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47 Cards in this Set

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Iron Deficient Anemia
A decrease in the number of red cells in the blood caused by too little iron.
Iron Deficient Anemia: Causes
Rapid growth
Insufficient dietary iron (too much milk/formula)
Blood loss
Iron Deficient Anemia: Clinical Manifestations
Pallor
Tachycardia
Systolic Murmurs
Irritability
Restlessness
Iron Deficient Anemia: Diagnosis
CBC with Diff
Retic count
Iron studies (Hgb 7-10=> Anemia)
Iron Deficient Anemia: Treatment
Decrease Formula
ELIMINATE Milk (<8oz/day)
Iron Supplementation (6mg/kg/day elemental iron)
Iron Deficient Anemia: Nursing Considerations
Education
-Iron admin. in back of mouth
-Stools become black and hard
-Family influence (don't have milk in home)
Sickle Cell Disease
Autosomal recessive trait resulting in abnormalities of the hemoglobin molecule. Sickling occurs with de-oxygenation.
Sickle Cell Disease: Complications
Result from occlusion of blood vessels from sickled cells
-Vaso-occlusive crisis
-Splenic Sequestration
-Aplasia
-CVA
-Acute Chest Syndrome
-Priapism
Sickle Cell Disease: Acute Chest Syndrome
Leading cause of death in SCD pts. >10
Sickle Cell Disease: Symptoms of Acute Chest Syndrome
Fever
Chest/back pain
Decreased O2 sat
Cough
Dyspnea
Sickle Cell Disease: Treatment of Acute Chest Syndrome
IV antibiotics/Steroids
Pain/Fever control (NSAID's)
Maintenance fluids
Monitoring
Bronchial Lavage
Exchange Transfusions
Sickle Cell Disease: Vaso-Occlusive Crisis
Acute episodes of severe pain that can occur anywhere in the body. May be isolated or in multiple sites.
Sickle Cell Disease: Treatment of Vaso-Occlusive Crisis
Hydration
Pain Management:
-Opioids
-NSAID's
Sickle Cell Disease: Priapism
Persistent, painful , unwanted erection. May be stuttering or prolonged.
Sickle Cell Disease: Treatment of Priapism
Hydration
Pain Management
Irrigation of the copora cavernosa
Exchange transfusion
Avoid temp. extremes
Sickle Cell Disease: Splenic Sequestration
Infarction of the spleen at the microvascular level. The spleen becomes enlarged by trapping the abnormal RBCs.
Sickle Cell Disease: Symptoms of Splenic Sequestration
Rapidly enlarging abdomen w/ L side pain
Dyspnea/cardio collapse/shock
Pallor
Weakness
Sickle Cell Disease: Treatment of Splenic Sequestration
ST:
-Restore Cardiovascular volume
LT:
-Chronic transfusion program
-Splenectomy
Sickle Cell Disease: Aplasia
Temporary cessation of bone marrow function. Usually follows an infection.
Sickle Cell Disease: Treatment of Aplasia
Supportive care
-Packed RBC transfusions
-Treat infection
Sickle Cell Disease: Infection
Leading cause of death in SCD. Most commonly caused by Strep and H. influ.
Sickle Cell Disease: Prevention of Infection
Prophylaxis
Young children:
-125mg BID amoxicilian
Older children:
-250mg BID amoxicilian
Sickle Cell Disease: Nursing considerations
Avoid hypoxic situations
Folate supplementation (1mg/day)
Genetic Counseling
Hemophilia
X-linked congenital bleeding disorder. Protein defficiency effects normal clotting.
Hemophilia A
Factor VII deficiency. 85% of all hemophilia
Hemophilia B
Factor IX deficiency
Hemophilia: Manifestations
Excessive prolonged bleeding
Hemarthrosis
Hemophilia: Treatment
Factor replacement therapy
Treat at first sign of bleed
-RICE
Prophylactic use of factor
Hemophilia: Nursing Considerations
Family teaching
Genetic counseling
Immune Thrombocytopenia Purpura
Acquired hemorrhagic disorder resulting in excess destruction of circulating platelets.
Immune Thrombocytopenia Purpura: Clinical Manisfestations
Petechiae/purpura
Decreased circulating platelets
Healthy looking child
Immune Thrombocytopenia Purpura: Treatment
Observation
Steroids
-only given after bone marrow biopsy
IVIG
WinRhO
-must be Rh+
Slenectomy (rare)
Henoch-Schonlein Purpura
IgA mediated vasculitis
Henoch-Schonlein Purpura: Clinical Manifestations
JARS
-joints (arthritis)
-abdominal pain
-renal (hematuria, proteinuria)
-skin (purpura)
Henoch-Schonlein Purpura: Treatment
Steroids
Fluids
Blood transfusions
Cancer: Treatment
Chemo
Radiation
Surgery
Bone Marrow Transplant
Leukemia
Unrestricted proliferation of immature white blood cells in the blood forming tissue.
Leukemia: Diagnosis
Blood Smear
Bone Marrow
-Both reveal high number of immature cells or blasts.
Leukemia: Clinical Manifestations
Present with:
-fever
-pallor
-bone/joint pain
-fatigue
-anorexia
-hemorrhage
Anemia
Infection
Bleeding
Hepatosplenomegaly
Leukemia: 2 Major Types
Acute Lymphoid Leukemia (ALL)
Acute Myelogenous Leukemia (AML)
Leukemia: ALL
3 sub-types (L1, L2, L3)
L1 is the most common and has best prognosis
Leukemia: AML
8 sub-types
Hodgkin Disease
Originates in the lymph system and involves the lymph nodes. Affects primarily adolescents
Hodgkin Disease: Clinical Manifestations
Presents as painless enlargement of lymph nodes (enlarged, firm, non-tender movable nodes in supra-clavicular or cervical area)
-fever
-weight loss
-night sweats
-enlarged liver
-enlarged spleen
Hodgkin Disease: Metastasize
Spleen
Liver
Bone Marrow
Lungs
Mediastinum
Non-Hodgkin Lymphoma
Similar to Hodgkin disease but is:
-diffuse rather then nodular
-cell type is undiff. or poorly diff.
-dissemination occurs earlier, more often and more rapidly
-mediastinal involvement and invasion of meninges occur
-May present as Burkett's
Burkett's Lymphoma
Rapidly growing neoplasm most commonly seen as a mass in the jaw, abdomen or orbit