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51 Cards in this Set
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this superficial skin infection occurs when this lipophilic fungus feeds on skin oil and removes stratum corneum layer
sx: red, brownish, blotchy, scaly, painless, hypopigmented or hyperpigmented skin of upper body of non suntanned individuals? |
tinea versicolor
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what will tinea versicolor look like in suntanned or dark skinned people?
what can this infection develop into? |
pinkish white patches on thes kin of upper body
can develop into atopic dermatitis on back and upper trunk (itchy papules and pustules) |
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what is a mold feeding on stratum corneum of HANDS AND FEET
sx: soliatry brownish black, nonscaly flat, painless MACULES what does the color of lesions come from? |
tinea nigra
color of lesions comes from black color of fungus |
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what is an infection of hair resulting in white, mycelial and nodular swellings along axis of hair shafts
sx: soft, white painless nodules that crust on skin of hair or genitals and is easy to break off? what does color od lesion come from? |
white piedra
color is from white fungus |
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what is an accumulation of large masses of epithelial debris containing fungi and/or bacteria and fungi in EXTERNAL AUDITORY MEATUS
sx: chornix infection of external ear with pain, itching, pain, inflammation and loss of hearing |
otomycosis
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what is invasion of glabrous skin of FACE, chin and upper lip of kids and females but NOT mustahce and beard of adult male
sx: scaling, annular or cirularlesions w/ raised margins and papules *pruritis, burning and erythema |
TInea faciei
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what is a mycelia that grow into pores around hair shafts and stratum corneum of skin. it produces edema, leukocytic infiltrations
sx. mild superficial type by scaling lesions on bearded areas of face and neck deep pustular type has deep folluclar pustules on bearded ares of face, neck what can these follicular pustules form into? |
Tinea barbae
can form info nodular kelids and kerions w/ alopecia and scarrin |
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what is also known as gray patch ringworm?
what do infected hairs look like? how do lesions begin and what do they develop into? what type of rxn can occur. what does allergic rxn cause? |
TInea capitis ectothrix
infected hairs, have a pale scaly grayish and breaksoff coalesced papules in ring form can develop into kerions and keloids allergix rxn producing dermatophytids can occur. this leads to alopecia, kertaomycosis |
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what is a form of tinea captitis that occurs when myclea interact with scalp proteins?
what does this produce |
Tinea favosa
develops into dead cell masses in mats of hair and yellowish brown cupshaped skin crusts(SCUTULA) with loss of hair and scarring |
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this infects the horny layer of skin and spreads circularly. center clears and rings of inflammtion on glabrous skin go from dry and scaly patches to pustural and crusty hyperkeratosis
what are two types |
tinea corporis
annular and vesicular |
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this starts from a nail injury. Enzyems that interact w/ skin and nail proteins produce a soft friable keratin that loosesn nail and THICKEns plate of fingernails or toenails
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tinea unguium
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what is a nail invasion restricted to white patches or pits on surface of nail?
what is a nail infection where edges are first involved and become brittle, friable and thickened w/ cracks |
leukonychia mycotica
invasive subungual dermatophytosis |
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what is an infection from maceration of skin from occupational activity that results in:
*exfoliative, erythmeatous, scaly sheets of skin that become vesicular red circumscribed patches |
Tinea manuum
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what is jock itch?
what do lesions begin as and develop into? what do old lesions become like? |
tinea cruris(moist area of groin)
begins as circular leion that develop into serpinginous, well marginated red lesions with raised borders and tiny vesicles old lesions become leather like from intense itching |
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athletes foot?
what promotes invasion of this fungus describe lesions? what are sx? |
tinea pedis
breakdown of host skin defenses promotes invasion infects localized to keratinized skin lesions are chornic papulosquamous sx: -intertriginous dermatitis(ppel soften and fissure b/w toes) -vesicles or bullar on dorsal surface of foot that ulcerate and can cause secondary bacterial infection |
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what is a hyphae that invaes subq tissue producing granulomas w/ allergic rxn
how does this organsim evade host defenses? |
Pseudallescheriasis
evades by depositing extracellular melanin and forming thick cell walls |
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someone comes in with eumycetomas
(painless tumor like swelling in skin that ulcerates and can scar w/ woody disfigurement) |
Pseudallescheriasis
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patient presentw wibh bronchial pneumonia acompanied with bronchial colonization and fungus ball formation?
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pulmonary pseudallescheriasis
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what are other forms of pseudallescheriasis
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*sinusitis, otomycosis, meningitis, arthritis, osteomyelitis, endocarditis, endopthalmitis, keratitis, cut and subq
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this is a mycelial invasion resulting in local lesions in lymphatics containing neutrophils, macrophages and giant cells
fociaare circumscribed microabscesses surrounded by leukocyte? where can this oranism infect? |
Sporotrichosis
1. lymphocutaneas 2. fixed cutaneous 3. mucocutaneous 4. pulmonary 4. extracutaneous |
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this forms gelatinous types of lesions that develop into masses of yeasts
how will mucocatenous form present? how will cutaneous candidiasis present? how will ysstemic present? |
Candidiasis, with granulomas and possible allergic rxn
muco as thrush, glossitis, stomatitis, cheilitis, perleche, vaginitis,k balantitis, gatroenteritis and bronchitis cutaneous as paroncyhia, diaper rash and onychomycosis systemic as fever, HA, anorexia, myalgia, |
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how can candidiasis be induced?
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iatrogenically following antibiotics or narcotic addiction
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what are yeasts seeded into blood?
what occurs in patiens on continuous ambulatory peritoneal dialysis from IV cath or machinery? |
Rhodotorulosis
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what are sx of rhodotorulosis?
can this be fatal? |
fever, hypotension, tachy, meningitis, chills
can be atal |
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what is a yeast that multiplies in lung tissue w/ formation of hyaline membrane around cyst like granulomas containing an eosinophic exuadte in the alveoli?
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Pneumocystocis
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what are sx of pneumocystosis?
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fever, chills, nonproductive cough, chest pain, congestion, anorexia, sob, weight loss and development of INTERSTITIAL PLASM CELL PNEUMONIA in immunocompromised hosts(COTTON WOOL SPOTS)
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what is a capsular polysaccarid that results in depletion of complemetn and inefficient opsonization and inefficient phagocytosis?
what does it form in large numbers? how is resolution of this? |
Cryptococcosis
forms granulomas with large number o encapsulatied yeast cells in clusters SLLOW resolution leaving walled off granuloma in tissue |
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what is the most frequently dx form of cryptococcosis?
what are other possible presetations? |
CNS (HA, fever, nausea, vomiting, lethargy, nuchal rigidity, photophobia, neck tenderness)
also: -cutaneous -mucocutaneous -disseminated dz -osseous *involves swelling and pain b/c of granulomas |
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what invades endothelial cells resulting in pyogenic suppuration and necrosis w/ release of fungal endotoxins
has presence of dsRNA mycoviruses what type of tissue are lesions? what are other types of infection? |
Aspergillosis
granulomas infect: -pulmonary -disseminated -iatrogenic |
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patient has fever, dyspnea, edema, hemoptysis, and pleuritic chest pain and fungus balls?
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aspergillosis
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patient has rhinocerebral prptosis? what is possible dx
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-desseminated aspergillosis
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infection results in development of necrotic foci that is
-bronchitis -trhush -colitis -cystic masses in skin |
Geotrichosis
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infection of nailr damages nail architecture with development of grooves and cavities but W/O thickening of nail plate
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onychomycosis
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fever, dyspnea, bronchitis and hempotptysis in patients w/ leukemia or lymphoma?
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Mucormycosis
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fever, HA cellulitis of nose, bilateral prpsis and swelling of brain in DM or leukemia
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rhinocerebral mucormycosis
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GI ulcers, diarrhea and peritonitis?
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abdoinal mucormycosis
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contamninated bandages presentw with abscesse and granulomas in skin of burn patients?
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Cutaneous murcormyosis
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debilitated patienst due to invasion of nose or paranasla sinuses ora s result of head trauma
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cNS mucormyocosis infection
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these hyphae directly invade blood vessels an cause necrosis, thrombosis and infarction w/ loss of artrial blood supply and can result in microabscesses in which eosinophils predomoinate as a sheath around hyphae
LEsions are necrotic and fibrotic w/ consistency o RUBBER. mucormycotic infections are aggressive and are TRUE MEDICAL EMERGENCIES in immunosuprressed DM or neutropneic patients |
MUCORMYCOSIS
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YEast forms multiply in tissue resulting in PYOGENIC abscess formation w/ accumulation of neutrophil
what is cellular response dependent on? |
Blastomycosis
*severity of disease and vaires w/ infiltration of macrophages and histiocytes,lymphocytes and plasma cells or fibroblasts |
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what is crab claw shadow?
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blastomycosis
lower lobe lung leison |
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dry cough, chills, pleuritic pain, pneumonia, sore throat, myalgia fever and weight loss?
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primary pulmonary blastomycosis
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ulcerative subq nodules?
osteomyelitis or septic arthrirtis |
cutaneous blastomycosis
osseous blastomycosis |
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with is a suppurative, neutrophil filled tisse with endospores w/ double refactile walles.
Fomration of granulomas produce cavities and nodules that result in scarring |
coccidioiodomycosis
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asymptomatic or with fever, chills, pleuritic pain, cough, anorexia and headaches referred to as VALLEY FEVER
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pulmonary coccidiomycosis
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Desert bumps
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allergic coccidiomycosis
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Desrt rhumatism
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arthrisis from coccidiomycosis
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solitary or mutliple necrotic granulomas and lesion with yeast infected monocytes and macrophages growin in bone marrow and disseminate the dz
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histoplasmosis
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fever, cough, loss of breath, bloody sputu, sweating, weight loss , malaise, cyanosis, hemoptysis, cavitation and penumonia
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severe pulmonary histoplasmosis
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oropharyngeal ulcer formation on mucous m embrnaes that become granulmatous?
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mucocutaneous histoplamsosis
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fever, anemia, meningitis, hepatsplenomaglay, leukopenia, septick shock and weight loss
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systemick histoplasmosis
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