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34 Cards in this Set
- Front
- Back
5 STAGES OF SPECFIC IMMUNITY
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1- LYMPHOCYTE DEVELOPMENT
2- PRESENTATION OF ANTIGENS 3- CHALLENGE OF B&T CELLS 4- PRODUCTION OF ANTIBODIES BY B-CELLS 5- T CELLS RESPONSE |
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MARKERS
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GLYCOPROTEIN ON THE CELL SURFACE
MHC-MAJOR HISTOCOMPATIBLITY COMPLEX |
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MAJOR HISTOCOMPATIBILITY COMPLEX(MHC)
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GENE COMPLEX GIVES RISE TO A SERIES OF GLYCOPROTEINS FOUND ON ALL CELLS EXCEPT RED BLOOD CELLS. PLAYS A VITAL ROLE IN REGONITION OF SELF BY IMMUNE SYSTEM AND IN THE REJECTION OF FOREIGN TISSUE
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CLONAL SELECTION
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THE SYNTHESIS OF VARIED RECEPTOR TYPES
CLONE EXPANSION CLONAL DELETION |
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CLONAL DELETION
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CELLS THAT RECONIZE SELF ARE REMOVED
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LYMPH NODES INFLAMMATION
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LYMPYH NODES SWELL BECAUSE OF INCREASE AMOUNTS OF B&T CELLS
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IMMUNOGLOBLOBULIN MOLECOLE=IG
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THE CHEMICAL CLASS OF PROTEINS TO WHICH ANTIBODIES BELONG
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ANTIBODIES SECRETED BY?
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B-CELL RECEPTORS
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4 TYPES OF ANTIGENS
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1-FOREIGN MATERIAL
2-SIZE AND SHAPE 3- ALLOANTIGENS 4- SUPERANTIGENS |
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SUPERANTIGEN
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BACTERIAL TOXINS
T CELL ACTIVATION MUCH GREATER THAN NORMAL ANTIGEN LARGE RELEASE OF CYTOKINES MAY RESULT IN TOXIC SHOCK SYNDROME |
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EXPLAIN ACTIVATION
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CLONAL SELECTION AND BINDING OF ANTIGEN
INST. BY CHEMICAL MEDIATORS TRANSMISSION OF SIGNAL TO NUCLEUS B-CELL CHANGES INTO PLASMA CELLS CLONAL EXPANSION AND MEMORY CELLS FORM ANTIBODY PRODUCTION& SECRETION |
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NAME 4 CLASSES OF IG MOLECULES
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1-IgG
2-IgA 3-IgM 4-IgE |
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IgG
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PRIMARY RESPONSE
MOST PREVALENT IN TISSUE FLUID AND BLOOD |
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IgA
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MUCOUS AND SEROUS MEMBRANE SECRETATIONS
PROTECTION FOR NEWBORNS( HIGH IN BREAST MILK) |
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IgM
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FIRST TO BE SYNTHESIZED DURING PRIMARY IMMUNE RESPONSE
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IgE
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PREVALENT IN ALLERIGES & PARASITE INFECTIONS
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OPSONIZATION
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THE PROCESS OF STIMULATING PHAGOCYTOSIS BY AFFIXING MOLECULES TO THE SURFACE OF FOREIGN CELLS AND PARTICLES
MAKES IT EASIER FOR MACROPHAGES TO ENGULF |
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PRIMARY RESPONSE
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FIRST EXPOSURE, THE TIME IT TAKES FOR HOST TO RECONZIE PARTICLES AS FOREIGN( IgM ELEVATES QUICKLY)
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SECONDARY RESPONSE
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RE-EXPOSURE TO THE SAME ANTIGEN IMMUNE SYSTEM REPONSE QUICKLY
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ANAMNESTIC RESPONSE
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MEMORY CELLS RESPOND QUICKLY BECAUSE ANTIGEN IS RECONZIED FROM PRIOR EXPOSURE
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5 TYPES OF SPECFIC IMMUNITIES
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1- ACTIVE
2- PASSIVE 3-NATURAL 4-ARTIFICAL 5- VACCINES |
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ACTIVE IMMUNITIES
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NAUTRAL OR ARTIFICAL
ANTIGEN ACTIVATES B&T CELLS MEMORY CELLS LONG TERM PROTECTRION |
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PASSIVE
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NATURAL & ARTIFICAL
RECIEVE ANTIBODIES FROM ANOTHER INDIVIDUAL OR ANIMAL NO MEMORY CELLS NO ANTIBODY PRODUCTION SHORT TERM PROTECTION EXAM- BREAST FEEDING |
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NATURAL
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IMMUNITY PRODUCED BY NORMAL BIOLOGICAL EXPERIENCES, NO MEDICAL INTERVENTION
EXAM.- INFECTION MOTHER TO CHILD |
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ARTIFICAL
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IMMUNE PROTECTION THROUGH MEDICAL PROCEDURES OR INTERVENTIONS
EXAM- VACCINATIONS+ GIVEN IMMUNOTHERAPY( RABIES) |
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VACCINES- TYPES
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KILLED WHOLE CELL OR INACTIVATED VIRUS
LIVE, ATTENUATED CELLS OR VIRUS ANTIGEN MOLECULES FROM BACTERIA OR VIRUS GENTECTICALLY MANUFACTED MICROBES OR MICROBIAL ANTIGENS |
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BENEFITS OF VACCINATIONS
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LONG-LASTING IMMUNITY
HERD IMMUNITY- INDIRECT PROTECTION OF NOIMMUNE PREVENTS EPIDEMICS |
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NAME 3 WAYS FOR ACTIVATION
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1- CELL-MEDIATED IMMUNITY
2- ANTIGEN PRESENTING CELL 3- TRANSFORMATION |
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CELL- MEDIATED IMMUNITY
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DIRECT INVOLEMENT OF T CELLS
PRODUCE & REACT TO CYTOKINS ACTIVATE WITH B-CELL ACTIVATION |
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ANTIGEN PRESENTING CELLS
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MACROPHAGES AND DENDRITIC CELLS
ASSOCIATED WITH MHC( MAJOR HISTOCOMPATABILITY COMPLEX) |
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TRANSFORMATION
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ACTIVATED T CELLS PREPARE FOR MITOSIS
MEMORY CELLS ARE PRODUCED |
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TWO TYPES OF T CELLS
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1- HELPER T-CELLS(Th)
2- CYTOXIC T-CELLS(Tc) |
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Th- HELPER TCELLS
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REGULATE IMMUNE REACTIONS TO ANTIGENS BY RELEASEING CYTOKINES
CYTOKINES ACTIVATE MAGROPHAGES MOST PREVALENT IN THE BLOOD |
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Tc- CYTOXIC T-CELLS
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BINDS AND LYSES CELL(APOPTOSIS)
PERFORINS-PUNCH HOLES IN CELLS DEGRADE PROTEINS(GRANZYMES) NATURAL KILLERS(NK)CELLS- ATTACK INFECTED CELLS AND CANCER CELLS |