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11 Cards in this Set
- Front
- Back
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All of the BB blockers are classified as Class II except which drug?
Which group of drugs are Class IV drugs? Class III? |
Sotalol - Class III
CCB's - Class IV Amiodarone - Class III |
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Which subclass of Class I (A, B or C) do Lignocaine, Quinidine, Procainamide, Flecainide and Phenytoin belong to?
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Easy - reverse order alphabetically and then group into classes e.g.
1A - Quinidine, Procainamide 1B - Phenytoin, Lignocaine 1C - Flecainide (NB does not work for entirety of classes, just those we actually use) |
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Unlike the rest of Class 1, which subclass SHORTENS repolarisation and AP duration?
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1B (1A and 1C lengthen repolarisation and AP duration).
NB all of Class I are Na CB's that slow depolarisation and conduction |
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The only antiarrhythmics that reduce mortality in any condition are...?
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BB's in MI. Prophylaxis for reperfusion and vent dysrhythmias post MI
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Which of the following is not a relative C/I for short term BB use?
1) asthma 2) heart failure 3) DM 4) PVD 5) 3rd TM pregnancy |
DM is a complication for LT use, NOT short term
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Which class of drugs may be used for both VF and AF?
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The 'antifibrillatory' class - Class III. Amiodarone. Can also be used for accessory pathway syndromes
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What are the short term AE's of amiodarone? The common and uncommon LT AE's?
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ST - bradycardia, hypotension, heart failure
Common LT (EYES too big for STOMACH) - corneal deposits, photosensitivity, GI disturbance Uncommon LT - proarrhythmia, bradycardia, hypotension; hypo and hyperthyroidism; pulmonary fibrosis |
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Class IV drugs block which channels?
When may Verapamil be used in Rx of accessory p-way syndromes? Absolute C/I to CCB? |
Slow Ca channels in both myocardial and vascular sm mm cc
Verapamil may be used in accessory p-way syndromes when AVN is anterograde p-way C/I - 2nd and 3rd degree blocks |
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Digoxin acts by inhibiting which protein - and what is the net electrolyte effect?
What may enhance the AE's of dig? |
Na K ATPase. Intracellular Na rises, K falls, and intracellular Ca rises (responsible for positive inotropic effect)
AE's of dig are enhanced by LOW K+ and Mg and HIGH Ca and catecholamines (excess); also severe acid base disturbance |
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For which arrhythmia is Magnesium first line rx? What else may it be useful for?
Dose for VT |
Torsades
Narrow complex tachycardia - e.g. AF, MAT, reentrant SVT VT VT - 2-4gm slow IV bolus |
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Regarding adenosine, which is incorrect
A slows AVN conduction B slows conduction in BOTH anterograde and retrograde paths of reentrant circuit C may rarely precipitate VT and AF D is a useful agent for AF with accessory pathway E is not a useful agent for non-reentrant SVT |
D Using ANY drug (e.g. adenosine, CCB, BB) that suppresses the AVN conduction with either a ventricular rhythm OR AF+accessory pathway (e.g. any wide complex tachycardia) may cause precipitious haemodynamic collapse R1212
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