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42 Cards in this Set

  • Front
  • Back
psychopathology
sickness of the mind

-problematic patterns of thought, feeling, or behavior that negatively impact a person's social, academic, and/or occupational functioning

4 perspectives of psychopathology
1.psychodynamic

2.cognitive-behavioral


3.biological


4.systems

psychodynamic perspective
3 broad classes of psychopathology that form a continuum of functioning from minor disturbance (neuroses) to serious disturbance (psychoses):

1.neuroses


2.personality disorders


3.psychoses



cognitive-behavioral perspective
integration of classical and operant conditioning within a cognitive social perspective

-focus on discrete processes


-assesses the conditions under which simp


toms (depression and anxiety) arise out the


environmental stimuli that elicit them


-psychopathology reflects dysfunctional cognitions (automatic negative thoughts)

biological perspective
psychopathology disease of brain: focus on aspects of nervous system

1. disturbance of neurotransmitters


2.neural circuits


3. gross pathology of the brain


4.diathesis stress model



systems perspective
seeks the roots of psychopathology in the broader social context:

-explains the individual's behavior in the context of a social group (family)


-group functions as a system with independent parts, a change in one member influences other members (work as the system as a whole (family therapy) to solve the problem

DSM-5
diagnostic and statistical manual of mental disorders published by the American Psychiatric Association

categorical: groups similar disorders into 20 dif. categories and provides specific diagnostic criteria for each mental disorder (if you don't have one of the symptoms required you don't get diagnoses)


atheoretical:not connected to any theoretical perspective, compatible with a disease (medical) model


symptoms cluster into syndromes to study people, syndromes have specific etiologies (causes) and should be treated using different therapies

ADHD
symptoms of inattention and/or impulsivity persisting for at least 6 months to a degree that is maladaptive and inconsistent with developmental level (5% school-aged children, 2.5% adults, more in males)

risk factors: low socio-economic status, severe marital discord, material psychopathology inattention: non-responsiveness to task demands (easily distracted) hyperactivity: impulsivity-failure to inhibit behavior (no control, interrupting conversation) underarousal hypothesis: insufficient inhibitory control over sensory input and motor output treatment: stimulants (75-85% approval)


-symptoms have to be present at 2+ settings and must exist before age 12 -as AHDH children age hyperactivity may decrease but inattention may not -ADHD children at risk for other adolescent and adult problems



conduct disorder
childhood disorder in which a child persistently violates the rights of others as well as societal norms. Prevalence: 6-16% boys 2-9% girls Etiology:unstable home environment particularly dangerous to children genetically vulnerable. overly punitive or ineffectively lax parenting lead to delinquency. biological: reduced emotional and physiological arousal (much longer resting heart rate)thus less responsive to conditioning. Experience less anxiety when violating social norms
CD and ADHD comorbidity
many children with ADHD are also diagnosed with CD. Often problems with family systems. Treatment: multi model treatments that combine stimulant medication and family psychotherapy
schizophrenia
heterogenous clinical syndrome that involves a range of cognitive, behavioral, and emotional disfunction's associated with impaired social/occupational functioning. Usually emerges in late adolescence/ early adulthood. Occurs cross culturally in ~1% of population. prevalence: higher among poor, may reflect the effect of poverty on ppl vulnerable to SZ. Only 10-20% of people with SZ recover. ~50% relapse w/in 1 yr of leaving hospital. Long term prognosis is better for women.
negative symptoms of SZ
reduction in normal behavior/emotions

Affect flattening: restrictions in range and intensity of emotional expression. Alogia: restrictions in the fluency and productivity of thought and speech. Avolition: restrictions in the initiation of goal-directed behavior

positive symptoms of SZ
excess of sensory perceptions and ideas. delusions (false belief), hallucinations (false perception), disordered thinking (loose association)
etiology of SZ
-genes play primary role: 48% MZ twins, 17% DZ twins, 9% siblings

-neurotransmitter dopamine implicated: most antipsychotic drugs used to treat SZ block activity of dopamine


-SZ people more likely to have had prenatal and/or delivery complications

biological of SZ
-smaller frontal & temporal lobes
expressed emotion of SZ
family interactions characterized by criticism, hostile comments, and emotional over involvement/intrusiveness.

-living in home low in EE buffers people with SZ and contributes to their remission


-when people with SZ are exposed to high EE environment, more likely to relapse and require re-hospitalization

major depressive disorder
form of psychopathology characterized by depressed mood, loss of interest in pleasurable activities (anhedonia), and disturbances in appetite, sleep, energy level, and concentration.


prevalence of major depressive disorder
2-3% males; 5-9% females

lifetime risk: 10-25% females

biological or major depressive disorder
1. genetics: family history of depression doubles to triples a persons risk of development

2. neurotransmission: monoamine hypothesis. MDD associated with abnormalities in seratonin & norapinephrine


3. hormones: hypothyroidism and over activation of the HPA axis


4. brain regions: hippocampal damage (due to +cortisol). overactivity in right frontal lobe (-) under activity in left frontal lobe (+)

psychological of major depressive disorder
depressed ppl process information about themselves in a negative way automatically and implicitly, perceiving neutral or even positive info. as negative


Aaron Beck
cognitive distortion: cognitive mechanisms by which a depressed person negatively transforms info.

negative triad theory: interpret events unfavorably, do not like themselves, look @ future pessimistically

anxiety disorders
characterized by intense, frequent, or continuous anxiety, which may lead to disruptive avoidance behavior
biological of anxiety disorders
genetic (mz twins > dz twins)

neurophysiological abnormalities in seratonin and dopamine pathways in the limbic system and low levels of GABA - results in a tendency to be especially reactive in anxiety producing situations

behavioral of anxiety disorders
classical conditioning
personality disorders
stable, persuasive, and severely maladaptive ways of perceiving, relating to and thinking about world and oneself

1. cluster A: odd or egocentric


2. cluster B: dramatic, eccentric


3. cluster C: anxious or fearful

paranoid personality disorder
distrust and suspicious
schizoid personality disorder
detachment from social relationships, restricted range of emotional expression
schizotypal personality disorder
acute discomfort in close relationships, cognitive or perceptual distortions, eccentricity
antisocial personality disorder
disregard for and violation of the rights of others
borderline personality disorder
impulsivity and instability in interpersonal relationships, self-concept, and emotion
histrionic personality disorder
excessive emotionality and attention seeking
narcissistic personality disorder
grandiosity, need for admiration, lack of empathy
avoidant personality disorder
social inhibition and avoidance, feelings of inadequacy and hypersensitivity to negative evaluation
dependent personality disorder
submissive and clinging behavior and excessive need to be taken care of
obsessive-compulsive personality disorder
preoccupation with orderliness, perfectionism, and control
psychodynamic perspective:neuroses
minor problems that cause anxiety and mild interpersonal conflict (not extreme, most people have these can be resolved)
psychodynamic perspective: personality disorders
chronic disturbances that impact interpersonal and occupational functioning
psychodynamic perspective:psychoses
(most severe) psychotic disorder, can't understand what is real and what is not real
biological perspective: disturbance of neurotransmitters
(i.e., depression: abnormal levels of seratonin and noreprinephine)
biological perspective: neural circuits
(i.e., anxiety disorders are associated with heightened activation of an anxiety circuit in the angdala and frontal lobes
biological perspective: gross pathology of the brain
SZ is associated with atrophy of the frontal and temporal lobes
biological perspective:diathesis stress model
an individual displays a disorder when he/she has 1. an underlying vulnerability predisposition (diathesis) AND 2.when they experience some psychological or environmental disturbance (stress prenatal and post)