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99 Cards in this Set

  • Front
  • Back
stress coping process
short lived, quick.

compensatory process with physiological and psychological components
day to day stress
has greatest impact on health
physiologic response to stress (what ns?)
SNS (norepinephrine) is activated. cascade of neural and hormonal events.
effects of stress
peripheral vasoconstriction
bronchodilation
pupils dilate
dec GI activity
inc blood sugar/fatty acids
feeling of tension
inc sweating
inc rate of blood coag
maladaptive stress
drugs and alcohol
type a personality
denial
avoidance
infant body fluid
70-80%
adults body fluid
60%
geriatric body fluid
45-50%
fluid loss sxs (4)
mild lightheadedness
convulsions
coma
death
intracellular (what ion? how much % body water?)
2/3 of body fluid
K+ is prime cation
what is extracellular fluid (3 things)
intravascular and interstital
1/3 body fluid
Na+ prime cation
input/outpt volume
2600 mL/day
inc volume
inc weight
causes more fluid to leak out into intravascular area, into interstitial area
hydrostatic pressure
wt and volume
more volume IV causes inc BP
pushing pressure - water out of vessel
osmotic pressure
the pressure which needs to be applied to a solution to prevent the inward flow of water thru membrane
osmolality
# particles in kg of fluid (1L H2O)
normal osmolarity of plasma
250-375 mOsm/L
Renin-Angiotensin-Aldosterone
holds on to Na, H2O
ADH
holds onto urine
Atrial Natiuretic Peptide
inc absorption of Na/H20 (lose Na/H20 to urine)

activate when renal blood flow is low. SNS kicks in
RAS system
renin -> angiotensin 1 -> angiotensin 2 -> inc BP

renin -> secretion of aldosterone -> retain H2O and inc BV and BP
hypervolemia/ FVE
caused by
excessive IV infusions
replace water w/o Na
ADH secretion
Aldosterone (renal, adrenal, CHF, liver failure)
excess salt intake
what FVE looks like
fast wt gain
peripheral/periorbital edema
JVD, bounding HR, inc BP
inc CVP, RA pressure
SOB, crackles
dec Hct, Na (dilution)
headache, confusion
normal CVP in RA
6-12 cm H2O
causes of edema
inc capillary hydrostatic pressure
dec plasma protiens
obst lymphatics
kidney malfuction
inc capillary permeability
meds: steroids, NSAIDs, estrogen
edema (2 types)
generalized (anascarca)
localized
1st space
intravascular fluid
2nd space
interstital fluid (pitting edema)
3rd space
pericardium, pulmonary edema, etc
intervention for FVE
(what position?)
dec interstital fluid
promote circulation
skin integrity
semi-fowlers postition
i&o / weights
FVD hypovolemia causes
decreased intake
increased ouptput
decreased abs of fluid
assessment for FVD
dry mothe, sordes
dec skin turgor
inc temp
olig/anuria
inc hct, serum na, bun
restless
dec bp
dec cvp, flat veins
interventions for FVD
fluids
i&o /weights
skin integrity
hypovolemic shock
na range
135-145
K range
3.5-5.5
chloride range
96-106
CO2
24-30
bun
10-20
creatinine
0.7-1.5
glucose
80-110
urine ph
4.5-8.0
urine spec gravity
1.010-1.020
hyponatremia sxs (6)
AMS
headache
anorexia, N/v, cramps
muscel twitches
hypernatremia sxs
restelssness
AMS
twitching
hypokalemia sxs
slow reflexes
fatigue
constipation
lo T wave, U wave
hyperkalemia sxs
peaked t wave
nausea
irregular HR
acidosis
hypokalemia sxs (4)
slow reflexes, numbness
fatigue
constipation
lo T wave, U wave
hyperkalemia sxs
peaked t wave
nausea
irregular HR
acidosis
hypotonic crystalloids
.33, .45 NS
D5W

fluid out of vessels
hypertonic crystalloids
D5W .45NS
d5w .9 NS
3% NS
5% NS

fluid into vessels
isotonic crystalloids
0.9% NS
LR

too much too fast - FVE
albumin 5, 25%
keep fluid in vessels
good for shock tx
dextran, hetastarch
synthetic
fluid into vessels
mannitol 5,25%
fluid out of vessels
oliguric diuresis
eliminates cerebral edema
hypercalemia sxs
lethargy, wekaness
dec reflexes
constipation
hypocalcemia sxs (6)
muscle cramp, twitching
tetany - convulsions
cardiac arrythmias
trousseaus
chvosteks sign
blood ph
7.35-7.45
pCO2
35-45
HCO3
22-26
resp acidosis cause
depressed resp center
decreased lung surface
resp alkalosis cause
hyperventiliation
METABOLIC acidosis cause
DM
starvation
ASA overdose
renal failure
diarrhea
tissue anoxia (anerobic)
metabolic alkalosis causes
loss of HCL
excessive bicarbonate
fluid and electrolyte loss
shock
inatequate circulation to brain, heart, lo 02 delivered
hypovoloemic shock cause
loss in volume
trauma, surgery, burns, FVD
cardiogenic shock
pump failure
MI
arrythmias
distributive shock
pooling of blood - massive vasodilation
neurogenic - spinal injury
allergy
septic
intervetions for shock
ABC!
oxygen
fluids
meds for shock
sympathomimetic (dopamin, dobutamine)
vasodilators (nitroglyerine)
diabetes insipidus
low ADH
cushings
hi aldosterone
addisons
lo aldosterone
parietal pleura
lines thorax
visceral pleura
lines lungs
what produces surfactant
alveoli
NIF
negative inspiratory force
ventilation
move air in and out
respiration
gas exchange at cellular level
inpiration
diaphram contracts (down)
thoracic cavity expands
negative inspiratory force
active
expiration
diaphragm relax (up)
thorax decreases
positive force pushes air out
passive
orthopnea
pillows at night
cyanosis
late sign of hypoxia
stridor
high pitched sound
medical emergency
usually obstruction
PEEP
usually 5-10 cm
keep open alveoli
inc delivery of O2
lower conc of O2 needed
pneumonia position
sit up
COPD
slow progressive destruction of alveoli
loss of lung elasticity
CO2 retained (r. acid)
decreased gas exchange
expiration becomes active

emphysema
chronic bronchitis
risk for COPD
cigs
air pollution
AAT deficiency
Sxs emphysema
SOB, shallow
chest rigid
productive cough
long expiration
wheezes
clubbing
R side HF
treat emphysema
stop smoking
increase fluid
position, 02 therapy
meds for emphysema
broncodilator: antolin, atroven (B2)
corticosteriods: beclovent
aminophylline
forced expiratory volume
how fast air can be moved in and out
forced vital capacity
how much volume can be moved in and out
sxs chronic bronchitis (4)
excessive mucus
cough
dyspnea
lasting 3 mo+ in 2 consec yrs
chronic bronchitis cause
smoking
recurrent lower RT infections
chronic bronchitis treat
no smoking
treat LRT at first sign
increase fluid to liquefy
asthma
reversible
bronchoconstriction
swelling of mucus lining
thick secretion
no anatomical changes
sxs asthma
cough
wheezeing
dyspnea
anxiety
pale color
diaphoresis
tachy pulse
hypoxia, hypercapnea
treat asthma
avoid triggers
oxygen
breathing exercises
comfort
asthma meds
beta 2 agonisits
corticosteriods
mast cell stabilizers
methylxanthines
leukotrienes