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55 Cards in this Set

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  • Back
  • 3rd side (hint)
what is the maximal duration of ARS?
which sinus issue can last up to 4 weeks?
what is ABRS?


what is a secondary bacterial infection of paranasal sinuses, usually following a viral URI


- relatively rare


prior to choosing an antimicrobial therapy, what are questions to ask?

to which situation to the following questions apply:


- what is/are the most common pathogen(s) causing this infection?


- what is the spectrum of a given antimicrobial's activity?


- what is the likelihood of a resistant pathogen?


- what is the danger if there is treatment failure?


what is the most common causative bacterial organism for ABRS, AOM and CAP?
S. pneumonia is the most common bacterial pathogen for which maladies?
Gram + diplococci, ABRS causative organism in adults 38%, children 21-33% - describes which organism?
what is the gram stain, morphology, and percentage of ABRS caused by S. pneumonia?
what is the percentage of drug resistance (DRSP) for S. pneumonia?

which organism has >= 25% DRSP?

in patient's with recurrent infection and tobacco use, what is the most common pathogen for ABRS, AOM and CAP?

H. influenza is the most common pathogen for which maladies especially under which patient conditions?

which common pathogen is a G - bacillus?

H. influenzae has what gram stain and morphology?

>= 30% PCN resistance via beta-lactamase is characteristic of which organism causing ABRS, AOM and CAP?

H. influenzae has which % of resistance using which resistance mechanism?
what is the less common pathogen associated with ABRS and AOM and an uncommon cause of CAP?
M. catarhallis is a less common pathogen causing which maladies?
Gram negative coccus uncommonly associated with ABRS, AOM and rarely associated with CAP

M.catarhallis has what gram stain and morphology?
which pathogen has >= 90% PCN resistance via beta-lactamase production?

M. catarhallis has what % of abx resistance to which abx?

in the treatment of ABRS, under what three conditions do you consider initiating antimicrobial therapy?

under the following conditions of ABRS, what is your next move?


- persistent and not improving >= 10 days


- severe >= 3-4 days


- worsening or double sickening >= 3-4 days

what is a special consideration for the following populations when choosing abx?


- < 2 yo or >65 yo


- prior abx w/in the past 2 months


- prior hospitalization w/in past 5 days


- comorbidities


- immunocompromised


what are patient characteristics that put them at risk for antimicrobial resistance?

when is the appropriate time to use intranasal corticosteroids in ABRS?

ABRS + allergic rhinitis allows the use of which medication for symptom relief?
what is first line therapy for ABRS?
amoxicillin-clavulanate (875/125 BID or 500/125 TID) is which line therapy for which malady?
what is second line therapy for ABRS?

amoxicillin-clavulanate (2000/125) or doxycycline (100 BID or 200 daily) is which line of therapy for which malady?
what is/are the indicated therapy/ies for ABRS in a pt with a beta-lactam allergy?


in the treatment of which malady in a pt with this allergy, these are the indicated therapies:




- doxycycline (100 BID or 200 daily)


- levofloxacin (500 daily)


- moxifloxacin (400 daily)



in ABRS, if there is a risk for antibiotic resistance or failed initial therapy what/which antibiotic(s) are recommended?


- Amoxicillin-clavulanate 2000/125


- Levofloxacin 500 daily


- Moxifloxacin 400 daily


are indicated for ABRS treatment under which circumstances?

what are first line treatments for allergic rhinitis (AR)?

intranasal corticosteroids (-sone, -sonide: Flonase, Nasacort, nasonex) are first line treatment for which malady?
leukotriene receptor antag (LTRA) or leukotriene modifiers (LTM) aka montelukast (Singulair) is best for AR when used alone or in combination with this other rx?

antihistamines work well in combination with which med(s) for AR?
mast cell stabilizers (cromolyn) are more or less effective than intranasal corticosteroids for control therapy of AR?

this class of AR controller medications is less effective than intranasal corticosteroids for sx relief BUT their ocular formulation are effective for ocular sx
what are the three major classes of reliever (rescue) therapy for AR?


- antihistamines (oral, nasal, ophthalmic)


- decongestants


- anticholinergics


are all which types of meds for AR?


for relief of nasal congestion in AR, which class of medications are most effective?

intranasal corticosteroids are most effective for relief of which AR symptom?


- nasal antihistamines are less effective

examples of second generation oral antihistamines include?

loratadine (Claritin), desloratidine (clarinex), cetirizine (Zyrtec), levocertrizine (Xyzal) and fexofenadine (Allegra) are all examples of which meds used in the treatment of AR?

when are short-term PO corticosteroids indicated for use in AR?

which medication(s) used in AR indicated for severe or intractable nasal symptoms or significant polyposis?
what are adverse effects of anticholinergics?


what class of meds cause the following sx:


- dry as a bone


- red as a beet


- mad as a hatter


- hot as a hare


- can't see


- can't pee


- can't spit


- can't (something that rhymes with spit [constipation])

which CN controls the puff out your cheeks?

CN VII controls which common neuro assessment technique?
which CN controls using eyes to follow fingers without moving head?
CN III controls which common neuro assessment?
which CN controls shoulder shrug?

CN XI controls which common neuro assessment?
which CN controls stick out tongue?

CN XII controls which neuro assessment?

what are the physical exam findings of a normal TM?

these describe a TM in which state?




- pale, gray, translucent appearance


- cone of light and bony landmarks visible


- mobile with pneumatic otoscopy

what are the physical exam findings of a TM with otitis media with effusion (aka serous otitis)?

these describe a TM in which state?




- air-fluid level visible (often with air bubbles)


- opaque yellow or blue color


- cone of line and bony landmarks diminished or absent


- TM mobility with pneumatic otoscopy limited



what is the treatment for otitis media with effusion (aka serous otitis)?

Tx of underlying cause (such as allergic rhinitis) and/or spontaneous resolution 1-3 weeks without special intervention

what are the pt statements in their history regarding otitis media with effusion (aka serous otitis)?

with this middle ear pathology, the pt will c/o:


- ear fullness or pressure


- otalgia or ear itch


- conductive hearing loss


- no fever or otorrhea

what are the pt statement(s) in their history regarding acute otitis media?

with this middle ear pathology, pt will c/o:


- otalgia


- ear fullness, pressure


- conductive hearing loss


- fever (common)

what TM findings are common with acute otitis media?

what middle ear pathology has these PE TM findings?


- TM redness


-TM bulging


- cone of light and bony landmarks absent


- absent TM mobility with pneumatic otoscopy


- otorrhea possible with TM rupture

what is the treatment for acute otitis media?

this/these are the tx(s) for which middle ear pathology?


- analgesia, ABX


- however, high rate of spontaneous resolution without abx tx

on funduscopic exam, the finding of a deeply-cupped optic disc indicates ?

what is the funduscopic exam finding of acute angle-closure glaucoma?

which of the following ophthalmologic conditions is considered an emergency:


open angle glaucoma or angle closure glaucoma?

angle closure (aka acute angle closure) glaucoma is considered an emergent or non-emergent eye condition?

which of the following ophthalmologic conditions is considered a chronic condition:


open angle glaucoma or close angle (or angle closure) glaucoma?

open angle glaucoma is considered a chronic condition or emergency?

when is surgical intervention indicated for acute angle closure glaucoma?

surgical intervention is indicated once the IOP is normalized in which type of glaucoma?

AV nicking is a funduscopic examination finding in which pathological state?

on funduscopic examination of a patient with longstanding uncontrolled HTN, what would you expect to find?

Papilledema on funduscopic examination indicates which pathological state?

Increased ICP is indicated by which funduscopic finding?

untreated open angle glaucoma is at times called the "silent thief" - why?

slow progressive peripheral vision loss esp in the elderly is usually caused by which pathological state?

the silent thief

what two types of medication are used for the tx of open angle glaucoma?

reduce production of intraocular fluid


- topical beta-adrenergic antagonists


- topical alpha-2 agonists


- less selective sympathomimetic


- topical carbonic anhydrase inhibitors




increased fluid outflow


- prostaglandin analogs


- miotic agents (parasympathomimetics)

what is the treatment for acute angle closure glaucoma?

treatment for which type of glaucoma includes:


- prompt ophthalmologic referral


- relief of acute intraocular pressure (eyedrops: beta-adrenergic antag, alpha-2 agonist, carbonic anhydrase inhibitors)


- increase fluid outflow (prostaglandin analog, hyperosmotic agents)


-SURGERY once IOP is normalized

components of the eye emergency triad?

- acute vision change


- eye redness


- eye pain




constitute which eye malady?

what are the three eye emergencies?

trauma, anterior uveitis (aka iritis) and angle closure glaucoma all have what in common?

what is the clinical presentation of anterior uveitis (aka iritis)?

- keratic precipitates in cornea (white patches in a red cornea)


- pupil usually constricted


- irregularly shaped pupil


- perilimbal injection (ciliary flush)




are indicative of which ophthalmologic emergency?

what is the intervention for anterior uveitis (aka iritis)?

- referral to ophthalmology


- topical or systemic corticosteroids


- cycloplegics


- more intervention determined by etiology bc this is often r/t autoimmune dz or rxn (ankylosing spondylitis, IBD, reactive arthritis [Reiter's syndrome], psoriatic arthritis)




are the interventions for which eye emergency?

what is the PE findings for angle-closure glaucoma?

- slit-lamp evaluation may reveal corneal edema, synechiae, corneal edema


-irregular pupil shape


- segmental iris atrophy


- cornea and scleral injection


- ciliary flush (red sclerae emerging from iris)


are PE findings for which ophthalmologic emergency?

amsler grid test is used to determine which pathological state?

early detection of macular degeneration is detected by which screening test?

what are the interventions necessary for acute angle closure glaucoma?

- referral to ophthalmology


- block aqueous production, reduce vitreous volume, facilitate aqueous outflow with Diamox (actetazolamide), topical beta blocker, and pilocarpine

tonometry is what?

what is the test for glaucoma (inc IOP)?