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76 Cards in this Set

  • Front
  • Back

respiration

not simply breathing, is a complex process involving several events

events of respiration

1. inspiration or inhalation- taking air in


2. exchange of O2 from air in alveoli (lung sac) to the blood to needy cells


3. O2 combines w/ hemoglobin


4. hemoglobin transports O2 to the general area of cells in need (based on bohr effect)


5. O2 is now released into blood plasma


6. plasma will now exit & carry O2 out of blood into interstital spaces


7. O2 will enter cells by diffusion


8. O2 is now utilized by needy cell in aerobic cellular respiration


9. CO2 (waste product of aerobic cellular resp.) will now diffuse into the blood & be carried back to the lungs (alveoli)


10. exchange of CO2 gas from blood to air (by diffusion)


11. expiration (exhalation) breathing out

3 different ways CO2 is carried

1. 67% will be carried as part of bicarb ion


2. 25% will be carried bound to amino acid chains of hemoglobin


3. in solution not chemically combined

chemically ways CO2 is carried

1. 67% will be carried as part of bicarb ion

2. 25% will be carried bound to amino acid chains of hemoglobin

substances found in inspired air




3 gases

O2


CO2


N2

substances in (inhaled air)

O2 20%


CO2 trace (less than 1%)


N2 79%

substances out (exhaled air)

O2 15%


CO2 5%


N2 79%

N2 in 79%, out 79% -though we need it,


we cant use it, we get nitrogen gas from

the food we eat

other substances found in inspired air

1. dust


2. pollen


3. spores


4. pollutants (gas, solid, or liquid)

Nostril aka external nares

lined w/ mucus secreting cilated epithelium


*they are fringed w/ course hair



nasal mucus

swallow ab 1 pint a day- nonspecific defense



do to air before it goes down



*air has to be hydrated- from blood supply (vascular)


* warm it - blood flow/friction (vascular)


*filter it - mucus, cilia (push mucus up & back) nasal hairs, nasal conchae

nasal conchae

projections of the ethmoid bone that cause the air stream to become turbulent as it goes down enhancing filtration

nasal septum

composed of nasal bone & cartilage, it bilaterally separates the 2 nasal cavities

deviated septum
Sideways displacement of the wall between the nostrils. interferes with all the ways air goes down

paranasal sinus

air filled spaces found in frontal bone, ethmoid, spinoid, & maxilla

purpose ofparanasal sinus

lighten the weight of the skull


* involved in the process of phonation

phonation

voice production

sinus headache

due to paranasal sinus-adjacent to nasal cavity lined w/ tiny mucus secreting openings..if blocked mucus keeps building within them, pressure building up and results in____

pharynx AKA throat

1.passage way for food & liquid traveling to esophagus


2. passage way for air into trachea

pharynx is divided into 3 regions

1. nasopharynx- behind nasal cavities


2. oropharynx - posterior to mouth


3. laryngopharynx - surrounding openings into esophagus & trachea



larynx AKA adams apple

highly cartilaginous. moves upward when you swallow causing (flap)epiglottis to come down.


*contains vocal cords, involved in voice production


*prevent air from leaving lower respiratory structures bc air volume maintains air pressure



epiglottis
flap, prevents food & liquid from entering trachea

high air pressure

vomiting, weight lifting, child birth, and during defecation

trachea AKA wind pipe

normally remains open




*composed of 20 c-shaped rings of hyaline cartilage

c-shaped rings of trachea

*are closed at the front. the open end is at the posterior end ( the back)


*found in the opening is a muscle with transverse fibers called the trachealis muscle



when contracting trachealis muscle

the trachea constricts (change in diameter big change in pressure) time to contract these muscles sneeze and cough



can exspel air at over 100 mph

bronchial tree

consists of branched air ways leading from trachea into the lungs

branches of bronchial tree

1. bronchi- 2 - left & right (have same ribbed cartilaginous as trachea


2. bronchioles- they lack cartilage & a mucus secreting epithelium


3. terminal bronchioles


4. respiratory bronchioles-


5. alveoli-

respiratory bronchioles & alveoli

make up a lobules where gas exchange occurs

blood supply for lungs

get oxygenated blood from bronchial arteries



get deoxygenated blood from bronchial veins

some blood from bronchial veins is diverted to

the pulmonary veins

lungs

occupying separate plural cavities. separated medially by the heart that occupies medisteinum. rest atop the diaphragm

each lung is covered by membrane

The pleural membrane
The pleural membrane

is folded. has inner and outer portion





parietal pleura

outer portion. lines inside rib cage

pulmonary (visceral) pleura

inner portion

pleura cavity

space between

surface tension

water attraction




the tension of the surface film of a liquid caused by the attraction of the particles in the surface layer by the bulk of the liquid, which tends to minimize surface area.

2 things to make pressure go down

1. contract external intercostal muscle -causes ribs to move up & out


2. diaphragm contracts (moves down)



surfactant

lungs secret a chemical _________ to reduce surface tension with in alveoli & prevent their collasps




if a new born doesnt breathe, they are given this

pleurisy

inflammation of plueral membranes resulting in coughing, fever, & very painful respiration

alveoli

walls are composed of simple squamous epithelium, thru these walls ans walls of blood capillaries (gas exchange) need massive surface area-get it bc extensive network of aleoli capillaries



*with every breath you take gases move across surface area size of tennis court

lungs also produce a portion of

amniotic fluid

dust cells

fixed macrophages associated with alveoli

bc of the presents of macrophages of alveoli

tiny blood clots are filtered (destoryed)

inspiration is

an active process- means there has to be control of it




*neural control


*autonomic


*located in brain stem- more specific medulla

2 autonomic reflex control center in the medulla

1. inspiratory area of the medulla- sends impulse that last 2 seconds thru the vegus nerves (cranial nerve pair #10 x) to internal intercostal and the diaphragm causing (the drawing)


2. expiratory area of the medulla- does nothing during normally quite respiration. bc its passive-we dont need it **only becomes active during forced expiration (you can breath out more air than you normally do)

pneumotaxic area

in the pons. its job is to tell inspiratory area to stop @ the end of 2 seconds thereby preventing over inflation

hering breuer reflex

involves stretch sensory receptors in the walls of the bronchi & bronchioles that also send impulses thru cranial nerve pair #10 (vegus) to inspiratory area telling it to stop

if everything goes as it should

hemoglobin/blood will be completely saturated/filled with O2 when it leaves the alveoli

2 things that determine things go as they should

1. perfusion


2. ventilation

perfusion

renewal of blood within the alveoli capillaries

ventilation
renewal of air within the alveoli capillaries

perfusion is the one that

our body prefers to control




controlled by constriction or dilation of arterioles leading into alveoli capillaries

if perfusion is too slow

time is wasted while needy cells need

if perfusion is too fast

does not become completely saturated

hyperventilation

rapid breathing.


-it does not/can not increase O2 saturation of blood


-instead of more O2 in, you are taking more CO2 out

respiratory alkalosis

when hyperventilating blood becomes basic




is a disturbance in acid and base balance due to alveolar hyperventilation. Alveolar hyperventilation leads to a decreased partial pressure of arterial carbon dioxide

the magnitude of the bohr effect is reduced bc

blood becomes more alkaline causing hemoglobin to hang on to O2 longer

widespread vasodilation

blood vessels dilate




decrease in BP- can cause you to pass out

apnea

temporary cessation of breathing, especially during sleep.

cheyne stokes respiration

periods of hyperventilation alternating periods of apnea

the death rattle

common name of cheyne stokes respiration- often heard in persons just before death

the cilia mucus escalator

refers to the mechanism that we utilize to move mucus from lower respiratory structures up the throat where it is then swallowed

lower layer of the cilia mucus escalator

sal- is fluid

upper layer of the cilia mucus escalator

gel- sticky, water insoluble mucus

effects of altitude on respiration

as altitude increases barometric pressure (air pressure) decreases

decompression sickness




AKA the bends

a rapid decrease in barometric pressure. nitrogen gas bubbles out of the blood but it cant be exhaled fast enough & bubbles block capillaries-dizzy, pain, paralysis, death

nitrogen narcosis


AKA rapture of the deep

when down too long, nitrogen builds up in blood & lipid membranes of neurons-reduces their excitability & produces the feeling of euphoria

inspiratory reserve volume

volume of air that can be forcibly inspired above normal (normal breath, then deeper breath)

tidal volume

max volume of air in the lungs during normal quite respiration
*when you reach this point the inspiratory area ceases

expiratory reserve volume

volume of air that can be forcibly expired above normal




the additional amount of air that can be expired from the lungs by determined effort after normal expiration

residual volume
lung volume representing the amount of air left in the lungs after a forced exhalation
vital capacity

total volume that can be moved in & out


=inspiratory reserve volume + tidal volume + expiratory reserve volume

dead space

surface area of respiratory passage ways not involved in gas exchange