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36 Cards in this Set

  • Front
  • Back
1. Cholelithiasis
2. Cholecystitis
3. Cholecystectasia
4. Choledocholithiasis
5. Cholangitis
6. primary sclerosing cholangitis-
1. Cholelithiasis- stone in GB
2. Cholecystitis- inflammation of GB
3. Cholecystectasia- dilation of GB
4. Choledocholithiasis- stone in common BD
5. Cholangitis- inflam of bile ducts
6. Primary sclerosing cholangitis- fibrosis of intra/extra hepatic bile ducts. see in UC, presents with recurrent/presistent obstructive jaundice
1. biliary colic
2. HIDA
3. GB dyskinesia
4. Gallstone Illeus
1. biliary colic- pain bc of contraction/stones
2. HIDA- 99m technetium labeled hepatobiliary imaging
3. GB dyskinesia- poor ejection from GB
4. Gallstone Illeus- sm intest obstruction due to large stone that errodes through GB into the duodenum, producing cholecystomenteris fistula and then gets into small bowel
1. what is a stone in GB
2. what is inflammation of GB
3. what is dilation of GB
4. what is removal of GB
5. what is stone in common bile duct
6. what is pain when GB contracts
7. what is inflammation of biliary tree
8. what is when the GB contracts bad
9. what is used to see the biliary system
10. what is it called when a gallstone gets into sm intestion and blocks it
1. Cholelithiasis
2. Cholecystitis
3. Cholecystectasia
4. cholesystectomy
5. Choledocholithiasis
6. biliary colic
7. Cholangitis
8. biliary dyskinesia
9. HIDA
10. gallstone illeus
what are the types of gallstones
1. Cholesterol

2. Pigment

3. Ca Carbonate

4. Mixed:
what are the 5 F of gallstone disease
1. female
2. fart
3. fat
4. fertile
5. forty
what makes up Charcots Triad and what does it suggest, how about Reynolds Pentad
Charcots:
1. RUQ pain
2. fever w/chills
3. jaundice

*

Reynolds
1.
2.
3.
4.
what nationality gets more cholelithiasis
women, native americans- Pima

Obesity and DM are factors
what are stones make of
cholesterol
lecithin
bile acids

**when bile salts decrease and cholesterol is increased --> ppt out stones
risk factors for cholelithiasis
obesity, cholesyscetomy in 1st degree relative, native america, old, women, rapid weight loss, glucose intolerance, increased insulin levels, ileal resection, chrons, DM, hypertriglyceridemia, cirrhosis, drugs, long fast (turn to slugde bc GB wont contract)
what can decrease gallstone formation
low carb
high fiber
low fat
exercise
caffeine in coffee

*vit c, NSAIDS, ASA
what is the main component in the following type of gall stones

1. pigment
2. cholesterol
3. Ca Carbonate
4. Mixed
1. Pigment: Ca bilirubinate, uncong bili ppt, Fe from bili lights them up on x ray

2. cholesterol- duh, its from excess cholesterol in bile. common in women fatties who have had kids, DM

3. Ca Carbonate: uncommon in adults, more common in kids

4. mixed-
what are sx of cholelithiasis
transient
related to eating fatty meals, when BG contracts it hits cystic duct and causes pain

NV, bloat, fart, RUQ pain, R shoulder pain

NO peritoneal signs: not localized, no rebound tenderness
whats on the ddx of biliary colic
1. cholycystisis
2. PUD
3. hepatitis
4. pneumonia
what is dx test for gallstones
US- will have acoustic shadow

** can be (-) if pts pain persits and your suspicion is high can do endoscopic US or HIDA US with CCK- CCK makes GB contract, wth stones EF is low
do you see acoustic shadow on US for stones or polyp
stones
tx for gall stones
NPO- dont want to stim GB, NSAIDS, wait it out.

**can do surgery, elective. low fat high fiber diet in teh meantime
what is the prognosis of taking orals to dissolve gall stones
not great
tell me about acute cholcystitis
inflammation of GB

PERSISTANT pain, + murphy sign, push your hand up the GB and pt stops breathing

pain to R scapula

peritoneal signs present: guarding, q
what are hte labs for acute cholecystitis
guarding
murphy
US- thick GB wall, pericholecystic fluid, stones +/-
whats HIDA
dye that follows bile, can tell you where an obsturction is

can also use CCK adn see teh ejection fraction
whats the complication of acute cholecystitis
gangrene- most common
perforation
cholecystenteric fistula
gallstone ileus
emphysematous cholecystitis
tell me about choledocholithisasis
stone in common bile duct- pain, NV

obstructive jaundice: dark pee and pale pooleukocytosis

liver fx tests are high adn LOTS of cong bili, alk phos increased. GGT high
when is cong bili high, GGt high and liver enzymes high
stoned in common bile duct

choledichilithiasis
what is ERCP
endoscopic retrograde cholangiopancreatography

*great way to see the cause location and extent of obstruction
*make dx, take out stone and place stent

complications of procedure include pancreatitis (HIDA risk was bile duct injury
do choecystectomy asap if you have stones in common bile duct (choledocholithiasis) T or F
treu
WHAT IS Acute cholangitis
inflammation of biliary duct system

ascending infection of biliary bc of obstruction in: common bile duct, or strictures

potentially fatal

caused by E coli, klebsiella, enterobacter
what signs tell us there is a high morbidity in acute cholangitis
charcots: pain, fever, jaundice
Reynolds
mental status change
hypotension
what is reynolds pentad
charchots (pain, jaundice, fever)+ alterd mental status + shock

**suggests ascending cholangitis
what aer hte labs assoc w/acute cholangitis
leukocytosis w/L shift
increased alk phos
increased GGT
increased direct bili
US: common BD stone/dilation

tx w/ERCP
can gallstones cause pancreatitis
yep

tx w/GB removal
flow chart

acute biliary pain

where do you go if it is short lived w/no fever or leukocytosis

what about prolonged with fever, murphy, leukocytosis
1. billiary colic, gallstones

2. acute cholcystitis
GB thickened pericholcystatic fluid increased WBC gever, gallstones, pain
acute cholcystitis

**tx with cholecystectomy
RUQ pain w.no GB thickening or fever or infection and stones presnet
cholethiasis
Gallstones present, increased bili, increased alk phos, dilated biliary tree
choledocholitsis
no gall stones, decreased EF
biliary dyskinesia
what does GB cancer look like
porcelin (calcified)
not common