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115 Cards in this Set
- Front
- Back
Describe c/s flexion glide and rotation. |
Superior glide of facet joint. Anterior rotation of vertebral body |
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Describe c/s extension glide and rotation. |
Inferior glide of facet joint. Posterior rotation of vertebral body |
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Describe c/s side flexion glide and rotation. |
Inferior glide ipsilateral side. Superior glide contralateral side. Rotation is combined and is ipsilateral |
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Describe c/s rotation glide. |
Inferior glide ipsilateral side. Superior glide contralateral side. Side flexion is combined ipsilaterally |
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What are three types of congenital cervical spine disorders? |
(1)Congenital Torticollis, (2)Klippel-Feil Syndrome, (3)Cervical Rib (Thoracic outlet syndrome—TOS) |
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What are two types of traumatic cervical spine disorders? |
(1)Whiplash, (2)Acute Torticollis |
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What is another name for congenital muscular torticollis? |
Wry neck |
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What is the cause of congenital muscular torticollis? |
Exact cause unknown. At least 40% of infants who have difficult deliveries → wry neck. Deformity minimal at birth. |
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How do infants with congenital muscular torticollis present? |
First few weeks a large and firm swelling develops in one sternocleidomastoid (SCM). Swelling gradually disappears but left with a contracture of SCM. |
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How does congenital muscular torticollis progress? |
Head becomes laterally flexed toward the affected side and rotated toward the opposite side. Progressive facial asymmetry (due to contracture of SCM). |
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Do infants with congenital muscular torticollis have any other conditions? |
20% of all infants with condition have dysplasia of one or both hips |
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How is congenital muscular torticollis treated? |
Early recognition and daily stretching - one year can reverse condition in 90% of children affected |
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What is acute acquired torticollis? |
Painful unilateral shortening or spasm of neck muscles resulting in an abnormal head position |
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What are five causes of acute acquired torticollis? |
(1)Activation of latent trigger points, (2)Subluxation of C1 or C2 due to trauma such as whiplash or sudden turning of the head, (3)Facet joint irritation, (4)Infection, (5)Disc related pain |
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What is spasmodic torticollis? |
Spasmodic torticollis is a localized dystonia (abnormal tonicity of muscle) resulting in an involuntary spasm of cervical muscles and an abnormal head position |
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What are three causes of spasmodic torticollis? |
What are three causes of spasmodic torticollis? |
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What is another name for Klippel-Feil syndrome? |
Synostosis of the cervical spine |
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What is Klippel-Feil syndrome? |
Failure of segmentation results in congenital fusion of several cervical vertebrae. Neck normally short and very stiff. |
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How may patient with Klippel-Feil syndrome present? |
Posterior hairline is low and transverse. Head is usually straight but occasionally tilted to one side (resembling Torticollis). A congenital high scapula may exist. Sometimes have a bilateral soft tissue web from mastoid process to shoulder. |
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What is the most common cause of TOS? |
cervical rib disorder |
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What is cervical rib disorder? |
Cervical rib (extra rib) arises from C7 transverse process. Rib narrows interval between scalenes creating a higher barrier for nerves and vessels to pass over. |
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What two things worsen the compressions of cervical rib disorder? |
(1)Shoulder sag (in elderly or muscle weakness), (2)Carrying heavy object in hand |
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Which area of the c/s is most commonly affected by cervical rib disorder? |
Levels C8 to T1 most commonly affected |
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What are four symptoms of cervical rib disorder? |
(1)Affects hand and inner forearm, (2)Pain and Paresthesia along Ulnar Nerve, (3)Hand weakness, numbness, and clumsiness, (4)Associated symptoms may be variably present (Hand cold sensation & gangrene) |
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What are five signs of cervical rib disorder? |
(1)Palpable cervical rib, (2)Tender Brachial Plexus distribution, (3)Muscle weakness and atrophy (lower trunk), (4)Sensation decreased, (5)Circulatory insufficiency |
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Which two muscle groups are affected by weakness and atrophy with cervical rib disorder? |
(1)Interosseous muscles, (2)Hypothenar muscles |
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What three areas can experience decreased sensation with cervical rib disorder? |
(1)Ulnar forearm, (2)Arm, (3)Ulnar 1.5 fingers |
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What four areas can experience circulatory insufficiency with cervical rib disorder? |
(1)Swelling, (2)Cold sensation, (3)Distal cyanosis, (4)Trophic skin change |
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What are four types of acquired cervical spine disorders? |
(1)Degenerative, (2)Nerve Root Compression, (3)Vertebral Artery Compression, (4)Rheumatoid Arthritis |
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What are three types of degenerative cervical spine disorders? |
(1)Osteoarthritis, (2)Degenerative Disc Disease, (3)Disc herniation/protrusion |
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What are three additional names for osteoarthritis of cervical spine? |
(1)cervical spondylosis, (2)degenerative disc disease, (3)degenerative joint disease |
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What part of the neck does cervical spondylosis affect? |
Involves degeneration of the facet joints and discs. Affects the C5/6 and C6/7 segments primarily. Upper-mid cervical spine (C1-4 rarely affected with OA) |
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What is the incidence of cervical spondylosis? |
Is very common in adults over 45 years |
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What are causes of C5-7 segments being most commonly affected with cervical spondylosis? |
(1)Maximal lordosis, (2)Most of flexion/extension occurs here, (3)Poor posture |
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What are two causes of nerve root impingement in cervical spondylosis? |
(1)Narrowing of intervertebral foramina, (2)Disc herniation |
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T/F: Most persons over 50 years have degenerative changes on x-rays |
True |
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How may a patient with a degenerative c/s present? |
Some have mild symptoms such as stiffness and others have severe neck pain and arm pain +/- paresthesia. |
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T/F: All nerve roots compressed give the same signs and symptoms |
False, specific nerve roots compressed give different signs and symptoms |
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What are four specific signs/symptoms of a compressed nerve at C6? |
(1)Weak deltoid, biceps + wrist extensors, (2)Decreased brachioradialis reflex, (3)Decreased skin sensation thumb and index finger, (4)Paresthesia C6 dermatome |
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At what age is blood supply cut off to the intervertebral discs? |
Age 20 |
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Why are nerve roots at C5-8 more likely to be compressed? |
They are larger |
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What is the most commonly compressed nerve root? |
C7 |
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What is radiculopathy? |
Compression of nerve root at intervertebral foramen |
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Where is the pain of radiculopathy felt? |
Radiating pain is felt along the affected nerve root |
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What can cause radiculopathy in the c/s? |
osteophytes or discherniation |
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Which nerve root will a disc herniation at C5-6 affect? |
C6 |
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Which nerve root will a disc herniation at C6-7 affect? |
C7 |
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Where is radiculopathy paresthesia experienced? |
Paresthesia may be felt in one or two digits innervated by the nerve root or slight clumsiness or weakness of the hand |
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Are the affects of radiculopathy paresthesia sensory, motor, or reflex? |
All three |
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Which nerve roots are most commonly affected by radiculopathy paresthesia? |
6th and 7th |
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60 - 70% of radiculopathy paresthesia occurs in which nerve root? |
7th |
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How can cervical myelopathy develop? |
Cervical spondylosis can progress to cause spinal cord compression |
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What may patients with cervical myelopathy present with? |
Patients usually present with difficulty in walking with slight unsteadiness of one or both legs. Legs feel stiff and heavy and can ‘give out’ quickly on activity or exercise. The patient reports tingling and numbness of the hands and/or feet. |
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How do the symptoms of cervical myelopathy begin and progress? |
Symptoms began insidiously and progress slowly |
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T/F: Fine hand movements may be impaired with cervical myelopathy |
True |
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Is pain associated with cervical myelopathy? |
No, there is usually no pain |
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What are five signs/symptoms associated with cervical myelopathy? |
(1) Increased frequency or dribbling incontinence, (2)Increased muscle tone in lower extremities, (3)Babinski positive, (4)Vibratory and tactile sensation may be impaired from hip down, (5)Flexion of neck may cause electric-like sensation down the spine/limbs (Lhermitte’s sign) |
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What is the vertebral artery? |
It is the main blood supply for the brainstem nuclei but also supplies many structures along its path |
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Where does the vertebral artery originate? |
Comes off the subclavian artery |
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What is the route of the vertebral artery from the subclavian artery? |
It then enters the Cervical spine at C6. It ascends the cervical spine in the transverse foramina. It enters the foramen magnum and joins the other VA and forms the Basilar artery |
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What four structures does the vertebral artery supply? |
(1)Upper spinal cord, (2)brainstem, (3)cerebellum, (4)posterior part of brain |
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What could cause compression or dissection of the vertebral artery? |
Instabilities, prolonged, sudden /extreme rotation, extension or traction (i.e.Trauma: MVA, sports, falls) |
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What are eight signs/symptoms of vertebral artery ischemia? |
(1)Drop Attacks, (2)Diplopia, (3)Dizziness, (4)Dysarthria, (5)Dysphagia, (6)Bilateral or Quadrilateral paresthesia, (7)Nystagmus, (8)Perioral paresthesia |
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Describe rheumatoid arthritis |
It is a systemic inflammatory disorder affecting the synovial membrane of joints. RA is an immune response |
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What is the incidence of RA? |
It is relatively common (1.5% of the adult population). Women:men = 3:1. |
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What is the peak period of onset of RA? |
Peak period of onset is between the ages of 40 and 60 years (but can occur at any age). |
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What is the incidence of RA in First Nation people? |
First Nation people have 5-6% incidence. |
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How does RA develop? |
Remains a mystery. Could be related to some bacterial or viral infection, Vitamin deficiency, Hormonal imbalance, Emotional stress. |
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What is the target of RA? |
The target of the disease is the synovial membrane of joints and tendon sheaths. |
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*How do the synovial membranes react to the inflammation of RA? |
The synovial membranes react to inflammation by congestion, edema, fibrin, exudation and proliferation. *The RA synovial membrane that extends to the cartilage and bone is known as pannus (“a rug”). |
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How does the pannus affect the joint? |
The pannus (fibrous tissue produced by synovial cells) interferes with nutrition of the articular cartilage and causes cartilage necrosis. |
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Describe the four steps of degeneration with RA. |
(1)Chronic inflammation of synovium, (2)Decreased movement of joint, (3)Pannus formation, (4)Erodes cartilage and bone. |
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What will happen if the pannus burrows into the subchondral bone? |
It will cause osteolysis in the bone. |
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What are three causes a joint to become unstable and sublux (a slight misalignment of the vertebrae)? |
(1)Articular instability, (2)Tendon pathology, (3)Ligament laxity |
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*What do 30% of RA patients develop? |
Rheumatoid nodules - non-tender subcutaneous bump on exterior surface. |
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T/F: Tenosynovitis is present in the majority of patients with RA. |
True |
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What are the seven most commonly involved joints with RA? |
(1)Metacarpal, (2)Wrist, (3)Shoulder, (4)Knees, (5)Subtalar, (6)Metatarsal, (7)Craniovertebral region (C1/2) |
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What are three systemic symptoms of RA? |
(1)Systemic flu-like symptoms (Generalized aching, Stiffness, Fatigue, Weight loss), (2)Affected joints are hot, swollen and skin over joint is shiny and tight, (3)Morning stiffness for 1 hour or more. |
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*What are seven later clinical features of RA? |
(1)C1/C2 instability & neurological symptoms (transverse ligament affected. Can cause nausea, vertigo, paralysis in UE/LE), (2)Positive Sharp Purser Test, *(3)Sjogren’s syndrome (dry eyes and mouth) 15% of those with RA (autoimmune disorder affecting saliva and lacrimal glands, skin, respiratory and GI tracts), (4)Muscle spasm (secondary to inflammation or joint instability), Muscle atrophy, (5)Muscle contracture, (6)Subluxation & luxation due to stretched joint capsule & ligaments, (7)Rupture of tendons |
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Can RA be cured? |
No, it can only be managed |
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What are seven treatment goals for RA? |
(1)Help patient understand the nature of the disease, (2)Provide psychological support, (3)Treat pain, (4)Suppress inflammatory reactions (NSAID’s, steroids, cortisone injections), (5)Maintain joint function & prevent deformity, (6)Correct existing deformity, (7)Improve function |
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What are two ways treatment goals of RA can be achieved? |
(1)External support commonly required (brace/splint), (2)Surgery is often indicated. |
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What is whiplash? |
The term ‘whiplash’ is not diagnostic but rather synonymous with the mechanism of injury. It merely implies that the cervical spine has been whipped in some direction |
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What is a better term for whiplash? |
A better term is ‘acceleration/deceleration’ injury or ‘flexion/extension’ injury |
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What are six causes of whiplash? |
(1)MVA, (2)Blows to head, face or neck, (3)Blows to the body, (4)Pulls and thrusts on the arms, (5)Blows to the top of the head, (6)Falls landing on shoulder |
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If whiplash symptoms have an immediate onset are the injuries more or less severe? |
Immediate onset typically implies more severe injuries |
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T/F: If the head is turned, whiplash injuries are less severe |
False, injuries are more severe if the head is turned |
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Are rear end collisions a hyperextension or hyperflexion injury? |
hyperextension |
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What is the first step of a rear end collision? |
Sudden acceleration of the portion of the body in contact with the seat |
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What is the second step of a rear end collision? |
The head (mass at rest) remains at rest until acted on by an external force |
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What is the third step of a rear end collision? |
The flexible neck, with its 8-12 pound weight, is initially hyperextended as the body accelerates forward |
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What is the fourth step of a rear end collision? |
The head hits the top of the back of the seat or the headrest |
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What is the fifth step of a rear end collision? |
This impact plus the reflex contraction of the anterior neck muscles starts the head in a forward motion |
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What is the sixth step of a rear end collision? |
The head continues forward until it contacts a stationary part of the car/chest and/or is restrained by the soft tissues of the posterior neck (hyperflexion) |
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Which joints are most commonly affected in whiplash hyperextension injuries? |
With hyperextension, the joints between C4-5 are compressed the most |
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Do hyperextension injuries have the best, worst, or neutral prognosis? |
Worst |
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What can decrease hyperextension whiplash injuries? |
Proper headrest height/position |
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What are 10 hyperextension injuries that can result from whiplash? |
(1)Traction of ALL, (2)Alar ligament if head rotated, (3)Avulse disc from end plate of vertebral body, (4)Compression fracture of facet joint or spinous process, (5)Posterior subluxation of facet joints, (6)Tearing of anterior muscles, (7)Vertebral artery compressed, (8)Nerve roots or spinal cord damaged, (9)Head injury (impact or contrecoup), (10)Esophagus tearing |
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Are head on collisions hyperextension or hyperflexion injuries? |
Hyperflexion |
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What is the first step of a head on collision? |
The body is thrown forward until it contacts an immovable object (seatbelt, steering wheel, dashboard) |
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What is the second step of a head on collision? |
The head continues moving forward until it hits an immovable object or is restrained by posterior cervical tissues |
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What is the third step of a head on collision? |
The head then recoils into extension |
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Which joints are most commonly affected in whiplash hyperflexion injuries? |
With hyperflexion, the joints between C5-6 are stretched the most |
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What are 10 hyperflexion injuries that can result from whiplash? |
(1)Posterior ligament sprain, (2)Posterior muscle strains, (3)Subluxation of facet joint, (4)Posterior bulge of IVD, (5)Vertebral artery traction, (6)Fractures of anterior vertebral body, (7)Nerve root or cord injury, (8)Head injury |
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Describe what happens to the body in a side collision? |
The body, head and neck are thrown first in the direction opposite the impacting force and then in the direction of the force. Rotation combines with side flexion so there is additional torque involved. If the head is already in rotation at impact the damage is usually more severe. |
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What is a rim lesion? |
A horizontal tear of the end plate |
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What type of injury can cause a rim lesion? |
Occur with hyperflexion or hyperextension injuries |
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How is a rim lesion diagnosed? |
Do not show on x-ray. MRI will confirm rim lesion. |
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How long can it take for a rim lesion to heal? |
May take 18 months to heal. |
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What are eight clinical features of whiplash? |
(1)Neck, thoracic or arm pain, (2)Headache, (3)Visual disturbance, (4)Dizziness, (5)Concentration and memory disturbances, (6)Paresthesia, (7)Weakness, (8)Swallowing difficulties |
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What percentage of patients are symptomatic at 2 year follow-up (after settlement)? |
45% |
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What percentage of patients develop early OA? |
39% |
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What percentage of MVA patients have persistent headache and dizziness for 2-4 years? |
20% |
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How is treatment determined for whiplash injuries? |
Must look at the SIN (severity, intensity and nature) of the injuries. Rule out any serious injuries. Open mouth x-rays taken?? (checks for dens fracture). |
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What are five treatments for whiplash? |
(1)Rest, (2)Collar (no longer than 4 weeks), (3)Maintain joint mobility and proper muscular healing i.e.: chiro/massage, (4)Medication, (5)Active exercise when ready |