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104 Cards in this Set

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Defining characteristics of ARF?
PaO2 <80 (hypoxemic)
PaCO2 >45 + acidosis (hypercapnic)
COPD w/severe ABG deterioration & clinical deterioration
What is Alveolar hypoventilation? And causes?
deficient movement of air into & out of the alveoli (usually from obstruction)
other causes- restrictive lung diseases, CNS disease/depression, chest wall dysfunction (pneumothorax), acute asthma, neuromuscular disease
What is V/Q mismatch (hypoxemia)?
ventilation/perfusion mismatch
decreases ventilation w/normal blood flow (increased secretions in airways/alveoli, bronchospasm, atelectasis, pain)
OR
normal ventilation w/decreased blood flow (PE)
Tx of V/Q mismatch?
tx underlying cause
Consequences of untx ARF in each system?
Resp- Increased rate at 1st, decreased w/worsening & muscle fatigue
Cardio- tachycardia, decreased CO
CNS- permanent brain damage
Renal- Acute tubular necrosis
GI- ischemia, bacterial translocation
2 major consequences of untx of ARF?
hypoxemia - tissue hypoxia - anaerobic metabolism & lactic acidosis = need NaHCO3 to buffer
renal hypoxia - release of erythropoietin from renal cells - bone marrow increases RBC to attempt to increase the blood's O2 carrying capacity- may lead to ATN
Early S&S of ARF? And tx?
dyspnea, restlessness, anxiety, H/A, fatigue, cool & dry skin, HTN, tachycardia
tx- elevate HOB, O2
Intermediate S&S of ARF? And tx?
confusion, lethargy, tachypnea, hypotension, dysrhythmias, ischemia
tx- Elevate HOB, O2 NC, may need to intubate
Late S&S of ARF? And tx?
cyanosis, diaphoresis, coma, resp arrest
tx- intubate, call code
What is a PE?
obstruction of the pulmonary arterial bed by a dislodged thrombus, fat or air embolus, heart valve growths or a foreign substance
What is the patho of PE?
vascular wall damage, venostasis, & hypercoagulability of blood cause thrombus formation, thrombus loosens from trauma, clot dissolution, muscle spasm, intravascular pressure change, or peripheral blood flow change, embolus travels to the R side of the heart, enters the lung through the pulmonary artery, embolus dissolves, fragments or grows
What happens with an untx PE?
alveoli are prevented from producing enough surfactant to maintain alveolar integrity, alveoli collapse = atelectasis
large enough embolus will clog pulmonary vessels = death
Complications of PE?
pulmonary infarction
pulmonary HTN
S&S of PE?
sudden unexplained dyspnea- hallmark sign
angina, pleuritic chest pain, tachycardia, air hunger, impending doom, productive cough that may be blood tinged
Less common S&S of PE?
low grade fever, pleural effusion, massive hemoptysis, chest splinting, lower extremity edema, cyanosis, syncope, distended neck veins, pleural friction rub, circulatory collapse, hypoxia
Risk factors of PE?
long car trips, long plane trips, cancer, pregnancy, hypercoagulability, birth control, previous DVT or PE
Dx of PE?
CXR, V/Q scan, pulmonary angiography, EKG, hemodynamics, ABGs, D-Dimer
What do you see on a CXR for a PE?
may see PE or other pulmonary conditions
What do you see on a V/Q scan?
reveals perfusion defects beyond occluded vessels
injection of radio-isotope, inhalation of radioactive gas
can't do on an intubated pt
Pulmonary angiography for PE?
most definitive test
insert cath through antecubital or femoral vein to pulmonary artery where contrast is injected
most beneficial when anticoagulation (heparin) is contraindicated
EKG for PE?
used to distinguish PE from MI
Expected hemodynamics for PE?
elevated pulmonary systolic pressure, 20-30
diastolic pressure, 8-15
mean pressure, 10-20
& wedge pressure 6-12
if no wedge pressure ordered go off of diastolic pressure
Early ABG results from PE?
hypoxemia
hypocapnia
resp alkalosis
(CO2- <35, pH > 7.45)
ABG if embolus is large?
hypoxia, hypercapnia, resp acidosis
How to tx Shunting?
intubate, increase O2, increase peep
What is SaO2?
% of O2 on Hgb
90-100
What is PaO2?
O2 dissolved in arterial blood
80-100
D-Dimer for PE?
measures amount of cross-linked fibrin fragments after clotting event ( DVT, acute MI, angina, acute CVA)
not specific or sensitive to PE or clot anywhere
When do you use an IVC filter?
when you can't use anticoagulants
Best tx for PE?
1st admin O2 then give heparin 60-70
if levels high, stop for 1 hr
decrease rate
Other tx for PE?
O2 prn, Heparin, SCDs, fibrinolytics, surgery
What is Pulmonary edema?
accumulation of fluid in extravascular spaces of the lungs
extravascular spaces = interstitium & alveoli
What are the causes of Pulmonary edema?
L sided heart failure d/t arteriosclerosis, cardiomyopathy, HTN, valvular heart disease
Patho of Pulmonary edema?
changes in pulmonary capillary hydostatic pressure, capillary oncotic pressure, capillary permeability, & lymphatic drainage
prevents fluid infiltration into the lungs because the fluid leaks into the interstitial space & alveoli causing impaired gas exchange & edema
What are the Early S&S of Acute pulmonary edema? And tx?
S3 audible- hallmark
exertional dyspnea, paroxysmal nocturnal dyspnea, orthopnea, cough, mild tachypnea, HTN, basilar crackles, tachycardia
O2, elevate HOB, Lasix
What are the Late S&S of APE & tx?
labored, rapid resps, diffuse crackles, blood tinged frothy sputum, increasing tachycardia, arrhythmias, cold clammy skin, diaphoresis, cyanosis, hypotension, weak pulses
Pt is drowning r/t forced fluid in alveoli
Dx for APE?
ABGs, CXR, Pox, Swan, EKG
ABGs will show what for an APE?
hypoxemia, & variable PaCO2
PaCO2- dependent on pt's level of fatigue, increased fatigue = increased CO2
CXR of APE will show what?
diffuse haziness (whited out) of lung fields, cardiomegaly r/t increased pumping, pleural effusion
Normal Cardiac output & cardiac index?
4-8L
2-4 (pt specific)
Pulse Ox will show what w/APE?
decreasing SaO2
Swan will show what for APE?
shows L sided heart failure & can be used to r/o ARDS
decreased CO & CI
EKG for an APE?
previous or current MI
Tx for APE?
elevate HOB, high concentrate O2 usually by NC (pt can't tolerate face mask, feels like they're suffocating more), meds
if can't tx soon enough intubate & mechanical vent
Meds for APE?
diuretics (Lasix), positive isotropes, vasopressors (Levofed), antiarrhythmics (Cardizem), arterial vasodilators (Tridil, nitropreside), Morphine, Natrecol, venous dilator (nitroglycerin IV)
Preload affects what?
fluid
Afterload affects what?
pressure
What is ARDS?
pulmonary edema in the absence of cardiac failure
NO L SIDED HEART FAILURE- NOT CAUSED BY L VENT FAILURE
What is primary pulmonary insult?
directly affects lung tissue/alveolar membranes
aspiration, near drowning, toxic inhalation, pulmonary contusion, pneumonia, thoracic trauma, O2 toxicity
What is secondary pulmonary insult?
Anywhere but the lungs
sepsis, non-thoracic trauma (immobility), excessive blood transfusions, pancreatitis, shock, PE, DIC, opioid overdose
What is the patho of exudative phase?
pulmonary capillary wall damage, fluid leaks into interstitial space, impaired gas exchange r/t widening of gap between alveoli & pulmonary capillaries, increased fluid volume that increases pressure exerted on alveoli causing atelectasis w/decreased lung volume
R-L shunt in Exudative phase?
blood passes from R to L w/o being oxygenated causing hypoxemia, hypoxia, multi system organ faliure
S&S of exudative phase in ARDS?
restless, apprehensive/afraid, progressive dyspnea & tachycardia, moderate use of accessory muscles, clear breath sounds, ABG of resp alkalosis & normal PaO2
Patho of Proliferative phase?
decreased lung compliance, decreased surfactant production, & refractory hypoxemia
Proliferative phase S&S of ARDS?
increased resp distress, increased tachypnea, increased accesory muscle use, fine crackles, mental status changes/agitation, resp acidosis
Patho of Fibrotic phase of ARDS?
irreversible, diffuse interstitial & alveolar fibrosis, increased dead space vent, increased hypoxemia & hypoxia, multiple organ dysfunction syndrome
Fibrotic S&S of ARDS?
increased tachycardia, hypotension, cool mottled ischemic skin, renal & liver failure, severe agitation, hallucinations, coma, death
Long-term effects in recovery phase of ARDS?
permanent loss of lung tissue, fibrotic = can't change
mild-moderate diminished vital capacity
impaired gas exchange
difficulty exercising, socializing & completing ADLs
Dx of ARDS?
decreased PaO2 despite increased FIO2- hallmark
CXR- diffuse, bilateral infiltrates
NO CHF
Tx of ARDS?
mechanical vent
goal- normal PaCo2
PEEP
Good causes of PEEP in ARDS?
decreases alveolar collapse, increases Functional Residual Capacity, increased lung compliance, decreased R-L shunt, increased clearing of lung fields
Risks of PEEP in ARDS?
increased alveolar distention causing weakness of alveolar walls causing rupture & decreased CO
Other tx of ARDS?
coughing, hyperoxygenate prior to in-line suctioning, turn, prone, specialty tube feedings, infection control techniques
DON'T INTERRUPT PEEP
Meds for ARDS?
antibiotics, corticosteroids, vasodilators, bronchodilators, mucolytics, diuretics, colloids, opioids/sedatives/neuromuscular block (Nimbex)
Cause of SARS?
coronavirus
How is SARS transmitted?
person-person contact
droplet precautions
DX of SARS?
pt hx (travel to area w/SARS, close contact w/other person w/SARS)
CXR- hazy opacities/ground glass appearance, progression to bilateral consolidation in 24-48hrs, culture from blood, stool, nasophary or orophary
Tx of SARS?
antipyretics, O2, vent support, IV fluids, antibiotics, antiviral, corticosteroids
Isolation precautions for SARS?
negative pressure
full barrier- gloves, gown, resp mask, eye shield
put hands together after leaving room to go wash hands
What is a Pneumonectomy?
removal of entire lung
empty space fills w/fluid
usually w/thoracoplasty
What do I do for a pneumonectomy & chest tube?
NO CHEST TUBE
Lay pt good lung up
What is a Lobectomy?
removal of lobe/lobes
lung over expands filling entire cavity
What do I do after lobectomy?
turn pt onto both sides
2 CHEST TUBES
What is an open lung biopsy?
resection of a small portion of lung for biopsy
CHEST TUBE
What is Decortication?
removal of visceral pleura
CHEST TUBE
What is a thoracotomy?
incision in thorax
What is VATS?
dx procedure
scope w/camera & video projector
biopsy specimen
CHEST TUBE
What are the benefits of VATS?
decrease blood loss, less invasive, decrease post-op rehab
2 types of thoracotomy & what are they?
median sternotomy- smaller tubes, split sternum; used for heart pts
posterolateral- anterior axillary line in 4, 5, 6 intercostal
anterolateral- sternal border to mid axillary line used for trauma pts
What to teach pt Pre-op for thoracotomy?
cough & deep breath techs, incentive spirometry, pain/comfort, stop smoking
What to teach pt post-op for thoracotomy?
pain, chest tube, arm movement, frequent assessment of breathing
What is a thoracentesis?
drains fluid from pleural spaces, test fluids for dx, if pneumothorax may need chest tube, monitor for pneumothorax
Post op care for lung surgeries?
assessment, auscultate, S&S hypoxemia/hypoxia, RR, ABG, oxygenation & ventilation, incentive spirometry, no suction unless absolute necessary, pain measurement using CPOT or WILDA, prevent atelectasis w/pt getting OOB, coughing & deep breathing, pain control, hydration
Post-op complications?
ARF, bronchopleural fistula, hemorrhage, subcutaneous emphysema, cardiovascular disturbances, pulmonary edema
Stages of bubbling?
initial bubbling-ok
intermittent bubbling
continuous bubbling- persistent air leak
fluctuations = tidaling
Nursing care w/chest tubes?
mark drainage levels, keep system below chest, monitor drainage amounts, check dressings, monitor crepitus, tubing, assess lungs sounds & PaO2, suction @ -20
Emergency equipment for chest tubes?
vasoline gauze, sterile water, clamps
Complications of chest tubes?
no drainage= malpositioning, kinks, suction, tubing, no milking or stripping
can gently manipulate
D/C of chest tubes when?
< 5% pneumo, no air leak, drainage, CXR
Procedure for D/C chest tubes?
explain procedure, medicate, position, MD removes, occlusive dressing, CXR
What to monitor for after chest tube removal?
bleeding, fluid leak, air leak, resp distress
Fx rib sites & areas affected at each site?
1 & 2- great vessels & brachial plexus
middle ribs- lung injuries
lower ribs- abd injuries
R sided 8th & below ribs- liver
L sided 8th & below- spleen
Dx of rib fx?
CXR
Complications of rib fx?
atelectasis, puncture/pneumothorax, spleen & liver injuries, flail chest, hemothorax, pneumonia
What is flail chest?
loss of stability r/t multiple rib fxs on 2 or more places fx on 1 rib
paradoxical resp- hallmark sign
possible contusion or hemopneumothorax
Tx for Flail chest?
stablize chest segment- no surgery/external fixation, mechanical vent, lie on affected side, IV fluids, pain control
What is a closed pneumothorax?
rupture in visceral or parietal pleural & chest wall
air source inside chest that separates 2 pleura & lung recoils by collapsing
What is a spontaneous pneumothorax & S&S?
unexpected air leak into visceral pleural space that can cause tension pneumothorax (life threatening)
sudden SOB at rest w/o exertion- hallmark
decreased breath sounds or absent
Tx for pneumothorax?
Chest tube
pleurodesis/mechanical abrasion, partial pleurectomy
What is an open pneumothorax?
air enters parietal pleural space through opening in chest wall causing barometric pressure in pleural space
What is a tension pneumothorax?
rapid accumulation of air in pleural space w/tension on heart & great vessels
one way valve effect, air trapping occurs
TRUE EMERGENCY
Tension pneumothorax can cause?
mediastinal shift or tracheal deviation
Tx for pneumothorax & hemothorax?
CHEST TUBE
Tx for tension pneumothorax?
medical emergency
needle decompression- 14G stick in chest to let air out