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44 Cards in this Set
- Front
- Back
ECG Criteria for VT vs. SVT + aberrancy
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1. Broad Complex > 0.16 msec
2. Tachycardia > 120 3. Absence of typical RBBB / LBBB morphology 4. Extreme Axis Deviation 5. AV dissociation 6. Capture Beats 7. Fusion Beats 8. Chest Lead Concordance 9. Brugada’s Sign {RS length > 100msec} 10. Josephson’s Sign {notching near nadir of S wave} 11. Rsr [ not rsR of RBBB] See side 3 |
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Which is incorrect in regards to Multifocal Atrial tachycardia {MAT}?
1. The rhythm is irregular 2. The R-R interval is chaotic, and the PR intervals vary. 3. The P waves have multiple morphologies 4. It is an unstable rhythm 5. It is associated with COPD/Chronic lung disease |
4. It is a stable rhythm
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Causes of Atrial Flutter
"Ship-RACV" |
S Stimulants {drugs}
H Hyperthyroidism I Idiopathic P Pericarditis RA Respiratory-Acute / Chronic C CCF V Valve dysfunction |
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Prognostic factors in CHADS2 scoring {for Embolic stroke reduction and anticoagulation in AF}
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Congestive Cardiac Failure
{EF < 35%} Hypertension Age > 75 years Diabetes Stroke / TIA Side 3 for Relative risk |
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Anticoagulation Guidelines for CHADS2 {Scores and Rx}
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0 = aspirin
1 = aspirin OR warfarin >= 2 = warfarin |
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in comparing AVNRT and AVRT, which is incorrect?
1. AVRT is less common than AVNRT 2. AVRT involves a macro reentry circuit 3. AVNRT involves a micro reentry circuit 4. AVNRT is associated with structural cardiac disease 5. AVRT involves an accessory AV pathway |
AVNRT is not usually associated with structural cardiac disease
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In regards to AVRT, which is incorrect?
1. Orthodromic conduction with AVRT is more common than antidromic conduction 2. Orthodromic conduction in AVRT involves retrograde conduction along the normal conducting AV pathway 3. Orthodromic conduction with AVRT tachycardia is difficult to distinguish from AVNRT tachycardia 4. AVNRT tachycardia rates are between 150-250bpm 5. Paroxysmal AVRT is associated with WPW |
2. Retrograde conduction occurs up the Accessory pathway
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Causes of completely irregular rhythms {7}
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1. Atrial Fibrillation
2. Atrial tachycardia with variable conduction 3. atrial flutter with variable conduction 4. Multifocal atrial tachycardia {MAT} 5. Multiple extrasystoles 6. Wandering pacemaker 7 Parasystole see side 3 |
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Which is incorrect regarding narrow complex tachycardias?
1. Atrial fluter is usually via a re-entrant mechanism 2. Normal AV nodal tissues rarely allow a ventricular response rate of more than 170-180 bpm 3. Atrial fibrillation is the result of multiple micro-reentry circuits. 4. Atrial fibrillation limits diastolic filling AND stroke volume of the ventricles. 5. Fibrillation waves are best seen in the inferior leads and V1 |
2. 150-165bpm
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Which is the association with atrial flutter:
1. Structural heart disease 2. Heart failure 3. Valve dysfunction {mitral} 4. Thyroid disease 5. All of the above |
5.
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Ashman Phenomenon rhythm associations {3}
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1. Atrial fibrillation
2. Atrial ectopy 3. Atrial tachycardia |
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Mnemonic for Causes of Atrial fibrillation:
SHH! I2 C3 VP2W |
S sick sinus syndrome
H hyperthyroidism H hypertensive heart disease Idiopathic I ischaemic heart disease C cardiomyopathy C CCF C cardiac surgery V valve disease {mitral*} P pericarditis P Pulmonary embolus WPW |
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IN regards to WPW, which is incorrect?
1. WPW is present in 0.1-0.3% of population 2. Males > females 3. only 25-50% WPW patients become symptomatic 4. 70% WPW patients have no underlying heart disease 5. Presenting rhythm in symptomatic patients is a reentrant tachycardia 50% |
5. 70-80%
AF seen in 10-30% patients with WPW. |
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In regards to WPW, which is incorrect?
1. Patients with WPW often have one or more classic features MISSING on the ECG. 2. The Kent fibres are the most common accessory pathway 3. Type A WPW = QRS mostly positive in V1. 4.The short PR interval is the result of the absent Av node conduction delay. 5. on the ECG: narrow QRS and no delta wave = ANTIDROMIC tachycardia |
5. Orthodromic tachycardia = narrow QRS and no delta wave ==> AV node anterograde conduction and retrograde accessory pathway conduction
Antidromic tachycardia = wide QRS and + delta wave = accessory pathway anterograde conduction , and AV node retrograde conduction. |
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In regards to WPW, which is incorrect?
1. Unstable patients should be cardioverted -synchronised-starting at 50-100J 2. A regular ORTHODROMIC [ narrow QRS] tachycardia is the single most common presentation of an accessory pathway syndrome. 3. Initial treatment for Orthodromic tachycardia {in order} = vagal ; adenosine ; BB; CCB ;digoxin ; amiodarone ; flecainide. 4. Symptomatic patients with an antidromic {wide QRS} tachycardia are at high risk of VT. 5. With a wide complex irregular tachycardic rhythm, adenosine{ and other nodal blockers} are contraindicated. |
5. VF
A wide complex irregular tachycardia at a ventricular rate > 250bpm is HIGHLY SUGGESTIVE OF AF AND WPW SEE SIDE 3 |
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Diseases associated with WPW {7}
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1. Idiopathic ***
2. Cardiomyopathy {HOCM} 3. Transposition of the great vessels {TOGV} 4. Ebstein's Disease 5. Mitral valve prolapse {MVP} 6. Tricuspid atresia 7. Endocardial fibroelastosis |
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4 ECG characteristics of Sinus tachycardia
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1. normal P waves
2. Normal P-R intervals 3.1:1 atrioventricular conduction 4. atrial rate 100-160 bpm |
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3 categories of sinus tachycardia stimuli
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1. Physiologic
2. Pharmacologic 3. Pathologic See side 3 |
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3 ECG characteristics of AVNRT
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1. P wave buried in QRS complex-and not visible
2. 1:1 conduction 3. normal QRS complex |
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Which is incorrect regarding management of SVT?
1. vagal manoevres have a success rate of 25% 2. The valsalva manouevre is the most effective vagal manouevre for conversion of reentrant SVT-particularly in supine position 3. early recurrences are seen in 25% patients treated with adenosine 4. adenosine is safe in pregnancy 5. Adenosine is contraindicated in narrow complex WPW |
5. It is not CI in NARROW COMPLEX WPW
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which is an incorrect association?
1. WPW and kent bundle 2. Lown-Ganong-Levine (LGL) Syndrome and James fibres 3.James fibres of LGL syndrome connect atrium and proximal HIS bundles 4. James fibres insert directly into infranodal conducting system 5. LGL syndrome ECG: short PR interval and wide QRS |
5. short PR interval and narrow QRS
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Mahaim bundles with AVRT originate from:
1. AV node 2. His Bundle 3. Bundle branches 4. All of the above 5. None of the above |
4.
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Which is incorrect in regards to Type A WPW:
1. V1 delta wave has + deflection 2. V1 has dominant R wave 3. Q waves present in inferior leads 4. Ventricular activation occurs first occurs in the inferior-posterior region of Left ventricle. 5. Delta wave has initial negative deflection in V1 |
5. V1 delta wave-positive deflection
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Type B WPW: which is incorrect?
1. V1 delta wave has initial positive deflection 2. May resemble an inferior wall MI 3. Lead V1 can have an RS or QS pattern 4. Delta wave has initial negative deflection in V1 5. Ventricular activation occurs in the inferior-posterior region of the Right ventricle |
1. V1 delta wave has initial negative deflection
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In regards to Type C WPW, which is incorrect?
1. Ventricular activation occurs first in the posterior lateral region of the Left ventricle. 2. Leads V5 has negative OR isoelectric pattern 3. Lead V6 can have a negative OR isoelectric pattern 4. Both Type A and Type C WPW have positive initial delta wave deflections 5. Delta wave is negative in lead V1. |
5. Positive delta wave deflection in V1.
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Brugada Criteria for VT
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1. Absent RS complex in all V leads==> VT
2. R to S nadir > 100msec in any V lead==> VT 3. AV Dissociation ==> VT 4. Typical BBB pattern ==> not VT |
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Main causes of VT
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1. coronary artery disease
2. Cardiomyopathy 3. Myocarditis 4. Valvular heart disease |
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Treatment options for VT
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1. DC cardioversion
2. pharmacological - Amiodarone 3. Rapid Right ventricular pacing {Overdrive pacing} |
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Which of the following is incorrect regarding Torsades de pointes?
A. It is more electrically unstable than monomorphic VT B. It is a depolarisation abnormality C. Syncope is the usual presenting symptom D. It can be caused by hypocalcaemia and hypomagnesaemia. |
B. Repolarisation abnormality
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List the causes of Torsades de Pointes.
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1. Hypo-electrolytes
a. magnesium b. calcium 2. Antiarrhythmic drugs [ I and II ] 3. TCA 4. Antidepressants / Antipsychotics a. phenothiazines b. citalopram 5. Congenital prolonged QT syndrome 6. Organophosphates 7. Complete heart Block [CHB] 8 Drug interactions a. erythromycin-terfenadine |
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Treatment options for Torsades de Pointes?
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1. Magnesium ***
2. Cardioversion [ haemodynamically compromised ] 3. Overdrive pacing [ rate 90-120] 4. isoprenaline infusion |
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Which is incorrect regarding Torsades de Pointes (TdP)
A. TdP may degenerate into VF B. The commonest cause of Polymorphic Ventricular tachycardia (PVT) is drugs C. TdP can be treated with Isoprenaline D. TdP is a specific form of PVT |
B. Ischaemia
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Which is incorrect regarding the pathophysiology regarding TdP?
A. A prolonged QT interval reflects prolonged myocyte depolarisation. B. Bigeminy, in the presence of a prolonged QT, can herald imminent TdP C. The sequence of progression from prolonged QT interval to TdP = EAD --> PVC --> 'R on T' --> TdP D. The prolonged repolarisation period gives rise to early after-depolarisations (EADs) |
D. Prolonged repolarisation
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Which is incorrect regarding Long QT Syndromes?
A. The normal QT interval is from the BEGINNING of the q, to THE END of the T wave. B. The Correct QT (QTc) via Bazett's Formula, corresponds to a QT duration at a Heart Rate of 60. C. The QT should be roughly < 50% of the R-R interval at any rate. D. The aim of External pacing for Torsades is to Overdrive the bradyarrhythmia. |
C. rate should be less than 90.
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In regards to overdrive pacing in Torsades de Pointes, which is incorrect ?
A. In the ED, it refers to setting the pacing greater than the rate of the bradyarrhythmia. B. There is little actual data to support its efficacy in the ED C. Monomorphic VT can also be directly overdrive paced-but requires transvenous pacing. D. Pacing occurs continuously, in the asynchronous mode. |
D. It actually occurs as brief trains of 6-10 beats of asynchronous pacing.
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Which is incorrect regarding Long QT and Torsades?
A. Prolonged QT can result in polymorphic VT or Mono-morphic VT B. Prolonged QT can result in VF. C. The arrhythmia of TdP is usually initiated by an 'R on T' ventricular extrasystole. D. The Romano-Ward Congenital Syndrome is associated with deafness. |
D. Lange-Nielson is associated with nerve deafness.
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Which electrolyte abnormality is least associated with Increased QT interval?
A. Decreased potassium B. Decreased magnesium. C. Decreased calcium. D. Increased calcium. |
D.
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Which is an unlikely drug cause of Long QT interval ?
A. Non-sedating antihistamines B. Organophosphates C. Class I and II Anti-arrhythmics D. Antipsychotics Typical and Atypical. |
C. Class Ia , Ic and III.
Not Class II |
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Which of the following Clinical conditions is least likely to cause a prolonged QT interval?
A. Severe bradyarrhythmias eg CHB. B. Subarachnoid Haemorrhage C. Hypothermia D. Hyperthermia |
D.
C. "Severe" Hypothermia does prolong QT interval |
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Which of the following is incorrect regarding the immediate Treatment for TdP?
A. Arrested = Defibrillate B. Conscious + Sustained = Cardiovert C. Conscious and non-sustained = External pacing D. Correct bradyarrhythmia and underlying cause ( MgSO4 ; atropine ; isoprenaline ) |
C. External pacing only after correcting bradyarrhythmia and underlying cause ( Hypokalaemia ; Hypocalcemia ; Hypothermia )
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List the 5 main ECG phenomena (Types) associated with Syncope in the young adult / athlete.
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1. HOCM
2. ARVC [ arrhythmogenic right ventricular cardiomyopathy] 3. Brugada Syndrome 4. Long QT Syndrome 5. WPW |
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In regards to the ECG of HOCM, which of the following is incorrect?
A. It has peaked P waves, representing Left atrial enlargement. B. Pathological Q waves exist in the lateral leads. C. High precordial voltages are present. D. There are ST-T wave abnormalities- particularly deep T wave inversion laterally / pre cordially. |
A. Left atrial enlargement = broad bifid P waves
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which is incorrect regarding HOCM?
A. It is the leading cause of sudden cardiac death in healthy athletes < 35 years. B. Its murmur is increased with valsalva and standing. C. It has Epsilon waves present. D. Handgrip reduces the murmur of HOCM. |
C. No -this is present in ARVC
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In regards to Arrhythmogenic Right Ventricular Cardiomyopathy (ARVC), which of the following is incorrect?
A. The epsilon waves present are similar to the Osborn waves of Hypothermia. B. It is the second most common cause of sudden cardiac death in young athletes. C. There is precordial WRS prolongation > 0.14 seconds. D. There is T wave inversion. |
C. QRS > 0.11 seconds in V1-3.
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