Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
110 Cards in this Set
- Front
- Back
the left anterior decending coro arter supplies ____ conduction system
|
supplies 2/3 conduction system
major clot here is know as the widower |
|
health history
|
childhood illness
infection dz immunization major illnesses/hosp meds allergies fam health hx occupation geo location environment nutrition substances elimination activity sleep and rest role in the life sexual orientation reproductive hx |
|
acute coronary syndrome
|
faitgue
sob sleep disturb anxiety passing discomfort neuropathy gender differences these are precurses to major events |
|
phsical exam heart
|
gen appereance
mental status skin/nails -color/texture bp -postural hypotension pulse -arterial -juglar -pulsus paradoxus or paradoxical pulse (heart rt decrease on inspiration) |
|
heart phys exam: extremities
|
cap refill
vascualr changes hematoma periph edma clubbing |
|
heart phys abdomen
|
hepatomegaly
hepatojugular refulx -venous overload -decreased returne ascites bladder distension |
|
heart phys exam: heart sounds
|
normal s1/2 sounds
pathologic splitting of s2 s3/4 abnormal clicks murmurs pericardiacl friction rub |
|
heart phys exam: lung
|
tachypnea/other patterns
crackles hemoptysis cough |
|
s3 heart sound may be normal in what stage of lfe
|
childhood
|
|
s3 hear sound sounds like a ____ beat
____ ____ ____ |
triplet
lub dub dub |
|
s4 heart sounds like
|
a horse galloping
heard late is diastole just before s1 |
|
rub sound around the heart are indivative of
|
pericarditis
|
|
s1 is
|
depolarization of ventricles
|
|
s2 is
|
repolarization of the ventricles after teh t wave
|
|
diagnostic tests for heart
|
hematology
coagulation -ptt/inr liver function metabolic panel cardiac enzymes -ckmb -tropinin t or I blood chemistry -lipid profile -cholesterol level -bnp -c reactive protein -homocystein -thyroid function |
|
cardiac cathetrization with angiography
indications |
sx environeent like in OR
mesure CO valve structures or defects |
|
right heart cath
|
usucally done before left heart cath
|
|
where is the cath placed for a right heart cath
|
radial vessel
femoral vein |
|
if a study on the coronary arteries is indicated which side of the heart do you enter the cath from?
|
left
otherwise go into the right |
|
what is the major risk post cardiac cath
|
bleeing
|
|
during cardiac cath, dye is used. what boddy system ar you worried about
|
renal system
can cause failure. flush them |
|
cardiac cath fasting
|
8-12 before the test
|
|
sedations during cardiac cath
|
moderate
versad/fentanyl pt will be awake but in a twilight |
|
risk during cardiac cath
|
death can occur. heart can stop
|
|
bedrest for cardiac cath
|
bedrest 4 hours post procedure
|
|
hemodynamic monitoring cvp
|
central venous pressure reading
-line is threaded into right side of heart. crude measuremtn of fluid volume in right side of heart. align the bottom of cvp device with 4th intecostal space, midclavicular line. |
|
cvp monitoring
|
fluid in right side of heart
normal 2-6mm/h20 >6indicated ^rventricular preload |
|
pulmonary artery pressure monitoring
|
ICU
arterial cath into radial artery -allows for frequent ABG -most monitor for disonnetion -set up with flush system: -transducer which converts the pressure into meaningful number |
|
swan cath
|
pulmonary artery pressure monitorying
--goes into pulmonary artery --gives us measurement of left ventricular functioning -more accurate than CVP |
|
PAPM vs CVP
|
PAPM=left heart
CVP=right heart |
|
qrs complex rhythm strips
|
5 are = 0.2 seconds
|
|
p wave
|
atrial depolarization
|
|
qrs shouldnt be wider than
|
3 squares, 0.12
|
|
qt interval should be (in length)
|
0.32-0.40
|
|
reading ekg
|
1. rate and rhythm
2. intervals |
|
angina define
|
clinical syndrom characterized by sudden onset of chest pain or pressure precipitated by an event the demands of the heart are ^ so the o2 supply to myocardium is diminished
|
|
patterns of angina
|
stable
unstable intractable varant silent |
|
stable angina
|
predictable
comes on with exertion relieved with rest or nitro when episodes worsen then they become more unpredictable |
|
unstable angina
|
event lasts longer
unable to tolerate as much as before |
|
intractable angina
|
severe incompasitating chest pain
|
|
varient angina
|
involuntarily contratction (spasm)
|
|
silent ischemia
|
no pain or discomfort felt
only seen during ekg |
|
angina medical manage
|
nitrates
beta block ccb asa/platelet aggregate inhibit |
|
angina dx
|
ecg/exercise stress testing
nuclear scanning coronary angiography |
|
someone arrives with chest pain. golden rule?
|
door too balloon time is 60 minutes
cath lab |
|
risk factor management angina
|
smoking
cholesterol control htn dm loose weight |
|
injury to heart = _____ on ECG
|
elevation ST
may not be elevated. but cardiac enzymes will be |
|
infarction in heart = ____ on ecg
|
inverted T wave
|
|
CKmb elevates how long after injury?
|
4-8
peaks 14 hours returns 24-48 |
|
enzymes are drawn _____ during suspected heart injury
|
admission
12hrs 24 hours |
|
troponin 1 specific to _____ tissue
|
cardiac
|
|
tropin eleveated upon injury
|
3 hours
peak 10-12 up for as long as 3 wks |
|
troponin drawn when injury is suspect
|
initially
12 hrs daily 3-5 days evaluate 5-6 wks |
|
diagnosistics in ER for mi damage
|
enzymes, cbc, electrolytes
12 lead ECG |
|
drugs to give ER mi injury
|
o2 keep %>90
iv + blood draw ms04 2-8mg iv asa 325 then maybe these thrombolytics antiplatelets nitrates aniticoag beta blockers antidysthymics ccb |
|
revascularizzation
|
restore blood supply
|
|
how to revascularize
|
pharm
-thrombolyitc glycoprotien receptor antagoinist persantine/pletal invasive |
|
fibrinolytic therapy contraindivations
|
active internal bleeding, inflam bowel disease, peptic ulcer disease
GI bleed < 6 mo known bleeding disorder aortic dissection neurosurgical proceduure <2mo |
|
acute Mi med management : in patient
|
treat the acute attack
manage pain improve perfusion monitor for complications tx dysrthimas and prevent life threatening ddysrthmias hemodynamic studies |
|
collaborative problems in acute MI
|
dysrhythmias
cardiogenic shcok hf PE P edema stroke recurrent MI |
|
dresslers syndrom
|
myocardical necrossic ocmp
fever pericarditis plueritis plureral effusion joint pain tx asa corticosteroids |
|
acute mi nursing dx
|
acute pain
impaired oxygenation activity intolerance risk for imbalance fluid volume decre cardiac output fear of dying impaired tissue perfussion altered nutrtition impaired gas exchange |
|
acute MI rehab/edu
|
strenthen the myocardium
phase 1 - get them up and ambulate. how to check pulse p2 - monitored situation. 12 wks. wear tele unit when in 3xwk p3 - outpatient not monitored p4 - maintenence |
|
cardic sx goals
|
repare, reconstruct, substitute
imporve cirucaltion and cardiac function |
|
preop nursing care cardiac sx
|
health hx/physical psychosocal assessment
reduce fear and anxiety discuss complications patient teaching -be clear about what to expect |
|
what to expect after cardiac sx
|
tubes
pace maker wires stockings dangle soon pain chest binder foley peripheral edema |
|
complications post op cardiac sx: caridac
|
hf
cardio shock mi dysrhyth pericardial effusion cardiac tampnade cardiac arrest |
|
complications post op cardiac sx; pulmo
|
pulmonary edema
PE pleural effusions pneumothorax hemothorax resp failure ARDS |
|
permanent pacemaker
|
placed beneath skin in pectoral region
lead is tunnelled subq between pacemaker and external jugalr vein pacemeaker lead enters external jugalr vein |
|
placement of pacemaker is done
|
during surgery under flouroscopy
|
|
batteries in pacemakers need to be changed
|
q10 yrs
outpatient |
|
external temp pacemaker
|
electrodes stuck to chest attatched to defrilator that is on at constant rate. defibr turned up until heart responds to electircal
|
|
universal lettering
|
5 letters
3 most common |
|
first 3 letter on pacemaker
|
signifies the chamber or chambers being paced
av or d |
|
second letter in pace
|
describes the chamber or chambers being sensed by the generator
|
|
third letter in pace
|
type of response by the pacemaker to what is sense
inhibited triggered dul or none |
|
fourth/fifth letter in pace
|
used for permanent pacemakers and generators
fourth has the ability to program or reset the device. fith indicates the anti tachycadia and or defibrillation capability |
|
complications of pace maker insertion
|
local infection
bleeding hematoma -localized pain at the site of insertion hemothorax dislocation of leads phrenic nerve stimulation and hicupping ventricular ectopy tachycard (irritation of centrical wall) movement of lead |
|
patient teaching with pace maker insertion
|
no airport metal detector
no heavy lifting no pulling activity no raising over head identifying |
|
post operative complications of pacemaker implant
|
hemorrhage or coagulopathy
stroke renal failure hepatic failure inection sepsis hypothermia |
|
infectious cardiac disorders
types |
rheumatic endocarditis
infective endocarditis myocarditis pericarditis -sub acute, acute, chronic |
|
medical management of infectious cardiac disorders
|
eradicate infection
maximize cardiac output promote comfort avoid nsaids |
|
which microbe can effect cardiac through infection
|
strep
|
|
nursing problems for infectious cardiac disorders
|
activity intolerance
acute pain decreased cardiac output ineffective health maintencne ineffective tissue perfusion risk for imbalanced nutrition |
|
valvular diesea and septal defects
types |
mitral
aortic tricuspid ulmonic |
|
valvular disease and petal defects pathophys
|
prolapse
regurgitation stenosis ruptured papillae related to infection or damange with disease congenital defects |
|
clinical manifestations of valvular disease/septal defects
|
activity intolerance
murmer fatiuge sob syncope palpatations chest pain |
|
tx valvular disease/septal defect
|
sx to correct
atb to prevent infxn during procedures |
|
mardiomyopathy
types |
dilated
hypertrophic restrictive series of events that end up in a decreased cardiac output |
|
dilated myopathy
|
most common
pregnant women etoh intake viral infections that cause great deal of stress on theart |
|
hypertrophi myopathy
|
inherited condition
|
|
medical management of myopathy
|
correct heart failure
implantable cardioverter defib, pacemaker infective edocarditis prophylaxis alcohol septal ablation systemic anticoagulation |
|
sx managment of myopathy
|
heart transplatation
mechanical assist devices artificial hearts |
|
HF etology/risk factors
|
abnormal loading conidtions
abnormal muscle function conditionas that precipitate heart failure |
|
causes of HF, chronic
|
CAD
hypertensive heart disease rheumatic /congenital heart disease disease of heart valves -valves can be leaky or overly stiff cor pulmonale/pulmonary hypertension cardiomyopathy anemia damage to hear from etoh/other drugs |
|
acute causes of hf
|
acute mi
arrhythmias PE thyrotoxicosis/hypothyroidsm HTN crisis rupture of papillary muscle ventricular septal defect myocarditis |
|
HF pathophys
|
ventricular dilation
increased sympathetic nervous system stim stim of RAAS decompensation heart failure |
|
HF class
|
high/low ouutput failure
left/right acute/chron back/forward |
|
HF med management
|
improve ventric pump
reduce workload reduce fluid retention reduce stress/risk of injury |
|
HF sx manage
|
ventricular assist device
heart transplant |
|
dx of hf is made when ejection fraction is less that what %?
|
40-systolic heart failure
|
|
when the ejection fraction is >40 but symptomatic it is called
|
diastolic heart fialure
|
|
factorsa aggravating HF
|
ischemia/infart
sodium excess excess fluid med noncompliance arrthmias intervurrent illness conditions associated with increased metabolic demand |
|
left vent dysfunction s/s
|
DOE
paroxysmal nocturnal dysnea tachy/palpitations cough/wheeze/hemoptysis AMS rales pulmonary edmea S3 oliguria decreased tissue perfusion, peristalsis, abosorption |
|
right sided HF s/s
|
abd pain
anorexia nausea bloating/depend edmea swelling nocturia decreased frequency |
|
physical s/s right sided hf
|
periphreal edema
venous engorgement JVD abd jugular reflux hepatomecaly ascites murmurs AMS |
|
compensated HF
|
whenc ompensatory mech are successfull at maintaining cardiac output need for metabolic demands and tisue perfusion
|
|
general measures for lifestyle mod in HF
|
weight reduction
stop smoking avoid ETOH daily weight exercise nutrition therapy -reduced sodium intake |
|
medical considerations for HF
|
treat HTN, hyperlipidema, diabetes, arhythmias
coronary revascularization anticoag immunization cose outpatient monitoring reduce readmissions |
|
outcomes and interventions
|
maintina fluid balance
decrease anxiety improved qual of sleep adhere to regimine stop smoking stop ETOH daily weight na restrict ortho hypo |