Week 7 Test: Pharm, Airway, And Durg Admin

Front 1

Sympathetic "fight or flight" hormones released by adrenal glands

Back 1

CATECHOLAMINES
Most abundant are Epinephrine, Norepinephrine, and Dopamine

Front 2

Epinephrine, Norepinephrine, and Dopamine

Back 2

CATECHOLAMINES

Front 3

a medication which inhibits the postganglionic functioning of the sympathetic nervous system. Can be used as antihypertensives.

Back 3

Sympatholytic

Front 4

secreted by pulmonary and renal endothelial cells and catalyzes the conversion of decapeptide angiotensin I to octapeptide angiotensin II.[1]

Back 4

ACE

Front 5

ACE

Back 5

Angiotensin I-converting enzyme

Front 6

Controls Sodium NA+ in the blood to regulate BP

Back 6

Angiotensin II

Front 7

prevent the angiotensin converting enzyme (ACE) from producing angiotensin II. This causes a central enhancement of parasympathetic activity

Back 7

Ace inhibitors

Front 8

can induce angioedema

Back 8

ACE inhibitors

Front 9

the rapid swelling (edema) of the dermis, subcutaneous tissue,[1] mucosa and submucosal tissues

Back 9

Angioedema

Front 10

Tricyclic overdose can cause what signs and symptoms

Back 10

Hypotension, Cardiac disrythmia with wide QRS complex, and metabolic acidosis

Front 11

Drug given for suspected Tricyclic overdose

Back 11

Bicarb

Front 12

Adrenergic

Back 12

Sympathetic

Front 13

Cholenergic

Back 13

Parasympathetic

Front 14

Adrnergic neurotransmitters

Back 14

Pre ganglionic: acetylcholine
Post ganglionic: Norepi

Front 15

Cholenergic neurotransmitters

Back 15

Pre and Postganglionic:
acetylcholine (ACh)

Front 16

also referred to as anticholinergics

Back 16

parasympatholytic

Front 17

reduces the activity of the parasympathetic nervous system

Back 17

parasympatholytic

Front 18

Actions of Alpha I and II receptors

Back 18

Vasoconstriction

Front 19

Actions of Beta I receptors

Back 19

a beta-adrenergic receptor

Increase cardiac output
Increase heart rate in (SA node) (chronotropic effect)

Increase atrial cardiac muscle contractility. (inotropic effect)

Increases contractility and automaticity[3] of ventricular cardiac muscle.

Increases conduction and automaticity[3] of atrioventricular node (AV node)

Front 20

What is meant by the "consequent absolute necessity" of the atonement?

Back 20

This has to do with the manner in which redemption was met. Christ alone is uniquely qualified to secure redemption for the elect. So while it was not inherently necessary for God to save anyone, if he was to do so, because of his very nature, it had to happen this way (through the sacrifice of his son).

Front 21

SLUDGE

Back 21

Salivation,
Lacrimation,
Urination,
Diaphoresis (or Defecation), Gastrointestinal motility, Emesis

Front 22

Pathophysiology of Organophosphate poisoning

Back 22

functioning by inhibiting the action of acetylcholinesterase (AChE) in nerve cells

Front 23

Drug Tx for Organophosphate poisoning

Back 23

ATROPINE
blocks the parasympathetic nervous system, both vagal effects on the heart

Front 24

an enzyme that degrades the neurotransmitter acetylcholine.. It is mainly found at neuromuscular junctions and cholinergic synapses in the central nervous system, where its activity serves to terminate synaptic transmission

Back 24

Acetylcholinesterase
also known as AChE

Front 25

Which types of meds work on clotting cascade

Back 25

ASA
Asprin

Front 26

Inotrope

Back 26

FOC
force of contraction

Front 27

Chronotrope

Back 27

(from chrono-, meaning time)
increases heart rate

Front 28

Dromotrope

Back 28

affects the conduction velocity of the AV node