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20 Cards in this Set
- Front
- Back
When blood flow to the the lungs is decreased due to pulmonary stenosis and a VSD is present a ? to ? shunt occurs mixing unoxygenated blood with oxygenated blood and these are two parts of which cardiac congenital defect of infants ?
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Right, Left,
Tetralogy of Fallot |
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When blood flow is impeded through a constricted area of the aorta near the ductus arteriosus, ? is increased behind the defect. This is known as ? The pressure is higher in the ? extremities than in the ? extremeties. ?,?,? pulses are bounding while pulses in the legs are ? or ?.
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pressure,
coarctation of the aorta, carotid, brachial, radial, weak, absent |
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Coarctation of the aorta increases ? ventricle pressure this results in ? of this ventricle, b/c this ventricle has to work harder to pump blood. As a result of the pressure and extra blood remaining in the heart ? may be the result.
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LEFT,
hypertrophy, CHF |
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The congenital defects that cause Tetralogy of fallot include ? , aorto positioned over the ? defect, pulmonary ? and hypertrophy of the ? ventricle. Use the acronym Vacations Are Pretty Helpful(V?A?P?H?) to remember the 4 characteristics of Tetralogy of Fallot.
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ventricular septal defect,
ventricular, stenosis, right. VSD, Aortoa positioned over VSD, Pulmonary Stenosis, Hypertrophy of right ventricle |
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With Tetralogy of fallot: If venous blood from the right ventricle flows through the septal defect and into the overriding aorta and blood flow to the lungs is diminshed b/c of the narrowed pulmonary artery, which reduces oxygenated blood returning to the heart for systemic oxygenation, ? will occur and the pulse-Ox reading will be ?
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cyanosis,
low |
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When a neonate has cyanosis that is not caused by respiratory distress the RN will be thinking that the cyanosis may be the result of a ? anomally. If the RN puts the pt on O2 it will not improve the infants ?. The Cyanosis increases with activities such as ? or ? Infants that do not have cyanosis may still present with a ? color, ? or ?.
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Cardiac,
color, crying, eating, gray, mottling, pallor |
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The RN reads the report from the Echocardiogram and it says that there is a hypertrophy of the right venticle, pulmonary stenosis, and venous blood flow through the VSD into the overriding aorta. The RN believes that this is what is causing the pts. ? do to the congenital cardiac defect ?
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cyanosis,
Tetralogy of Fallot |
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When the positions of the aorta and pulmonary artery are reversed this is called ?
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transposition of the great arteries.
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With transposition of the great arteries the Aorta carries ? blood from the right ventricle back into general ? and the ? artery returns oxygenated blood from the left ventricle to the ? So the venous blood is not getting ? and the arterial blood is staying ? The only way the infant can survive is if there is another source for mixing ? and ? blood. e.g a ? which may be kept open by the pt receiving meds such as ?-? until surgery.
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venous, circulation,
pulmonary, lungs, oxygenated, oxygenated, oxygenated, venous, PDA, indamethacin- (NSAID)prostaglandin inhibitor |
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If a baby is IUGR it doesn't mean that they fit into a ? category. If they only meet 1or 2 of the SGA requirements then they are considered ? which means the babies are just small and in a lower category of ?
The baby must meet all 3 criteria to fall into the SGA category and the categories are ?,?,? under the 10th percentile. SGA babies can be ?,?,? |
SGA,
IUGR, AGA, head circumference, weight, lenght preterm,term, post-term |
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Poor uteral placental function can be the result of 4 uterus specific things, name them ? placenta, small ?, ?, or ? may interfere with fetal growth. Illness in the expectant mother, such as ? and ? restrict uterplacental blood flow and decreases fetal growth.
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aging, small size, separation, malformation,
preeclampsia, severe diabetes |
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The % of babies that are born SGA with congenital anomalies is ?%
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35%
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Maternal condition risk factors that can cause IUGR and SGA include, R-O-Y-D-S-L-A-C
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R-renal disease,
O-older>40yrs, Y-young<17yrs, D-drug exposure, S-smoking, L-Lupus, A-Anemia, C-cardiorespiratory disease |
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Fetal condition risk factors that can cause IUGR and SGA, M-I-C-H
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M-multiple gestation,
I-intrauterine infection(toxoplasmosis, Rubella, CMV), C-chromosomal abnormalities, H-heart disease |
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Uterine Condition risk factors that can cause IUGR and SGA, C-D-A-D
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C-chronic hypertension,
D-decreased uteroplacental blood flow, A-Arteriosclerosis D-diabetes mellitus |
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Placental condition risk factors for IUGR and SGA,
P-I-C-A |
P-placenta previa,
I-infarctions, C-chorioamnionitis, A-abruptio placenta |
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Environmental condition risk factors for IUGR and SGA,
H-X |
H-high altitude,
X-ray |
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Nsg Dx- high risk for ?
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infection
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Measurements of ?,?,? are taken to see what percentile that an infant fits into on the growth chart.
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weight,
length, head circumference |
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Nsg Dx for SGA/IUGR= High risk for alteration in ? perfussion.
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tissue
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