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136 Cards in this Set

  • Front
  • Back
When osteopathy founded?
June 22 1874
What was the significance of the spanish flu pandemic?
1918
Ribraising dramatically reduces morbidity and mortality
Who was the founder of the following OMM Technique:
Cranial
William Sutherland
Who was the founder of the following OMM Technique: Muscle Energy
Fred Mitchell
Who was the founder of the following OMM Technique: Counterstrain
Lawrence Jones
Who was the founder of the following OMM Technique: Facilitated Positional release
Stanley Schiowitz
Who was the founder of the following OMM Technique: "resdiscoverd" AT Still technique?
Richard Van Buskirk
What are the 4 Osteopathic Principles?
1.The body is a unit, and represents a combination
of body, mind & spirit.
2.The body is capable of self regulation, self-
healing, and health maintenance.
3.Structure and function are reciprocally
interrelated.
4.Rational treatment is based on an understanding
of these principles: body unity, self-regulation,
and the interrelationship of structure and
function.
What does this define:
Is an impaired or
altered function of related components of
the somatic (body framework) system;
skeletal, arthrodial and myofascial
structures, and related vascular, lymphatic
and neural elements.
somatic dysfunction
What does TART stand for?
Tissue texture changes, Asymmetry,
Restricted motion &
Tenderness)
Motion within the transverse plane about a
longitudinal or vertical axis is what motion?
Rotation
Motion within the coronal/frontal plane
about a AP axis is what motion?
Sidebending
Motion within the sagittal plane about a
transverse axis is what motion?
Flex/Extension
What is a Type I Fryette?
Type I: Neutral position, sidebending and
rotation occur to opposite sides.
What is a Type II Fryette?
Type II: Non-neutral, sidebending and
rotation occur to the same side.
What Fryettes Third Law?
Type III: Motion in one plane alters motion
in other planes of motion.
Where would you find the spinous process of the T1-T3? What other level behaves this way?
T1-3 Spinous process of segment is with its
transverse process
T12 Also follows this rule
Where would you find the spinous process of the T4-T6? What other level behaves this way?
T4-6 Spinous process of segment is half
way, to t-process of segment below
T11 also
Where would you find the spinous process of the T7-T9? What other level behaves this way?
Spinous process of seg. is at level with
t-process of seg. below
T10 also
What is the motion of the OA (C0-C1)?
Motion of the occiput on the
atlas. Atypical motion. Sidebending and
rotation occur in opposite directions in a
non- neutral position
What is the motion of the AA (C1/C2)?
Motion of the atlas on the
axis. Atypical motion. Pure Rotation.
What is the motion of C2-C7?
Generally follow Fryette’s type II
motion. Non- neutral, rotation and
sidebending to the same side.
If T4 is compared with T5. We notice that T4
moves into flexion freer and sidebending and
rotation is easier to the right. How do we name
this?
T4FRSR

Rotation is listed FIRST in Type II Dysfunctions
If segments T5 through T9 have
paravertebral humping noted on the right
side and the patient is restricted with right
sidebending without flexion or extension.
How would you name this?
T5-T9NSLRR

Side-bending is listed FIRST in Type II Dysfunctions
What is active motion testing?
When the patient moves them selves - Quantitative
What is passive motion testing?
When the doctor moves the patient - Qualitative
Name the Barrier: End range of joint motion
limited by bones, ligaments, and tendons.
Passively tested.
anatomic
Name the Barrier: Limit of end range of motion
produced by the patient. Actively tested.
Physiologic
Name the Barrier: Abnormal limited
motion within the physiologic range that is
altered by somatic dysfunction. OMM
deals with this.
Restrictive/Pathological
Treatment
consists of moving into the restrictive
barrier.
Direct
Treatment
consists of moving away from the
restrictive barrier.
Indirect
What Techniques are Direct
HVLA, ME, Myofascial Release (both) Cranial on Kids
What Techniques are INDirect
Conterstrain
Cranial in adults
FPR
Still (Indirect to direct)
Which are the true ribs?
1-7 True ribs. Articulate with sternum.
Which are the false ribs?
8-10 False ribs. Articulate with cartilage
Which are the floating ribs?
11-12 Floating ribs. No anterior
articulation.
What Ribs have a Pump handle motion
1-5
AP diameter expansion with respirations.
What Ribs have a bucket handle motion?
Ribs 6-10
lateral
diameter expansion with respirations.
If ribs 5-9 are inhaled you must treat which rib?
Key rib is 9
If ribs 5-9 are exhaled you must treat
which rib?
5
Viscerosomatic Reflexes head and neck?
Head and Neck T1-4
Viscerosomatic Reflexes Heart
Heart T1-5
Viscerosomatic Reflexes Lung
Lung T2-5
Viscerosomatic Reflexes Cervical Upper Esophagus
Cervical Upper Esophagus T2-4
Viscerosomatic Reflexes Thoracic Mid Esophagus
Thoracic Mid Esophagus T3-T6
Viscerosomatic Reflexes Lower Esophagus and Stomach
Lower Esophagus and Stomach T5-T8
Viscerosomatic Reflexes Spleen and Pancreas
Spleen and Pancreas T5-11
Viscerosomatic Reflexes Liver
Liver T6-9
Viscerosomatic Reflexes Gallbladder
Gallbladder T9-10 R
Viscerosomatic Reflexes Small Intestine
Small Intestine T9-11
Viscerosomatic Reflexes Appendix
Appendix T12
Viscerosomatic Reflexes Ovary/testes
Ovary / Testes T9-10
Viscerosomatic Reflexes Kidney, Ureter, Bladder
Kidney, Ureter and Bladder ALL T10-L1
Viscerosomatic Reflexes Prostate and Urethra
Prostate and Urethra L1-2
Viscerosomatic Reflexes Cervix
Cervix L1-2
Viscerosomatic Reflexes Ascending and transverse colon
Ascend. and Trans. Colon T10-12
Viscerosomatic Reflexes genitals
Genitals T12
Viscerosomatic Reflexes Uterus
Uterus T10-L1
Viscerosomatic Reflexes Descending and Sigmoid Colon and
Rectum
Descending and Sigmoid Colon and
Rectum L1-2
Viscerosomatic Reflexes Adrenals
Adrenal T8-T10
Lower extremity viscerosomatic reflex?
LE T10-L2
What re Chapmans reflexes?
Neuro-lymphatic tissue texture abnormalities
that are reflections of visceral dysfunction or
pathology (viscerosomatic reflexes)
What do Chapmans points indicate?
Indicate increased activity of the
sympathetic nervous system.
NOT PARASYMP
Where is the Chapmans Reflex for the heart?
Cardiac- 2 nd anterior intercostal space
Where is the Chapmans Reflex for Upper respiratory infections and the Lungs?
3 rd and 4 th anterior intercostal
space
Where is the Chapmans Reflex for otitis Medai
Middle clavicle
Where is the Chapmans Reflex for sinusitis
1st Rib
Where is the Chapmans Reflex for eye pathology
Lateral Humerus
Where is the Chapmans Reflex for Colon and prostate pathology?
IT Band
Where is the Chapmans Reflex for the Appendix
Tip of right 12 th rib
Where is the Chapmans Reflex for the Rectum?
Lesser trochanter
Anterior chapmans points are for diagnosis... Where do you treat?
Can treat both anterior and posterior points
What is piriformis insertion?
GREATER trochanter
Where does illiopsoas insert?
Lesser troch
Finish the following sentence: When L5 is sidebent, a sacral oblique axis is engaged to the same side as the ____________

(chose sidebending or rotation)
Sidebending
Finish the following sentence:
When L5 is rotated, the sacrum rotates the _____________ way on an oblique axis
(choose same or opposite)
Opposite
The seated flexion test is found on the ____________ side of the oblique axis.
(choose same or opposite)
Opposite
If L5 is FRSR, what side is the seated flexion test positive on?
How is the sacrum rotates?
+SFT on the LEFT
Sacrum rotated to the left on a right oblique axis or L on R
What type of torsion is a L on L?
Forward
It would have a negative spring test indicating that the sacrum DID moce
What type of torsion is a R on L?
backwards
Spring test would be positive indicating LACK of movement
What is a shear?
The slippage of one sacroiliac joint about a
vertical axis with translation of the sacral
base.
Diagnosis?
Positive spring test
Sulcus is shallow on the same side as an
anterior ILA
Positive seated flexion test on shallow
sulcus side.
Unilateral Sacral Extension
What happens in Cranial/sacral flexion:
1. midline bones flex.
2. Paired bones externally rotate.
3. SBS rises.
4. Sacral base moves posterior / counternutation.
5. Respiratory inhalation encourages flexion.
What happens in Cranial/sacral extension:
1.Midline bones extend
2.Paired bones internally rotate.
3. SBS lowers
4. Sacral base moves anterior / nutation.
5. Respiratory exhalation encourages
extension.
Cranial Pearls -Part I
Torsion: Named for the high greater wing
of the sphenoid.
Sidebending Rotation: Named for the side
of the produced convexity. Convexity side
is rotated toward the feet.
Vertical Strain: Named for the position of
basisphenoid. Superior/inferior shearing of
the BSB.
Cranial Pearls Part II
Vertical strain associated with trauma such
as an uppercut.
Lateral Strain: Named for the position of
basisphenoid. Side to side shearing of the
BSB. Caused by trauma such as a hook.
Seen in infants with a “parallelogram
shaped head”
Compression: No motion, caused by
trauma.
What is the Spencer technique used to treat? What motion is NOT part of the technique?
Adhesive Capsulits
Not included: external rotation
Grading Deep Tendon Reflexes
What is Normal?
Grade 2 is normal

grade 0--no response;
grade 1--minimal response;
grade 2--mid-range normal
response;
grade 3--slightly hyperactive
response;
grade 4--hyperactive response
with clonus.
Muscle Strength Grading
What is normal?
5 is normal

Grade 0: Total Paralysis
Grade 1: Palpable or visible contraction
Grade 2: Full range of motion with
gravity eliminated
Grade 3: Full range of motion against
gravity
Grade 4: Full range of motion with
decreased strength
Grade 5: Normal Strength
What is a ligament injury called? Muscle injury (tendon)?
A sprain for ligament
strain=muscle
What are the Grades of Sprains?
Grade I sprain: stretch of ligament
Grade II sprain : partial tear.
Grade III sprain: complete tear.
What is O'Donahue's triad?
Pop goes the ACL, Medial meniscus, and
medial collateral ligament
Which is the most common mechanism of injury of the ACL, contact or noncontact?
non contact. a planted foot with subsequent twisting motion is the usual cause
What is the best test for a torn ACL? What other tests can you do?
Lachman is the best

Other options re anterior drawer and pivot shift
Torn Meniscus Facts
Pain often localized to the joint line and
popliteal region with knee flexion.
Locking of the joint, which prevents full
knee extension.
Buckling or “ giving out” sensation.
Knee pain often worse with full knee
flexion or extension.
Torn Meniscus Facts
Pain often localized to the joint line and
popliteal region with knee flexion.
Locking of the joint, which prevents full
knee extension.
Buckling or “ giving out” sensation.
Knee pain often worse with full knee
flexion or extension.
What tests/physical exam findings would you anticipate with a torn meniscus
Joint line tenderness with bent knee
+Apley Grind Test
+McMurray
+Pain and instability with walking, pt may "baby" leg by keeping knee bent
Knee effusion and pain with full flexion or extension
What is the most common type of ankle
sprain?
Inversion injury
What ligaments are injured with inversion? List in order of how they tear
ATF
CFL
PTF

(In that order)
What are the rotator cuff muscles?
What are the motions of each muscle?
SITS
Supraspinatous=Abduction
Infraspinatus=EX Rot
Teres MINOR= EX Rot
Subscapularis = Internal Rotation
Jobe’s Test?
Supraspinatous
Speed’s Test?
Biceps brachii
Hawkin’s Test?
Impingement
Neer’s Test?
Impingement
Yergeson’s Test?
Long head of biceps
Cross Arm Test?
AC Joint dysfunction
Patrick’s Test?
Hip Joint Dysf
Straight Leg Raise?
nerve root impingement
(
Thomas Test?
Illiopsoas
Thompson Test?
Torn Achillies
Ober’s Test?
IT Band
Stork Test?
Apondylolysis
Trendelenberg Test?
Superior gluteal nerve/Glut Med on side opposite the fallen side
Common Features of Herniated Disks:

What levles/direction?
More males or Females?
L4/5 or L5/S1 most common in posterior lateral direction (due to posterior Longitudinal Ligament)

Males more ofter and pain is worse with increasing intrathecal pressure
What are the possible treatments for a Herniated nucleus pulposus?
What imaging to diagnose
Treatment: Local heat/ice, OMT,
Corticosteroids, NSAIDS, Opioids, epidural injection, Surgery ( last resort ).
Tricyclic antidepressants and gabapentin are
often used for neuropathic pain unresponsive
to opioids.

Diagnosis: MRI, CT, Myelogram.
Connective tissue and bone overgrowth
reducing the size of the vertebral foramina =
Spinal Stenosis
Normal aging proocess most common in peopl over 60
How does spinal stenosis present?
Chronic and progressive
Bilateral and poorly locaized
Pain that RADIATES wo butt thigh or legs
Worse with Extension (standing or walking)
Better with flexion (sitting or stooping.)
Stopping ambulation may not improve sx like it would if the problem were vascular claudication
What are the risk factors for peripheral vascular disease/claudication?
Smoking, DM, hyperlipidemia, FMHX
What is the presentation of peripheral vascular disease/claudication?
Chronic, progressive
bliateral or unilateral, poorly localized
worse with any lower extremity exertion
Diminished puses, delayed cap refill and cyanotic cool extremities are classic clues on physical exam
A Separation of the pars interarticularis of the
vertebral arch =
Spondylolysis
Slippage of one vertebral body onto the next. =
Spondylolithesis
(must be lilateral for slippage to occur, most common L5/S1)
What type of injuries or athletes are most prone to Spondylolithesis?
Hyperextension injuries as seen in football lineman and gymnastic participants
Common Features of Herniated Disks:

What levles/direction?
More males or Females?
L4/5 or L5/S1 most common in posterior lateral direction (due to posterior Longitudinal Ligament)

Males more ofter and pain is worse with increasing intrathecal pressure
What are the possible treatments for a Herniated nucleus pulposus?
What imaging to diagnose
Treatment: Local heat/ice, OMT,
Corticosteroids, NSAIDS, Opioids, epidural injection, Surgery ( last resort ).
Tricyclic antidepressants and gabapentin are
often used for neuropathic pain unresponsive
to opioids.

Diagnosis: MRI, CT, Myelogram.
What % of 20-80 yr olds have bulging or protruding discs on MRI and are ASYMPTOMATIC!!!
52
Connective tissue and bone overgrowth
reducing the size of the vertebral foramina =
Spinal Stenosis
Normal aging proocess most common in peopl over 60
How does spinal stenosis present?
Chronic and progressive
Bilateral and poorly locaized
Pain that RADIATES wo butt thigh or legs
Worse with Extension (standing or walking)
Better with flexion (sitting or stooping.)
Stopping ambulation may not improve sx like it would if the problem were vascular claudication
What are the risk factors for peripheral vascular disease/claudication?
Smoking, DM, hyperlipidemia, FMHX
What is the presentation of peripheral vascular disease/claudication?
Chronic, progressive
bliateral or unilateral, poorly localized
worse with any lower extremity exertion
Diminished puses, delayed cap refill and cyanotic cool extremities are classic clues on physical exam
A Separation of the pars interarticularis of the
vertebral arch =
Spondylolysis
Slippage of one vertebral body onto the next. =
Spondylolithesis (scotty dog)
(must be lilateral for slippage to occur, most common L5/S1)
What type of injuries or athletes are most prone to Spondylolithesis?
Hyperextension injuries as seen in football lineman and gymnastic participants
What is the grading scale for Spondylolithesis? What grades is atpropriate to monitor the injury?
Grade I – 0-25% displacement.
Grade II – 25-50 % displacement.
Grade III – 50-75 % displacement.
Grade IV -75-100% displacement.
Grade III and IV are often surgical.
Grade I and II monitoring is appropriate.
What is the disorder based on the following description?
Pain in the back, in the facet region with no
radiation below the knee
Signs of spasm at segmental level
Paravertebral tenderness
Pain is felt in the morning upon rising, tending
to lessen with physical activity.
Painful motion especially hyperextension
Normal neuro exam
Facet joint syndrome
Who gets Fibromyalgia? What is the criteria?
Incidence in ambulatory edicine, 3-10% population
Over 75% are women between 20-60 years of age
Characterized by diffuse aches,
stiffness, and fatigue
American College of Rheumatology: at
least 11/18 tenderpoints for greater then 3 months duration
What sould be ruled or or a DDx when considering Fibromyalgia?
Rule out thyroid disease, lupus,
rheumatoid arthritis, malignancy, infectious
disease, etc.
What things exacerbate Fibromyalgia?
Pain and fatigue are worse with stress,
cold, and physical activity.
Greater than 70% of people with fibromyalgia have problems with this normal activity...
Sleep!

70% of patients have alpha
wave intrusion into non-REM delta wave sleep.

Assocaiated with IBS, depression and headaches