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192 Cards in this Set

  • Front
  • Back
Third spacing occurs where?
In the transcellular spaces, including pericardial and peritoneal.
Ecf consists of what?
Intravascular space, interstitial space, transcellular
Edema is considered what type of spacing?
2nd spacing in the interstitial space.
Third spacing may present with what symptoms?
Inc hr, decr bp, decr uo, wt gain rapid, I greater than o.
how does the pit react to incr osmolality? Decr osmol?
Secretes adh to retain water to decrease the concentration of blood = less pee. Decr osmo = less adh to increase blood concentration = more pee
What is the def of dehydration?
Pure water loss without loss of Na
Hypovolemia s/sx
Inc hr, decr bp, incr rr, decr uo, decr skin turgor, sudden wt decr, resstlessness, lethargy
Nursing int for hypovolemia
Assess electros, give fluids
Hypervolemia s/sx
Incr hr, incr bp, incr wt, incr edema, neck vein distention, crackles, ha, confusion
Pt presents with crackles in lungs and confusion with ha. Possible dx?
Hypervolemia
Pt has vascular dehydration. What orders wood the rn expect?
Isotonic solution such as normal saline (0.9% nacl), lactated ringers, or dextrose in water (d5w).
Pt has cellular dehydration, the rn expects the md to order what? Explain
Hypotonic soltn, ie 0.45% NaCl or 0.33% NaCl. Hypotonic soltn is less concentrated than cells thus fluid enters cells to try and dilute the higher concentrated cells.
Pt has cellular dehydration, the rn expects the md to order what? Explain
Hypotonic soltn, ie 0.45% NaCl or 0.33% NaCl. Hypotonic soltn is less concentrated than cells thus fluid enters cells to try and dilute the higher concentrated cells.
Pt has vascular dehydrarion with edema. Rn expects dr to order what? When would u hold that order and call dr?
A hypertonic soltn like d5ns (5% dextrose in ns), 5% dextrrose in 0.45% nacl, (d5 1/2 ns), 5% dextrose in lactated ringers (d5lr), or hypertonic saline 3%:::::::: would hold those orders if pt has kidney or heart disease.or if pt is cellular dehydrated.
Pt has vascular dehydrarion with edema. Rn expects dr to order what? When would u hold that order and call dr?
A hypertonic soltn like d5ns (5% dextrose in ns), 5% dextrrose in 0.45% nacl, (d5 1/2 ns), 5% dextrose in lactated ringers (d5lr), or hypertonic saline 3%:::::::: would hold those orders if pt has kidney or heart disease.or if pt is cellular dehydrated.
How do hypertomic soltns work?
They have greater concentration than in the cells, so fluid leaves the cells to try and dilute the vasculature, causing cells to shrink.
A pt on lasix loses 4.4 lbs/1 kg in 24 hours. How much fluid loss is this equal to?
2 liters. One liter of water weighs 2.2 lbs or 1 kg.
Pt at risk for fluid imbalance. Rn should do what seven thing?
I&O
Monitor vs, hr, posteral changes in bp of 10% or more
Neuro status
Daily wt
Skin assess
Monitor iv fluids
The rn knows that ____ is the primary electrolyte in the ecf?
What is the normal range?
Sodium
135-145 mEq
a pt with poorly controlled dm (hyperglycemia) is at risk for what electrolyte imbalance?
hypernatremia
when replacing water in hypernatremia, the RN knows that it is important to replace fluids rapidly or slowly?
slowly, or else cells will swell
what is the most common electrolyte imbalance seen in hospitalized pts?
hyponatremia
from n/v/d, ng suctioning, excessive sweating, diuretics, and too much D5W
what does SIADH cause?
syndrom of inappropriate antidiuretic hormone is whe adh is released regardless of plasma osmolality.
Causes hyponatremia because of the increased water retention diluting the na concentration.
the nurse knows that the elderly are at increased risk of this electrolyte imbalance because of decreased renal function
hyponatremia
as Na is lost, what is reabsorbed?
potassium
as potassium is lost, what is reabsorbed?
sodium
what is the primary route for K loss?
kidneys
pt presents with a severe crushing injury to the legs, what does the nurse monitor for in electrolytes? what does the rn expect to be ordered?
hyperkalemia caused by K released from hemoloyzation.
iv insulin and glucose to drive K+ back into cells
pt just had a new ileostomy, what does the rn watch for?
hypokalemia
when giving potassium, the RN always assesses what before giving?
urine output to see if it decreases
and also checks mag levels - if too low the kidneys will excrete K to preserv mag, so replace mag with or before K
the nurse knows that as phosporus goes down, what goes up and vice versa?
Calcium
how does the parathyroid gland regulate Ca levels in serum?
releases parathyroid hormone which stims movment of ca from bone to plasma and increases GI absorption of Ca
what is needed for pth to regulate Ca?
Vit. D
pt is hypercalcemic, the nurse expects to do what
administer diuretics to promote excretion of Ca
admin IVF to hydrate and dilute Ca and increase renal excretion
pt is hypocalcemic, the nurse knows this by what s/sx?
positive chvosteks or trousseau's sign
parestesia, numbness, muscle spasm, hyperreflexia progressing to tetany
pt is receiving a blood transfusion, what electrolyte imbalance might he have
hypocalcemia
nursing interventions/collaborative care for hypocalcemia
replace Ca+, treat cause
IV Ca give slowly over time, fall preventions
acronym for electrolytes with values:
Meg's - Mg: 2 (1.5 - 2.5)
Phone - Phosphorus: 2 (2.5-4.5)
Keeps - K (Potassium): 3.5 - 5.0
Calling - Calcium: 8-10
Nancy - Na (Sodium) 135-145
pt presents with premature arrhythmias, bradycardia, and heart irritability, fatigue/lethargy, what electrolyte imbalance might he have
hypokalemia
the nurse knows to do what when giving potassium iv?
use a pump and infuse slowly
decreased clotting time can be caused by what e- imbalance?
hypercalcemia
pt presents with slowed cardiac activity and asystole, what e- imbalance could be the cause?
hyperkalemia
loss of deep tendon reflexes can be caused by what e- imbalance?
hypermagnesemia
pt with new ileostomy and receiving nasogastric suctioning is feeling lethargic, hr 125 and irregular, bp 90-52, ingling in fingers and toes. What imbalances might be going on.
Hypokalemia - ileostomy and ng suctioning can cause na and k loss through fluid loss.
Pt on digoxin for arrythmias... what imbalance would you hold his med for?
hypokalemia because it increases the risk of dig toxicity
what two imbalances can be caused by ng suctioning?
hypomagnesemia and
hypokalemia
what imbalance can be caused by crushing and burn injuries?
hyperkalemia - potassium normally is in the icf but burns tumor lysis or crushing injuries release the K into the ECF
addisons disease increases risk of
hypomagnesemia
hypermagnesemia may be treated how?
with IV calcium to restro Ca-Mag balance
pt with crushing or burn injury may present with

How treated?
muscle weakness

bradycardia, asystole

Hyperkalemia treated with iv insulin and glucose to drive K back into cells
WBC normal values
5-10
Hgb normal values
13 - 17 (15)
Hct normal values
42-52%
Platelet normal values
140000-400000
Should be above 100,000
normal prothrombin time

normal partial thromboplastin time

normal INR
11-13 sec

24-32 sec

2-3
Normal BUN
Bunnies less than twunny (less than 20)
Normal creatine
creatures blue should be less than two (less than 2)
acronym for electrolytes with values:
Meg's - Mg: 2 (1.5 - 2.5)
Phone - Phosphorus: 2 (2.5-4.5)
Keeps - K (Potassium): 3.5 - 5.0
Calling - Calcium: 8-10
Nancy - Na (Sodium) 135-145
nursing care for the pt with hyponatremia includes what?
fluid restriction because water is leaving the ecf and entering the cells causing swelling - we don't want more water adding to the problem
a pt receiving loop diuretics should be observed for what symptoms?
weak, irregular pulse,, poor muscle tone
sx of dehydration because loop diuretics cause excretion of NaCl, which causes water to follow
a pt with renal failure is at high risk of developing what e- imbalance?
hypermagnesemia because mag is secreted via kidneys
a pt who has just undergone a total thyroidectomy must be assessed for what?
positive chvosteks or trousseaus (hypocalcemia) to see if hyperthyroid is intact
a pt with hyperphosphatemia will be treated with
calcium supplements because as Ca rises in serum, it causes exretion of phosphate
hypokalemia is associated with what ECG changes?
the presence of a U wave
hyperactive bowel sounds are a sx of what imbalance?
hyponatremia
foods high in potassium include
avacado, banana, cantaloupe, carrots, fish, mushrooms, oranges, ptoatoes, raisins, spinach, strawberries, and tomatotoes, pork, beef, veal
foods high in calcium include
dairy, collard greens, rhubarb, sardines, spinach, tofu
rn interventions for anemia
O2 admin
blood/blood product admin if symptomatic
drug therapy (iron, erythropoietin)
dietary changes to increase iron intake
education
Hgb measures what?
the total amount of Hgb in peripheral blood
Hct measures what?
Normal value should be?
percentage of total blood volume made up of RBCs'
Should be around 40
males 40-54
females 38-47
what is TRALI
transfusion related lung injury
what is TRACO and how does it present differently than TRALI? What common sx does it share with trali?
transfusion related circulatory overload
no fever or hyptension
dyspnea, crackles, change in LOC
15 minutes after starting a PRBC transfusion, the pt becomes restless and complains of itching. what should the nurse do FIRST?
Stop the transfusion!!!!

Then open up saline, get vs and call for help
pt has been diagnosed with anemia, what s/sx might the rn expect
PALENESS #1 sign
glossitis
cheilitis (inflam of lips)
headache
parasthesias
burning sensation in tongue
when and what might the rn give to a pt with his po iron supplement?
1 hour before meals with vitamin c/orange juice.
If liquid, give with straw to avoid teeth staining
if pt is put on life time iron supplements, what is necessary to monitor?
liver function because iron is stored in the liver
pts with chronic kidney disease are susceptible to what blood condition? why?
anemia, because the kidneys production of erythropoitin is reduced so new blood cell production goes down.
why does inflammatory chronic disease cause anemia?
cytokines cause macrophages to uptake and retain Fe, leaving inadequate Fe for RBC production
definition of thrombocytopenia
platelets below 150,000
the nurse notices oozing from iv site on her patient, what might she suspect?
thrombocytopenia
define ecchymoses
large purplish lesions, flat or raised, painful and tender, caused by thrombocytopenia
thrombotic thrombocytopenic purpura (TTP)
agglutination of platelets cause microthrombi that depositi in the vasculature accompanied by bleeding from low platelets and clumping of the ones that do exist.
when might a platelet transfusion be performed in a thrombocytopenic pt?
when platelets go below 10000
what meds might be used in a thrombocytopenic pt?
immunosuppressants such as azathioprine (Imuran) or cyclosporine
nursing eductation of the thrombocytopenic pt?
dab nose instead of blowing it
define neutropenia
decreased neutrophil count
pt has been on long term chemotherapy, what are they at high risk for developing?
neutropenia
pt presents with an oportunistic infection but has now s/sx of inflammation, no redness, heat, swelling, or puss. What does the nurse suspect?
neutropenia
in a neutropenic pt, what is the best indicator of infection?
fever greater than 100.4
appropriate nursing actions when caring for a severely neutropenic pt include
strict hand hygiene and frequent VSs
pt presents with bleeding gums, petechiae, what does the nurse suspect?
thrombocytopenia
neutrophil count in normal and neutropenic conditions
Normal = 4000 - 10000
neutropenia = <500 or an absolute neutrophil count (ANC) of less than 1000
neutropenic precautions and education
reverse isolation,
limited visitors, no kids or sick
positive pressure or HEPA filter
no fresh fruits/veg, flowers
no sunburn, avoid public places and animal
describe the RBCs in iron-deficiency anemia
microcytic and hypochromic
what deficiency might result in macrocytic yet normochromic RBCs?
Vit B12 or folic acid
what is the most important, mature form of WBCs?
neutrophils
pt presents with splenomegaly, jaundice and pruritus, pallor, tachycardia, and fatigue. What does the nurse suspect?
anemia with hemolysis occuring
polycythemic pts are at high risk of what?
thrombosis
describe the RBCs in iron-deficiency anemia
microcytic and hypochromic
polycythemia
stem cell mutation leads to increased productionof rbcs, wbc, and platelets
s/sx of polycythemia vera
htn, ha, vertigo, tinnitus, visual disturbances,
pruritis (itchiness), parethesias and redness of hands and feet, thrombophlebitis, intermittant claudication
tx for polycythemia vera?
phlebotomy to reduce Hct to less than 45%, removal of 300-500 mL, iv hydration, myelosuppression (bone marrow activity suppression with drugs), I&O, thrombus prevention
Normal arterial blood pH should be between
7.35 and 7.45
normal PaO2
65 - 100
nsg interventions for atelectasis
prevention
cough/deepbreathing/incentive spriometer (CDB/IS)
turning
ambulation
pain mgmt
if PN is hospital acquired, then it occured when
greater than 48 hours after hospitalization
risk of aspiration increases with what?
a decrease in LOC
pt presents with coughing of purulent sputum and hemoptysis, decreased bs and crackles, fever, chills pleuratic pain, what does the RN suspect?
Lung abscess
who is at highest risk of lung abcess?
anyone with increased aspiration risk ie from lowered LOC from anesthesia, etc. because most lung abcesses are caused by aspiration of material from the GI tract
definition of pulmonary hypertension (PPH)
mean pulmonary arterial pressure > 25 mmHg at rest

Normal pressure is 12-15
Pulmonary Hypertension mimics what disorder?
right sided heart (right ventricular) failure
Upon cardiac assessment, what might the RN hear in a pt with pulmonary hypertension?
the RN detects an S4 gallop,
pt presents with white fingertips which turn blue and then red with tingling and burning. What does the RN suspect?
Raynauds syndrome, which could be associated with pulmonary hypertension
during a status asthmaticus attack, what might the nurse request to add to the treatment regimen of bronchodilators?
Lorazepam
Normal Cardiac output.
4-8 L/min
general cardiac symptoms
SOB, dyspea, rales, wt gain, nocturnal diuresi, generalized fatigue.
Arterioles are resistance vessels that change diameter under what stimulation
Autonomic nervous system (ANS
pulse pressure is what?

what is normal
systolic - distolic

30-40 mmHG
your patient's pulse pressure is 20 mmHG, what does this indicated
90/70 pp = 20mmHg (patient is vasoconstricted b/c of BP; trying to compensate)
blunted baroreceptor response is a clinical implication of what?
normal aging in the CV system
preload definition
The volume blood in the ventricles at end diastole before the next contraction begins.
↑preload: more fluid is left in the ventricle so, the harder the heart has to pump, to move it forward (↑ O2 consumption)

If preload stretches the myocardium beyond its physiological limits, contractile force ↓, SV is reduced, therefore cardiac output is reduced
afterload definition
The resistance against which the left ventricle must pump against to eject the volume. (open the aortic valve)
what is a physical change that occurs in the heart r/t increased afterload?
left ventricular hypertrophy
LDL is considered "_____? and should be at what level?
BAD
less than 130
HDL is considered "__? and should be at what level?
GOOD
35-85
triglycerides should be at what level?

homosysteines?
less than 150

4-15
what is MAP?
mean arterial pressure:(SBP+2DBP)÷3 (avg BP in arterial system felt by organs

adequate arterial pressure is needed for cap perfusion
describe the two theories of HTN cause:
hemodynamic
vasoconstrictor
Hemodynamic hypothesis: ↑SNS activity, inappropriate renin-angiotension-aldosterone system (RAAS) stimulation
Vasoconstrictor hypothesis: inappropriate Ca+2 in smooth muscles
the sympathetic nervous system stimulates the renin angiotensi system to do what
increase Na and increase h2o, which increase preload and cardiac output.
baroreceptors sense low BP and cause the secreation of what hormone?
ADH
vasoconstriction does what?
increases preload AND afterlowad, which increases o2 consumption and stresses heart muscle/
what is the single biggest problem resulting from HTN>
end organ damage
heart: lv hypertrophy
kidneys: increased RAAS stim perpetuates HN
Brain: increased risk of hemorrhage
Retinas: hemorrhage
Aorta: aneurysm and rupture
the RN knows to question orders for what type of diuretic for a heart pt with renal failure?
potassium sparing duretics
what nursing concern should you monitor for for anyone on any type of diuretic?
hypostatic hyptension and electrolyte imbalances
you pt presents with obesity, triglycerides over 150, HDL less than 40, fasting BG over 100, and HTN. What do you expect his diagnosis will be? What is needed to prevent a cardiovascular event?
Metabolic syndrome
Heart Healthy Life Style Education
Exercise
Tight BG management
Management with medications
antihypertensives, statins, insulin,
a pt with a HR of 180 bpm, what is your concern and why?
coronoary arteries receive o2 during diastole, which is not happening with that high of a HR. Concern is MI
what is number one modifiable risk factor for all CAD/MI pts?
smoking cessation
If chest pain occurs with activity, lasts 3-5 min but is relieved by rest and/or nitrates, what might the dx be?
chronic stable angina
a diabetic pt presents with increased blood glucose and fatigue - what would the nurse be concerned about?
angina - neuropathies may be present, lowering sensitivity to pain.
your female pt has been diagnosed with panic/anxiety disorder and complains of an aching jaw/choking sensation, what would the RN be concerned about?
angina
over the age of 70, chest pain does not aloways occur. what might you see instead?
dyspnea and indigestion, confusion or disorientation
in your pt with chronic stable angina, what medications would you expect?
Nitrates, sublingual nitroglycerin
Beta blockers
Low dose ASA
Calcium channel blockers
how does nitroglycerin help in angina or ischemic events?
Vasodilates coronary veins and arteries
Relaxes systemic arteriolar bed, decreasing SVR
your pt is on nitroglycerin what must you monitor for?
orthostatilc hypotension
and
tolerance over long term
how do beta blockers work?
block the excitation of beta receptors which lowers o2 consumption, lowers HR, lowers BP, lowers contractility. This all increases stroke volume and increase o2 to myocardial tissues by slowing the heart down (more blood to coronary arteries during diastole)
your pt is on beta blockers, what must you watch for?
Hypotension
Hyperglycemia (drug specific)
Bronchoconstriction
Hyperlipidemia
calcium channel blockers work how?
block mvment of Ca into cells, causing vasodilation, decreased HR, decr contractility and systemic vascular resistance . Reduces O2 demand by the heart.
Your pt is on Ca channel blockers, what must you watch for?
Cardiac arrhythmias
Hypotension
Constipation
Gastric distress
ACE inhibitors work how?
ACE is in blood vessel lumens and converts angiotensin I IIpreload andBP
ACE inhibitors BLOCK this mechanism
decreases BP
decreases SVR
decrease myocardial work during systole
Causes Diuresis
your pt has been admitted to the ER with anxiety, increased HR, increase RR, diaphorsis and fever, arrhythmias, n/v and
FEELINGS OF DOOM
What is the probable diagnosis?
Acute Coronary Syndrome
your pt who has had a h/o chronic stable angina complains that his chest pain has "changed". What are you thinking?
Unstable angina: acute coronary syndrome
your pt has just had a cardia catheterization procedure. What are you doing and watching for?
Assess circulation in extremity used for catheter insertion
Observe insertion site for hematoma and bleeding
Place compression device over catheter insertion site
Monitor VS and cardiac rhythm
Monitor for s/s of PE
Affected leg should remain straight
Pt presents in the ER with severe CP unrelieved by rest, pressure, tightness radiation to jaw, neck, down arms, weakness, SOB
what is probable diagnosis
myocardial infarction
what labs would you expect to see rising 3-12 hours and peak 24 hours after an MI?
Creatine kinase myocardial bands (Ck-MB)
it has been 4 days since your pt says he experienced chest pain but didn't come in for it. What labs are you interested in seeing?
cardiac-specific tronponin I (cTni) because they rise 3 hours after an MI but don't return to baseline until 5-14 days after. Ck-MB would not be a good lab for indicating MI this far after because it returns to normal after 48 hours
your pt has a Ck-MB level of 1.0 - what is your expectation of dx?
that an MI has not occurred. Levels of more than 2.5–3 are more indicative of an MI.
What cTni levels are you looking for to indicated an MI has occurred?
over five or any detection at all really
in a pt with a 40 year history of diabetes with newly diagnosed angina, how will an MI most likely present?
Malaise, hyperglycemia, nausea and vomiting
you have a pt in your ER who has suffered a non-STEMI. The nurse would question orders for what?
Clot buster medications such as:

tissue plasminogen activator
recominant plasminogen activator
streptokinase
ONLY USED IN STEMI
What would you see as a sign that the clot buster is working when given to a pt with STEMI/
reduced ST elevation and improved cardiac enzyme level
you would monitor the effectiveness of Heparin with what lab?
therapeutic PTT value should be 65 to 95 seconds, but this varies by the hospital.
If the PTT shortens to below the therapeutic range, danger of having a second heart attack. Often second attacks are fatal. PTT results that are longer than the high limit indicate a risk of bleeding.
how would you monitor the effectiveness of coumadin?
Most people taking Coumadin should have an INR between 2 and 3. An (PT) INR below 2 signals the danger of a new clot, whereas an INR over 4 means a risk of dangerous bleeding.
what does the RN do post-op for a pt who has just had an artery bypass surgery?
Ambulation
Sternal precautions
Pain control
IS/deep breathing
Incision care
Manage chest tubes/pacer wires.
Monitor hemodynamics, urine output, heart rhythm, fluid & e-lytes
your pt's ejection fraction has just been determined to be 30%. what might your patient's diagnosis be? Define it.
Low Output Heart Failure (systolic dysfunction): LV is unable to eject normal blood volume with each beat
Normal EF = 60-75%
what are some causes of low output HF
LV or RV failure
PHTN (pulmonary hypertension)
Valve stenosis
define high output heart failure and list some causes
Too much blood for the LV to eject
Excessive volume leads to LV dilatation and fluid backup into the Pul vasculature
Causes; valve regurgitation, hyperthyroidism, anemia, hypervolemia, lack of inotropic ventricular force
what are some compensatory mechanisms with HF?
increased HR to increase CO
increased rennin to cause vasocxn and volume retention to increase stroke volume
vasopressin released to increase vasocxn
when sv is adequate BNP/ANP are relased to stop these processes and decrease bp.
describe dilated cardiomyopathy
most common
increased dilated ventricle= decreased contraction & decreased SV, increased diastolic volume in V= increased stretch (dilate)
describe hypertrophic cardiomyopathy
size, mass, esp septum
incrase mass = decreased ventricle size = longer diastole, poor passive fill, increased septal mass = increased O2 consumption
restrictive cardiomyopathy
least common
ventricle can’t stretch
Amyloidosis (F>M
in HF that results in poor forward flow, what doess the RN assess for and what are the treatment goals?
Assess for cause of poor CO and treatment goals are:
Maximize CO
Improve tissue perfusion
Minimize symptoms
what are some s/sx of decreased cardiac output?
increased HR
****decrease Urine output/decreased renal function******
arrhythmias, ppor nutrition
****increased Diastolic BP (chronic vasoconstriction)*****
POOR ACTIVITY TOLERANCE
your pt has SOB, rales/crackles, wet cough, nocturnal dyspnea, confusion/restlessness
WHAT DO YOU SUSPECT?
left sided HF
your pt has systemic edema, weight gain, hepatomegaly, ascites, peripheral edema, JVD, anorexia
What do you suspect?
Right sided HF
what is the goal of Nursing and collaborative care for Heart Failure?
TO MAXIMIZE CO
what are the ways we maximize CO in HF?
Diuretics
ACE inhibitors (ARBs if ACE intolerant)
Beta and Ca channel blockers
Digoxin
Nitrates
Synthetic BNP (Natrecor)
Manage dyspnea
Monitor labs and treat electrolytes
Renal function tests, BNP, K, Ca, Mg, Na
Exercise programs; manage fatigue
Diet mgmt
Cardiac transplantation
In a pt with HF, the nurse knows to establish what in order to track progress.
establish baselines
frequently, actue heart failure presents what?
acute pulmonary edema, usually associated with LV failure
your pt presents with SOB, increased RR, decreased spO2, agitation/anxiety, wheezing, wet cough, and rales. What do you suspect and what is your priority?
Left sided heart failure causing pulmonary edema.

Priority is to improve O2, and move the fluid out of the lungs with diuretics.
How does the nurse assess for Peripheral vascular disease?
CMS checks: Circulation, Motion, Sensation.
Peripheral pulses
Motor function
Numbness? Tingling? Pain? Loss of sensation?
Color, tempreture, swelling, hair loss?
Skin integrety
Ankle-brachial index (ABI)
Doppler probe
your pt presents with severe pain in lower extremities, especially with activity, intermittent claudication (impairment in walking, or a "painful, aching, cramping, uncomfortable, or tired feeling in the legs that occurs during walking and is relieved by rest)
decreased to absent pedal pulses, and deep ulcers on toes and heels. No edema
WHat do you suspect?
Peripheral Arterial Disease
your pt presents with dull aching craming pain in lower extremities. Pulses are +1, and there is uneven pigmentation with a blue hue in the toughened skin. Moderate edema with medial and lateral ulcers are present.

What do you suspect?
Peripheral Venous Disease
what are your nursing interventions in PVD?
pain relief with meds,
position pt to relieve pressure and edema in Lower extremities
Wound healing
Keep kin clean, dry, moisturized
ENCOURAGE Mobility
antiplatelet agents
your pt has venous stasis, immobility, hypercoagulability and is dehydrated. What is he at risk for?
Deep vein thrombosis
your pt has dull aching pain in his leg. you pull back the covers to reveal erythema and incrased temperature in the leg. He has a + Homan's sign.
What do you suspect?
Deep vein thrombosis
interventions once a DVT develops
Bed Rest
Elevate extremity
Heparin or Warfarin
Monitor for s/sx clot migration
what are aortic aneurysms caused by?
CAD leading to atherosclerosis: plaques lead to degeneration of vessel walls  weaken and dilate
HTN
Genetics
Congenital abnormalities
Trauma
Infxn
in a pt with thoracic aortic aneurysm, (TAA) what is your sign and what is your priority when diagnosed?
embolic showers may be first s/sx in brain and fingers.
back pain, SOB, hoarsenes, difficulty swallowing

control BP (HTN)
control pain
what is the usual primary s/sx of abdominal aoritic aneurysms?
Renal compromise
your pt states that it feels like her heart is in her abdomen, what do you suspect?
abdominal aortic aneurysm
renal and Lower extremity embolic showers could be indicative of what
abdominal aortic aneurysm
what do elevated levels of homocystine tell you? What 's elevated?
+ for presence of CV disease
15-30 micromoles per liter as moderate


30-100 micromoles per liter as intermediate


Greater than 100 micromoles per liter as severe