Glaucoma Meds

Front 1

Classifications of Glaucoma Meds

Back 1

adregenic agents - sympathomimetics
beta adregenic blockers
carbonic anyhdrase inhibitors
hyperosmotic agents
adenergic egents - alpha agonists
prostaglandins

Front 2

Miotics have _______ caps

Back 2

green

Front 3

Most common miotic

Back 3

pilocarpine

Front 4

pilocarpine is given ________ /day

Back 4

QID (4x)

Front 5

Pilocarpine Ocuserts are

Back 5

wafers worn under the upper lid for 1 week

Front 6

Phospholine Iodide may cause

Back 6

iris cysts

Front 7

Pilopine HS is the only _______ form miotic

Back 7

gel

Front 8

Adrenergic agents come in these two forms

Back 8

sympathomimetics
alpha 2 agonists

Front 9

sympathomimetics work by

Back 9

improving aqueous outflow, dilating pupil

Front 10

these may leave epinephrine deposits

Back 10

sympathomimetics

Front 11

Examples of sympathomimetics

Back 11

epifrin, eppy N, glaucon, Dipivefrin, propine

Front 12

Dilations of pupil may be harmful in ___________ patients

Back 12

narrow- angle glaucoma patients

Front 13

Alpha 2 agonists are similar to _________ but more effective

Back 13

eppy

Front 14

Adrenergic agents - Alpha 2 agonists mostly end in with -_______

Back 14

-idine

Front 15

Apraclonidine (Iopidine) is for ________ therapy

Back 15

short term

Front 16

This alpha 2 agonists has a 75% incidence of allergy

Back 16

apraclonidine (Iopidine)

Front 17

Brimonidine (Alphagan) is for __________ term use

Back 17

long term

Front 18

Beta Blockers make ______ worse

Back 18

asthma

Front 19

Beta Blockers work by ________ aqueous production

Back 19

decreasing

Front 20

Side effects of beta blockers

Back 20

bradycardia, wheezing, hypotension, CNS problems, nightmares, psychosis

Front 21

Beta Blockers end in -_____

Back 21

-ol

Front 22

Betaxol (Betoptic) has less _________ effect

Back 22

repiratory

Front 23

Timolol was the ________ beta blocker

Back 23

first

Front 24

Beta blockers with 0.25% are ______ capped

Back 24

blue

Front 25

Beta blockers with 0.50% are ______ capped

Back 25

yellow

Front 26

Hyperosmotic agents move fluid into the ________

Back 26

bloodstream

Front 27

Glycerin (Osmoglyn) is a

Back 27

hyperosmotic

Front 28

Mannitol (osmitrol) has an extremely _______ taste

Back 28

sweet

Front 29

If your patient is diabetic you would want to avoid giving them this hyperosmotic

Back 29

glycerine

Front 30

Carbonic anhydrase inhibitors __________ the production of ______

Back 30

decrease, aqueous

Front 31

One can determine if a pt. has been taking their CAI's by asking if ___________

Back 31

they have paresthesias

Front 32

Side effects of CAIs

Back 32

kidney stones, tingling, weakness, impotence, GI problems

Front 33

Systemic CAIs

Back 33

acetazolamide ( diamox), methazolamide (neptazane), Dichlorphenamide (daranide)

Front 34

Acetazolamide (diamox) can be administered in ______ and _______ forms

Back 34

pill and IV

Front 35

Methazolamide is _______ tolerated

Back 35

better

Front 36

Topical CAIs

Back 36

Brinzolamide (Azopt), Dorzolamide (Trusopt)

Front 37

Topical CAIs have _______ caps

Back 37

gold

Front 38

CAIs end with -__________

Back 38

-amide

Front 39

Combigan is a combination of this beta blocker and alpha agnoist

Back 39

Timolol, brimonidine

Front 40

Prostaglandins end with -________

Back 40

-prost

Front 41

Prostaglandins have ______ tops

Back 41

teal

Front 42

Given qd (1x day)

Back 42

Prostaglandins

Front 43

Examples of prostaglandins

Back 43

latanaprost (xalatan), bimatoprost (lumigan), travoprost (travatan), unoprostone isopropyl (rescula)

Front 44

Side effects of prostaglandins

Back 44

iris color change, longer lashes, conjunctiveal hyperemia

Front 45

Glaucoma is associated with

Back 45

RGC death

Front 46

Alphagan has a ______ top

Back 46

purple

Front 47

Trusopt (Dorazolamide) and TImolol make up

Back 47

Cosopt