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110 Cards in this Set

  • Front
  • Back
the left anterior decending coro arter supplies ____ conduction system
supplies 2/3 conduction system

major clot here is know as the widower
health history
childhood illness
infection dz
immunization
major illnesses/hosp
meds
allergies
fam health hx
occupation
geo location
environment
nutrition
substances
elimination
activity
sleep and rest
role in the life
sexual orientation
reproductive hx
acute coronary syndrome
faitgue
sob
sleep disturb
anxiety
passing discomfort
neuropathy
gender differences

these are precurses to major events
phsical exam heart
gen appereance
mental status

skin/nails
-color/texture

bp
-postural hypotension

pulse
-arterial
-juglar
-pulsus paradoxus or paradoxical pulse (heart rt decrease on inspiration)
heart phys exam: extremities
cap refill
vascualr changes
hematoma
periph edma
clubbing
heart phys abdomen
hepatomegaly
hepatojugular refulx
-venous overload
-decreased returne

ascites
bladder distension
heart phys exam: heart sounds
normal s1/2 sounds
pathologic splitting of s2
s3/4 abnormal
clicks
murmurs
pericardiacl friction rub
heart phys exam: lung
tachypnea/other patterns
crackles
hemoptysis
cough
s3 heart sound may be normal in what stage of lfe
childhood
s3 hear sound sounds like a ____ beat

____ ____ ____
triplet

lub dub dub
s4 heart sounds like
a horse galloping

heard late is diastole just before s1
rub sound around the heart are indivative of
pericarditis
s1 is
depolarization of ventricles
s2 is
repolarization of the ventricles after teh t wave
diagnostic tests for heart
hematology
coagulation
-ptt/inr
liver function
metabolic panel
cardiac enzymes
-ckmb
-tropinin t or I
blood chemistry
-lipid profile
-cholesterol level
-bnp
-c reactive protein
-homocystein
-thyroid function
cardiac cathetrization with angiography

indications
sx environeent like in OR

mesure CO
valve structures or defects
right heart cath
usucally done before left heart cath
where is the cath placed for a right heart cath
radial vessel
femoral vein
if a study on the coronary arteries is indicated which side of the heart do you enter the cath from?
left

otherwise go into the right
what is the major risk post cardiac cath
bleeing
during cardiac cath, dye is used. what boddy system ar you worried about
renal system

can cause failure.
flush them
cardiac cath fasting
8-12 before the test
sedations during cardiac cath
moderate

versad/fentanyl

pt will be awake but in a twilight
risk during cardiac cath
death can occur. heart can stop
bedrest for cardiac cath
bedrest 4 hours post procedure
hemodynamic monitoring cvp
central venous pressure reading
-line is threaded into right side of heart. crude measuremtn of fluid volume in right side of heart. align the bottom of cvp device with 4th intecostal space, midclavicular line.
cvp monitoring
fluid in right side of heart
normal 2-6mm/h20
>6indicated ^rventricular preload
pulmonary artery pressure monitoring
ICU
arterial cath into radial artery
-allows for frequent ABG
-most monitor for disonnetion
-set up with flush system:
-transducer which converts the pressure into meaningful number
swan cath
pulmonary artery pressure monitorying
--goes into pulmonary artery
--gives us measurement of left ventricular functioning
-more accurate than CVP
PAPM vs CVP
PAPM=left heart
CVP=right heart
qrs complex rhythm strips
5 are = 0.2 seconds
p wave
atrial depolarization
qrs shouldnt be wider than
3 squares, 0.12
qt interval should be (in length)
0.32-0.40
reading ekg
1. rate and rhythm
2. intervals
angina define
clinical syndrom characterized by sudden onset of chest pain or pressure precipitated by an event the demands of the heart are ^ so the o2 supply to myocardium is diminished
patterns of angina
stable
unstable
intractable
varant
silent
stable angina
predictable
comes on with exertion
relieved with rest or nitro
when episodes worsen then they become more unpredictable
unstable angina
event lasts longer
unable to tolerate as much as before
intractable angina
severe incompasitating chest pain
varient angina
involuntarily contratction (spasm)
silent ischemia
no pain or discomfort felt
only seen during ekg
angina medical manage
nitrates
beta block
ccb
asa/platelet aggregate inhibit
angina dx
ecg/exercise stress testing
nuclear scanning
coronary angiography
someone arrives with chest pain. golden rule?
door too balloon time is 60 minutes

cath lab
risk factor management angina
smoking
cholesterol
control htn dm
loose weight
injury to heart = _____ on ECG
elevation ST

may not be elevated. but cardiac enzymes will be
infarction in heart = ____ on ecg
inverted T wave
CKmb elevates how long after injury?
4-8
peaks 14 hours
returns 24-48
enzymes are drawn _____ during suspected heart injury
admission
12hrs
24 hours
troponin 1 specific to _____ tissue
cardiac
tropin eleveated upon injury
3 hours
peak 10-12
up for as long as 3 wks
troponin drawn when injury is suspect
initially
12 hrs
daily 3-5 days
evaluate 5-6 wks
diagnosistics in ER for mi damage
enzymes, cbc, electrolytes
12 lead ECG
drugs to give ER mi injury
o2 keep %>90
iv + blood draw
ms04 2-8mg iv
asa 325

then maybe these

thrombolytics
antiplatelets
nitrates
aniticoag
beta blockers
antidysthymics
ccb
revascularizzation
restore blood supply
how to revascularize
pharm
-thrombolyitc
glycoprotien receptor antagoinist
persantine/pletal
invasive
fibrinolytic therapy contraindivations
active internal bleeding, inflam bowel disease, peptic ulcer disease

GI bleed < 6 mo
known bleeding disorder
aortic dissection
neurosurgical proceduure <2mo
acute Mi med management : in patient
treat the acute attack
manage pain
improve perfusion
monitor for complications
tx dysrthimas and prevent life threatening ddysrthmias
hemodynamic studies
collaborative problems in acute MI
dysrhythmias
cardiogenic shcok
hf
PE
P edema
stroke
recurrent MI
dresslers syndrom
myocardical necrossic ocmp
fever
pericarditis
plueritis
plureral effusion
joint pain

tx
asa
corticosteroids
acute mi nursing dx
acute pain
impaired oxygenation
activity intolerance
risk for imbalance fluid volume
decre cardiac output
fear of dying
impaired tissue perfussion
altered nutrtition
impaired gas exchange
acute MI rehab/edu
strenthen the myocardium
phase 1 - get them up and ambulate. how to check pulse
p2 - monitored situation. 12 wks. wear tele unit when in 3xwk
p3 - outpatient not monitored
p4 - maintenence
cardic sx goals
repare, reconstruct, substitute

imporve cirucaltion and cardiac function
preop nursing care cardiac sx
health hx/physical psychosocal assessment


reduce fear and anxiety

discuss complications

patient teaching
-be clear about what to expect
what to expect after cardiac sx
tubes
pace maker wires
stockings
dangle soon
pain
chest binder
foley
peripheral edema
complications post op cardiac sx: caridac
hf
cardio shock
mi
dysrhyth
pericardial effusion
cardiac tampnade
cardiac arrest
complications post op cardiac sx; pulmo
pulmonary edema
PE
pleural effusions
pneumothorax
hemothorax
resp failure
ARDS
permanent pacemaker
placed beneath skin in pectoral region
lead is tunnelled subq between pacemaker and external jugalr vein
pacemeaker lead enters external jugalr vein
placement of pacemaker is done
during surgery under flouroscopy
batteries in pacemakers need to be changed
q10 yrs
outpatient
external temp pacemaker
electrodes stuck to chest attatched to defrilator that is on at constant rate. defibr turned up until heart responds to electircal
universal lettering
5 letters
3 most common
first 3 letter on pacemaker
signifies the chamber or chambers being paced

av or d
second letter in pace
describes the chamber or chambers being sensed by the generator
third letter in pace
type of response by the pacemaker to what is sense

inhibited triggered dul or none
fourth/fifth letter in pace
used for permanent pacemakers and generators

fourth has the ability to program or reset the device.

fith indicates the anti tachycadia and or defibrillation capability
 complications of pace maker insertion
local infection
bleeding
hematoma
-localized pain at the site of insertion

hemothorax
dislocation of leads
phrenic nerve stimulation and hicupping
ventricular ectopy
tachycard (irritation of centrical wall)
movement of lead
patient teaching with pace maker insertion
no airport metal detector
no heavy lifting
no pulling activity
no raising over head
identifying
post operative complications of pacemaker implant
hemorrhage or coagulopathy
stroke
renal failure
hepatic failure
inection
sepsis
hypothermia
infectious cardiac disorders

types
rheumatic endocarditis
infective endocarditis
myocarditis
pericarditis
-sub acute, acute, chronic
medical management of infectious cardiac disorders
eradicate infection
maximize cardiac output
promote comfort
avoid nsaids
which microbe can effect cardiac through infection
strep
nursing problems for infectious cardiac disorders
activity intolerance
acute pain
decreased cardiac output
ineffective health maintencne
ineffective tissue perfusion
risk for imbalanced nutrition
valvular diesea and septal defects

types
mitral
aortic
tricuspid
ulmonic
valvular disease and petal defects pathophys
prolapse
regurgitation
stenosis
ruptured papillae
related to infection or damange with disease
congenital defects
clinical manifestations of valvular disease/septal defects
activity intolerance
murmer
fatiuge
sob
syncope
palpatations
chest pain
tx valvular disease/septal defect
sx to correct
atb to prevent infxn during procedures
mardiomyopathy

types
dilated
hypertrophic
restrictive

series of events that end up in a decreased cardiac output
dilated myopathy
most common
pregnant women
etoh intake
viral infections that cause great deal of stress on theart
hypertrophi myopathy
inherited condition
medical management of myopathy
correct heart failure
implantable cardioverter defib, pacemaker
infective edocarditis prophylaxis
alcohol septal ablation
systemic anticoagulation
sx managment of myopathy
heart transplatation
mechanical assist devices
artificial hearts
HF etology/risk factors
abnormal loading conidtions
abnormal muscle function
conditionas that precipitate heart failure
causes of HF, chronic
CAD
hypertensive heart disease
rheumatic /congenital heart disease
disease of heart valves
-valves can be leaky or overly stiff
cor pulmonale/pulmonary hypertension
cardiomyopathy
anemia
damage to hear from etoh/other drugs
acute causes of hf
acute mi
arrhythmias
PE
thyrotoxicosis/hypothyroidsm
HTN crisis
rupture of papillary muscle
ventricular septal defect
myocarditis
HF pathophys
ventricular dilation
increased sympathetic nervous system stim
stim of RAAS
decompensation heart failure
HF class
high/low ouutput failure
left/right
acute/chron
back/forward
HF med management
improve ventric pump
reduce workload
reduce fluid retention
reduce stress/risk of injury
HF sx manage
ventricular assist device
heart transplant
dx of hf is made when ejection fraction is less that what %?
40-systolic heart failure
when the ejection fraction is >40 but symptomatic it is called
diastolic heart fialure
factorsa aggravating HF
ischemia/infart
sodium excess
excess fluid
med noncompliance
arrthmias
intervurrent illness
conditions associated with increased metabolic demand
left vent dysfunction s/s
DOE
paroxysmal nocturnal dysnea
tachy/palpitations
cough/wheeze/hemoptysis
AMS

rales
pulmonary edmea
S3
oliguria
decreased tissue perfusion, peristalsis, abosorption
right sided HF s/s
abd pain
anorexia
nausea
bloating/depend edmea
swelling
nocturia decreased frequency
physical s/s right sided hf
periphreal edema
venous engorgement
JVD
abd jugular reflux
hepatomecaly
ascites
murmurs
AMS
compensated HF
whenc ompensatory mech are successfull at maintaining cardiac output need for metabolic demands and tisue perfusion
general measures for lifestyle mod in HF
weight reduction
stop smoking avoid ETOH
daily weight
exercise
nutrition therapy
-reduced sodium intake
medical considerations for HF
treat HTN, hyperlipidema, diabetes, arhythmias
coronary revascularization
anticoag
immunization
cose outpatient monitoring
reduce readmissions
outcomes and interventions
maintina fluid balance
decrease anxiety
improved qual of sleep
adhere to regimine

stop smoking
stop ETOH
daily weight
na restrict
ortho hypo