• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/30

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

30 Cards in this Set

  • Front
  • Back
Trichuris trichura
Trichuriasis- whip worm- Large Intestine, found in warm/moist areas, south east coast of United States eggs prone to desiccation.
Lifecycle- eggs released L1-L3 in environment humans ingest L3 from night soil/pollution. L4 in large intestine.
Symptoms- abdominal pain, nauseous, constipation, prolapsed rectum
Trichinella spiralis
Trichiniosis SMALL intestine of meat eating animals, low host specificity, northern hemisphere, poorly cooked pork can cause infection,
Lifecylce-L1 stage INFECTIVE, penetrates small intestine and goes into circulatory system into skeletal muscle and creates a NURSE CELL.
(1)eye, tongue, masticatory
(2) diaphragm, arms, legs
-calcifies
>>>second host ingest and becomes infected with L1 (L2,L3,L4) adult
symptoms- may be fatal with large infection
(1) 10days-2weeks becomes adult abdominal pain.
(2) MOST SERIOUS 10d-2w females produce migrating L1 inflammatory response strong swelling in fingers, eyes photophobia
(3) L1 larvae maintained in human can cause heart failure/cns damage
**can be inside and outside of a cell at once only known parasite to do that
Strongyloides stercoralis
"intestinal hairworm" subtropical, tropical (asia, africa) susceptible to desiccation.
Lifecylce- Direct life cylce adult in SMALL intestine L1 passes out with fecal material into environment and penetrates skin-circ system-lungs-trachea- swallowed- small intestine
Homogonic- female parasitic to parasitic lifecylce (parthenogenesis)
Heterogonic lifecylce- L3 larvae stage becomes L4 in soil produces L1 eggs and says in soil until produces an L1 parasitic offspring
Autoinfection- L1 hatches in digestive system penetrate SI and circ-lungs-trachea-smallI
symptoms- in SI only increase mucus secretion
Autoinfection cause biggest problem-molting in lungs destroy alveolar.
Larvae migrate to kidney-mothers milk-autoinfection
Must Have: Night soil,
fecal contamination,
H20 contact with L3 larval stage
Necator americans
New world "hookworm" found in humans SMALL intestine in tropical/subtropical areas
Ancylostoma duodenale
n/s america old world, se united states adult in humans hookworm
Ancylostoma brazilience
dogs "creeping eruption" hookworm
Ancylostoma caninum
dogs cats, cause death quickly to dogs and cats also creeping eruption "cutaneous larval migrans" hookworm
Uncinaria
cold weather hookworm Canada N. America Europe, N. Europe, hookworm
Hookworm info
egg-passed-L1 and L2 free living- L3 penetrates skin-lungs-circ-traq- small intestine
no autoinfection
seals get hookworm (TRANS-MAMMARY TRANSMISSION)
-feed on blood anterior cuticle and cut tissue, live 15 years. inhibit development anemia ,lethargy, swollen abdomen, less muscle cross eyed directly related to blood loss.
Ascaris lumbricoides
Round worm world wide distribution MOST COMMON NEMATODE IN AGRICULTURAL SETTING. humans only host, with poor sanitation
-lifecylce- adult in small intestine- produces egg (WARTY) L1 or L2 with egg ingested by humans, circ system, molts in lungs, goes up trachea L4,
-lots of eggs laid in small intestine
-WARTY egg resistant to chemicals
-lungs damaged with eggs in lungs ASCARIS PNEUMONIA fatal
-caloric deficiency/PROTEIN deficiency
-VERMINOUS INTOXICATION
-intestinal blockage gangrene
-peritonitis
-appendicitis
-destroy liver
-migrate to nose anus mouth
Ascaris suum
exactly like human round worm only in pigs, impacts size potential
Toxocara canis
dogs definitive host
Toxocara cati
cats definitive host
Lifecycle- puppy/cat small intestine-eggs-L2 infective stage- small intestine- lungs molts-up trachea- small intestine
ADULT- L2 larvael stage will hatch in smalli migrate from lungs and remain all of life- if pregnant blasts through placenta or mammary gland- TRANS-PLACENTAL TRANSMISSION
Any TRANSPORT HOST-can pick up L2 larvae, mice, chickens, rodents.

-effects growth, same symptoms, can be fatal, liver failure, visceral larval migrans
Enterobius verminicularis
only humans- pinworms DESCENDING COLON of large intestine.
-lifecylce- large intestine- adult female produces eggs- lays them in peri-anal skin- humans ingest extracellular not much damage.
-eggs prone to desiccation
Dracunculus medinensis
Guinea worm- female found underneath skin-legs-scrotum-long lived takes a year to show infection (AFRICA/MIDDLE EAST)
Life-cycle- female in subcutaneous-produces blister-ruptures when H20 touches it-dries out and goes back into sore-L1 larvae ingested by copepod,L2-L3 stage in copepod, humans ingest with L3 stage-penetrate small intestine male dies but female goes to skin-

split twig method-match stick method
go into anaphalectic shock and die
Filarids- Filariasis
humans/domestic animals, circ system, lymphatic, digestive, intermediate host blood sucking insect
Life-cycle- 1st stage microfilaria specialized to stay in host unrecognized
general- adult def. host- microfilaria produced- skin surface-ingested by insect blood feeding- L1,L2,L3 infects new definitive host while insect feeds by saliva pool and penetrates into skin becomes adult
Dirofilaria immitis
dogs/cats not normal host bc doesn't produce microfiliara found in East of the Mississippi, Oregon and Washington
-right atrium of canids
normallyheavy- problems with valves interferes with heart function
-blood flow blocked-fatigue
Hypertrophy- growing of muscle
10-20 worms kills dogs 1-2 kills cats
-if dog is excited the worms can flow into lungs and kill dog that way
Wuchereria bancrofti
lympatic filariasis elephantitis
more severe
-global, asia,africa
Brugia malai
lymphatic filariasis
arms legs scrotal sack, boobs,
takes 30 years for symptoms to appear
S.e. Asia pacific islands
3 phases-
(1) asymptompatic- normal immune response microfilaria but not rejecting
(2) inflammatory phase- well into 20's 30's hyper immunogenic response to microfilaria
(3) acute phase- lymphatic accumulation, permanent skin loses epithelial tissue, keep bacteria out of skin can survive
-vector prefers stagnant water
Onchocera volvulus
Onchoceriasis (river blindness or hanging groin)
Adults found beneath the skin connective tissue forms, NODULE called the ONCHOCERCOMA head is river blindness and groin is hanging groin.
life cycle- adult in dermis- microfilaria-- SIMULIUM- L2,L3L,L4 in insect- enters definitive host adults develop. Becomes adult contained in Onchocercoma produce microfilarai that go to skin.
Blindness- onchocercoma - microfilaria migrate to eye and cause scarring, tissue + wolbachia cause blindness.
found in- Africa- central america
FAST MOVING water.
Trypanosoma brucei brucei
african ungulates
Trypanosoma brucei gambiense
c.w. africa chronic
Trypanosoma brucei rhodesiense
eastern africa- acute phase
Trypanosoma cruzi
chagas disease south america metacyclic trypomastigote
Leishmania tropica
cutaneous- india/asia/africa
Leishmania braziliensis
mucotaneous c.s america
Leishmania donovani
visceral- africa, middle east
Schistosoma mansoni
africa- large mesenteric vein to large intestine
Schistosoma japonicum
asia- small intestine mesenteric vein
Schistosoma hematobium
africa/middle east urinary bladder