• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/27

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

27 Cards in this Set

  • Front
  • Back
Skin Normal Flora:
Diptheroids
Gram +, varied shape, low virulence
Cause body odor
Cornyebacterium diptheria
Propionibacterium acnes
Skin Normal Flora:
Staphylococci
Gram +, salt tolerant
Prevent pathogenic colonization
Maintains normal flora balance
S. epidermis
Skin Normal Flora:
Yeast
Tiny lipophilic, round or oval shapes, some rods
Generally harmless
Folliculitis Agent
S. aureus - Gram +
Folliculitis Symptoms
Small red bump/pimple at hair follicle
Furuncle - infection spreads to adjacent tissue
Carbuncle - large infected area
Folliculitis Pathogenesis
Infection induces inflammatory response and draws leukocytes to site
Systemic infection can infect other organs/tissues
Folliculitis Epidemiology
Normally found in nostrils
Spread through contact
Survives outside well
Folliculitis Prevention/Treatment
Very little treatment
Resistant to drugs
Surgical drainage of boils
Scalded Skin Syndrome Symptoms
Skin appears burned
Begins as redness
Nose, mouth, genitals painful
Symptoms appear within 48 hours
Scalded Skin Syndrome Agent
S. aureus
Caused by exfoliatin toxin
Destroys integral layer of epidermis
Scalded Skin Syndrome Pathogenesis
Toxin released at infection site
Outer skin is lost
40% mortality
Scalded Skin Syndrome Epidemiology
Can appear in any age group
Transmitted person to person
Scalded Skin Syndrome Prev/Treat
Prev - isolation
Treat - bactericidal antibiotics, removal of dead skin
Streptococcal Impetigo Agent
S. pyogenes - Gram +, Beta hemolytic cocci
Streptococcal Impetigo Symptoms
Superficial infection
Blisters develop, break, and crust over
Streptococcal Impetigo Pathogenesis
Infection through minor injuries
Bacteria produce proteases and nucleases
Bacteria surface proteins interfere with phagocytosis
Streptococcal Impetigo Epidemiology
Prevalent in children
Spread person to person
Streptococcal Impetigo Prev/Treat
Prev - cleanliness, avoidance of infected
Treat - Antiseptics, penicillin/erythromycin
Rocky Mountain Spotted Fever Agent
Rickettsia ricketrsii
Gram -, non-motile, coccobacillus
Obligate, intracellular, parasite
Very small, hard to see on stain
Rocky Mountain Spotted Fever Symptoms
Initial pink spot rash - starts on hands and feet
Spots spread +become hemorrhagic
Can cause shock or death if spreads to organs
Rocky Mountain Spotted Fever Pathogenesis
Spread through bite then into blood
Endocytosed by cells
Endotoxin causes disseminated intravascular coagulation - leads to shock
Rocky Mountain Spotted Fever Epidemiology
Zoonoses
Passed by ticks - Dermacentor andersoni and variablis
Rocky Mountain Spotted Fever Prev/Treat
Prev - Avoidance/removal of ticks
Treat - early doxycycline and chloramphenicol is effective
Lyme Disease Agent
Borrelia burgdorferi
large, microaerophillic spirochete
Circular, not linear genome
Multiple copies
Lyme Disease Symptoms
Stage 1 - rash
Stage 2 - 2-8 weeks after, heart+nerves impaired
Stage 3 - 6 months after, arthritis, chronic nerve impairment
Lyme Disease Pathogenesis
Spreads radially from bite
Causes inflammatory reaction
Lyme Disease Prev/Treat
Prev - avoidance/removal ticks
Treat - early drugs