Back 123

Agonist

Front 124

Activates the Ach receptor to open for prolonged periods of time. Leads to prolonged depolarization (tremors). Eventually

Back 124

cells become desensitized because Na Channels are inactivated after prolonged depolarizations.. (relaxation);curare

Front 125

Competitive inhibitor of Ach.. binding of the chemical prevents binding of Ach. Reduces the amplitude of EPP's

Back 125

leading to flaccid paralysis;physostigmine/neostigmine

Front 126

prolongs duration and amplitude of EPP... leads to flaccid paralysis of musculature...... will slowly hydrolyze and lose effectiveness (reversible);organophosphorous cmpds

Back 126

Inhibitor of Ach-esterase