Apoptosis

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  • Neuroblastoma And Microrna-34a

    Neuroblastoma and microRNA- 34a is commonly mentioned together in many articles and studies around the world. With the advance in medicine and new discoveries, there has been found a dependence relation of neuroblastoma with these specific type of microRNA since it has been discovered how it could eliminate cancer eternally. MicroRNA-34a has not only revolutionized scientists but the world in general because of its ability to destroy neuroblastoma but also because is a new natural genetic regulator of a tumor suppressor. Its level of dominance allows it to control different processes like apoptosis, metastasis, and differentiation in the cell cycle. MicroRNAs have taken control over diverse organisms, not only humans but also plants…

    Words: 2340 - Pages: 10
  • Epilepsy Case Studies

    sensory, autonomic or psychic behaviours (Eucare 2003). Seizures in epilepsy whether they are brief or repetitive can lead to neuronal and cell death in the brain regions might be the cause for further seizures in epilepsy (Henshall David et al., 2010). Cell death or apoptosis takes place by either extrinsic or intrinsic pathway and intrinsic pathway was thought to be the most relevant in explaining neuronal death mechanisms in epilepsy (Youle et al., 2008). The intrinsic pathway is mainly…

    Words: 2305 - Pages: 10
  • Alkylated Polycyclic Aromatic Cells: The Effects Of PAH?

    2001). It has been suggested that effects of ROS on human cells may induce the oxidative injury leading to apoptosis (Salganik 2001). To study the relationship between ROS production and apoptosis, BaP-, DBT-, and Ret-induced ROS production in SK-N-SH cells was measured following exposure to CM-H2DCFDA, which forms a fluorescent oxidation product in the presence of ROS. Fig. ….shows the ROS levels after exposure to BaP, DBT, Ret, and H2O2 (positive control). A dose-dependent significant increase…

    Words: 934 - Pages: 4
  • JNK Biochemistry

    JNK is one of the three branches of MAPK superfamily of serine/threonine protein kinases. Other members of this superfamily are p38 kinases and the ERKs (Kyriakis and Avruch, 2012). In mammals, there are 3 JNK genes: Jnk1, Jnk2, and Jnk3 on 3 different chromosomes, and each mammalian JNK gene has alternative splicing forms (Derijard et al., 1994). JNK is involved in many physiological and pathological processes. The JNK pathway plays a major role in apoptosis. JNK pathway is required for…

    Words: 993 - Pages: 4
  • The Effects Of Lead In The Body

    oxygen species from the body. Lead’s binding to Glutathione inactivates the enzyme and makes it unavailable to combat ROS formed in the system. Through the generation reactive oxygen species (ROS) and the inhibition of antioxidants similar to Glutathione that have sulfhydryl functional groups, Lead diminishes the human body’s ability to relieve itself from the accumulation of oxidative stressors.**** TOXICITY IN CANCER CELLS Studies suggest a mechanism for lead-induced apoptosis of Cancer cells…

    Words: 1364 - Pages: 5
  • Acetylcholine Enzyme Report

    that have specific effects on the interactions and communications between cells (Zhang & An, 2009). Pro-inflammatory cytokines induce inflammation by activating the Nuclear Factor Kappa B (NF-κB) signaling pathway. NF-κB is a DNA transcription factor which, when activated, will transcribe pro-inflammatory genes into a cell’s DNA. The dysregulation of the NF-κB signaling pathway is responsible for various chronic inflammatory diseases, including IBD. Inflammation is an immune process that…

    Words: 2240 - Pages: 9
  • Necrosis Research Paper

    Necrosis occurs as a result of external injury due to cellular injury, bacterial toxin or trauma to a particular area of the body. Necrosis differs from apoptosis in a couple of ways. First, the morphological changes that a cell undergoes are different in necrosis compared to apoptosis. Secondly, apoptosis is highly regulated, however, necrosis is unregulated. The morphological changes that a cell undergoes during necrosis are the swelling of the cytoplasm, irreversible damage to the plasma…

    Words: 817 - Pages: 4
  • Kaempferol Research Paper

    inactivation of PI3K signaling pathways and the establishment of the mitochondrial apoptotic program via increase of Bax, decrease of Bcl-2, release of cytochrome c, caspase-3 activation as a consequence of which cell death may result. Kaempferol induced apoptosis via endoplasmic reticulum stress and mitochondria-dependent pathway in human osteosarcoma U2OS cells. Effects of Kaempferol in vivo are evaluated in BALB/cnu/nu mice inoculated with U-2 OS cells; indicate inhibition of tumor growth by…

    Words: 2081 - Pages: 9
  • Breast Cancer Argumentative Essay

    Some can halt the growth of the tumor inside our body; some can prevent its spread etc. Today I would like to highlight the benefits of eating pomegranate and its effects on breast cancer. A normal cell has a tendency to divide as many times and then stop by a programmed cell death (apoptosis) when they are no longer needed; however a cancer cell loses its tendency to stop dividing, to attach to other cells, and to die by apoptosis. One of the reasons of breast cancer is the mutation in some…

    Words: 800 - Pages: 4
  • Mutant SOD1 And Pro-Apoptotic

    Some experiments were aimed at finding how SOD1 becomes pro-apoptotic (promoting programmed cell death) due to the fact that healthy SOD1 are against programmed cell death (Pasinelli et al. 2004). The pro-apoptotic characteristic of mutant SOD1 is demonstrated in vivo and in vitro. The mitochondria inside cells firmly control apoptosis, and the mutant SOD1 that aggregates inside mitochondria triggers the programmed cell death of motor neurons (Pasinelli et al. 2004). One experiment studied…

    Words: 960 - Pages: 4
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