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23 Cards in this Set
- Front
- Back
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THE HEART DEVICES ARE FOR __ UNTIL A DONOR HEART BECOMES AVAILABLE FOR TRANSPLANTATION
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SHORT TERM USE AND TEMPORARY
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COMPLICATIONS OF ARTIFICIAL
DEVICES |
1.BLEEDING D/O'S
2.HEMORRHAGE 3.THROMBUS 4.EMBOLI 5.HEMOLYSIS 6.INFECTION 7.MECHANICAL FAILURE |
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TOTAL ARTIFICIAL HEART
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USED TO REPLACE BOTH VENTRICLES AND REQUIRES THE PT'S HEART TO BE REMOVED TO IMPLANT THE ARTIFICAL HEART
-IS STILL CONSIDERED EXPERIMENTAL -THE DEVICE IN USE IS CALLED THE *JARVIK-7* |
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POST OPERATIVE CONCERNS
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1.RISK OF REJECTION
2.RISK OF INFECTION 3.COMPLIANCE W/ COMPLEX MEDICAL REGIME OF DIET,MEDICATIONS,FOLLOW UP LAB STUDIES AND BIOPSIES |
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VENTRICULAR ASSIST DEVICE
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CAN PERFORM SOME OR ALL OF THE PUMPING FCN OF THE HEART AND CIRCULATES AS MUCH BLOOD PER MINUTE
-**CENTRIFUGAL PUMP**MOST COMMON -1 DEVICE IS NEEDED FOR EACH VENTRICLE |
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HETEROTOPIC TRANSPLANT
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LESS COMMON PROCEDURE
-DONOR HEART IS PLACED TO THE RIGHT AND SLIGHTLY ANTERIOR TO THE RECIPIENT'S HEART AND THE RECIPIENT HEART IS NOT REMOVED -PROCEDURE USED IN EMERGENCY SITUATIONS,WHEN THERE IS ISCHEMIA OR SOME OTHER COMPROMISE TO THE DONOR HEART |
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ORTHOTOPIC TRANSPLANT
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*MOST COMMON PROCEDURE*
-RECIPIENTS HEART REMOVED LEAVING A PORTION OF THE ATRIA -DONOR HEART IMPLANTED AT VENA CAVA AND PULMONARY VEINS BY SUTURING THE DONOR ATRIA TO THE RESIDUAL TISSUE TO RECIPIENT'S HEART -RECIPIENT'S PULMONARY ARTERY AND AORTA ARE CONNECTED TO THOSE OF DONOR HEART |
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SURGICAL MANAGEMENT
HEART TRANSPLANTATION |
USED WHEN MEDICAL TXT IS INEFFECTIVE AND THE PROGNOSIS IS LESS THAN 1 YR
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REDUCING ANXIETY
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1.OFFER EMOTIONAL AND SPIRITUAL SUPPORT
2.ASSIST TO DECREASE PERCIEVED STRESSORS 3.SET AND ACCOMPLISH REALISTIC GOALS NO MATTER HOW SMALL,FOR WELL-BEING 4.ALLOW OPEN COMMUNICATION |
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INCREASING ANXIETY TOLERANCE
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1.PACE ACTIVITIES IN CYCLES THAT ALTERNATE WITH REST PERIODS
2.DETERMINE WHICH ACTIVITIES THE CLT CAN DO INDEPENDENTLY THAN ALLOW REST BEFORE COMPLETING OTHER ACTIVITIES 3.REINFORCE SYMPTOMS THAT INDICATE THE NEED FOR REST AND WHAT ACTION THE CLT CAN THEN TAKE |
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NURSING INTERVENTIONS
(IMPROVING CARDIAC OUTPUT0 |
1.REST BY RECLINING CHAIR
(HELPS POOL VENOUS BLD IN THE PERIPHERY AND REDUCE PRELOAD) 2.MONITORING BY WEIGHT DAILY (DETERMINES RESPONSE TO TXT..ASSESSING SOB AND ACTIVITY INTOLERANCE) 3.MONITORING O2 SATURATING AT REST AND W/ACTIVITY-DELIVER O2 AS NEEDED |
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MEDICAL MANAGEMENT
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1.DIGOXIN LASIX
2.DIET-LOW SODIUM,HIGH POTASSIUM BALANCE OF EXERCISE AND REST 3.CONTROLLING DYSRYTHMIAS BY ANTIARRHYTHMIC MEDS OR PACEMAKER 4.IMPLANTABLE CARDIOVERTER DEFRIBILLATOR |
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ECHOCARDIOGRAM
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TO DETERMINE THE STRUCTURE AND FUNCTIONA OF THE VENTRICLES
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CHEST X-RAY
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CONFIRMS HEART ENLARGEMENT AND PULMONARY CONGESTION
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EKG
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CONFIRMS DYSRHYTHMIAS AND LEFT VENTRICULAR HYPERTROPHY
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WHICH TESTS ARE DONE FOR DIAGNOSIS
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1.EKG
2.CHEST X-RAY 3.ECHOCARDIOGRAM 4.BASED ON HISTORY AND PHYSICAL EXAMINATION |
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PHYSICAL FINDINGS OF CARDIOMYOPATHY
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1.TACHYCARDIA
2.EXTRA HEART SOUNDS 3.DYSRHYTHMIAS AND CONDUCTION ABNORMALITIES 4.PULMONARY CRACKLES 5.JUGULAR VEIN DISTENTION 6.PITTING EDEMA OF DEPENDENT BODY PARTS 7.ENLARGED LIVER |
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S/S OF CARDIOMYOPATHY
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1.MAY BE ASYMPTOMATIC FOR YRS
2.SOB ON EXERTION 3.PAROXYSMAL NOCTURNAL DYSPNEA 4.COUGH 5.CHEST PAIN 6.PALPITATIONS 7.FATIGUE***MOST COMMON 8.DIZZINESS AND SYNCOPE |
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PATHOLOGY OF CARDIOMYOPATHY
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ALL FORMS ARE PROGRESSIVE AND LEAD TO IMPAIRED PUMPING OF HEART
-RESULTS IN GRADUAL DECREASE IN STROKE VOLUME -RESULTS IN INCREASED SYSTEMIC VASCULAR RESISTANCE -VENTRICLE ENLARGES TO COMPENSATE AND THEN EVENTUALLY FAILS(RT VENTRICULAR FAILURE) |
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RESTRICTIVE CARDIOMYOPATHY
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RAREST TYPE
-IMPAIRED VENTRICULAR STRETCH AND VOLUME |
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HYPERTROPIC CARDIOMYOPATHY
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LESS COMMON
-INCREASES SIZE IN HEART MUSCLE ALONG SEPTUM -LEADS TO MISALIGNMENT OF PAPILLARY MUSCLE -MITRAL VALVE OBSTRUCTS BLD FLOW FROM LEFT VENTRICLE TO AORTA -A REDUCED SIZE OF VENTRICULAR CAVITIES AND INCREASES TIME TO RELAX CAUSES DIFFICULTY FOR VENTRICLES TO FILL -CLASSIFIED AS:OBSTRUCTIVE/NON OBSTRUCTIVE |
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DILATED CARDIOMYOPATHY
OR CONGESTIVE HEART FAILURE |
MOST COMMON TYPE
-SIGNIFICANT DILATION OF VENTRICLES W/O SIGNIFICANT INCREASE IN MUSCLE WALL THICKNESS RESULTS IN:POOR SYSTOLIC FUNCTIONING,GREATER AMT OF BLD REMAINING IN VENTRICLES AFTER CONTRACTION,STASIS OF BLD, AND A DECREASED CONTRACTION OF MUSCLE FIBERS OF HEART |
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CARDIOMYOPATHY
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A DISEASE OF THE HEART MUSCLE THAT LEADS TO SEVERE HEART FAILURE
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