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100 Cards in this Set

  • Front
  • Back
Digitalis
1. Tx for
2. adverse effects
3. toxicity ->
4. what should you do for pt. chronically taking dig?
5. how is it cleared from body
6. causes of toxicity
1. afib and HF
2. GI stuff (N&V, diarrhea), vision changes, and arrythmias
3. >ed ectopy and >ed vagal tone -> A. tachy + AV block
4. routine monitoring of dig levels
5. renally
6. viral illness, excessive diuretic use (loops -> hypokal -> dig tox)
Diastolic dysfnxn:
1. what's wrong?
2. what is the cause
3. Tx
4. other manifestations
4. aka
5. chronically elevated LV pressure can cause what?
1. impaired filling from poor myocardial relaxation or dimished ventricular compliance
2. HTN
3. diuretics, and antiHTNs
4. chronically elevated LV diastolic pressures -> LA dilation -> Afib
4. CHF w/ preserved or >ed Left EF (ie =/> 60%)
5. LA dilation -> Afib
B-type natriuretic peptide
1. what does it Dx
2. what is the value that dxes this?
1. CHF
2. > 100 pg/mL
34 yo angina present w/ active sports and AS
1. what is most likely cause of AS?
2. how does the AS cause the angina?
1. bc of age, congen. bicuspid aortic valve
2. severe AS -> large LV mass = requires additional O2 -> angina
Prinzemetal's angina:
1. aka
2. cause
3. greatest risk factor
4. classic finding
5. ECG findings
6. Tx
7. meds that you shouldn't give
8. other vasospastic disorders assoc.d w/ varient angina
9. most accurate test
1. variant angina
2. coronary vasospasm
3. smoking
4.pain @ night, wakes pt.
5. transient ST elevations
6. stop smoking + CCBs + nitrates (promote vasodil and prevent vasocnstrxn)
7. nonselective Beta blockers, and ASA bc they can -> vasocnstrxn
8. raynaud's phenom, and migraine headaches
9. ergonovine test
*HY*
EKG shows ST elevation in II, III, and aVF:
1. what does this mean?
1. inferior wall MI <- RCA occlusion
what would giving lidocaine in ACS (angina or MI) do?
> risk of asystole
1.MCC of SBE? (damaged heart)
2. Acute endocarditis (normal heart valves)
3. GU procedures
4. upper Resp procedures
5. coag neg
1. Viridans group streptococ, enterococci, staph epi
2. S. Aureus
3. enterococci
4. viridans strep.
5. staph epi
Afib
1. EKG
2. MCC. other causes
3.Tx
1. irreg irreg R-R w/ absent P waves and narrow QRS complexes
2. HTN. valv. heart dz, MI, HF, hyperthyr, and ROH (PIRATES)
3. Tx:
- unstable = (low BP, altered MS). Immediate cardioversion
- Stable:
< 48 hours: cardioversion
> 48 hours: 3-4 wk rate control and anticoag b4 cardioversion.
(Rate control beta blockers or CCB)
SVT
1. EKG presentation
2. hemodynamically stable Tx
3. "" unstable TX
1.tachycard + narrow QRS
2. vagal maneuvers followed by adenosine and AV nodal blockers
3. (dangerously low BP) emergent cardioversion
clopidogrel uses:
1. 2ndary prevention following UA/NSTEMI
2. following PCI - prevents thrombosis
meds w/ mortality benefits post MI
(SSAAB)
1. ASA
2. beta blockers
3. ACE inhibs
4. statins
5.K sparing diuretics (spiro)
heat stroke
= clinical appearance
systemic effects:
= >105 F. dehyd, hypOTN, tachycard, tachypnea.

can have systemic effects: seizures, ARDS, DIC, hepatic/renal failure
V tach Tx
- w/ stable BP: amiodarone or lidocaine
- w/ unstable BP: cardioversion
what is the most important factor for the survival of pt. who collapses in street and has no pulse?
Leading cause of wtinessed out of hosp. cardiac arrests is?
Time to defibrillation.
Leading cause of wtinessed out of hosp. cardiac arrests is Vtach, Vfib.
how nitrates work in heart
-> venodilation (ie dilation of CAPACITANCE vessels) -> blood to pool in systemic venous circ. -> <es preload -> <es ventric vol and stretch -> <es myocard O2 demand
1. risk factors for aortic disxn
2. if HTN present what's the 1st thing you do?
3. when HTN is taken care of or not present?
1. bicuspid aortic valve, pregnancy, coarctation of aorta, and marfan's syndrome
2. AntiHTN meds b4 dxic studies
3. TEE is initial investigation of choice
PEA:
1. what is it?
2. tx
3. important to know
4. potentially treatable causes
1. discernible rhythm (that isn't vfib or vtach) on cardiac monitor w/ NO palpable pulse
2. CPR and chest compressions, ABCs, and 100% O2, IV access for epi, atropine, and vasopressin
3. NOT A SHOCKABLE RHYTHM!
4. 6 Hs and 5Ts:
6Hs: hypo/hyperkal, hypoxia, H2 (acidosis), hypoglycemia, hypovolemia, hypothermia
5Ts: tablets (drugs), toxins, tamponade (cardiac), tension pneumothorax, thrombosis (MI, PE)
atrial flutter
1. pattern
2. cause of pattern
3. classic symptoms
4. Tx
1. saw-tooth pattern at rate of 300/min
2. RE-ENTRANT RYTHM FROM VARIABLE AV NODE CONDUCTION
3. palpitations, chest pain, sob, lightheaded
4. rate control w/ CCB, beta block; if meds don't work then cardiovert, if still doesn't work then ablate ectopic focus
young, healthy person who develops CHF.
1. what is probable cause?
1. myocarditis from viral infxn, coxsackie B is MCC
cardiac tamponade:
1. becks' triad
2. pathophys
1. hypOTN, JVD, muffled heart sounds.
2. pericardial space fills w/ fluid -> ventricles can't expand as much -> decreasing amount of preload
aortic stenosis
1. typical symptoms
2. phys exam
1. exertional dyspnea, syncope, angina
2. systolic ejxn murmur radiating to apex and carotid arteries
PVCs:
1. morphology
2. Tx
1. wide QRS (>120msec)
2. asymptomatic pt. : observation
symptomatic pt. : beta blockers
-what is a bad prognostic factor in pt.s w/ HF?
-what can help control this?
- hyponatremia indicates severe HF . also hypo/hyperkal can reflect activity of RAAS.
- <ing H2O intake (not >ing Na intake)
Tx of aortic dsxn:
1) Type A: ascending aorta : Tx = medical (labetolol) + Qx
2) Type B: descending : Tx = labetolol
MS
1. MS -> changes in heart?
2. MS is commonly caused by
3. classic auscultative findings
4. ECG findings
1. MS -> LA dilation -> afib -> thrombus embolizes -> stroke
MS -> >ed LA Press -> >ed pulmonary vascular Press -> hemoptysis
2. rheumatic dz
3. loud S1 and mid diastolic rumbling murmur at apex
4. irreg irreg rhythm and loss of P waves w/ afib
*HY*
<ed CO + >ed PCWP : indicative of LV failure = ?
indicative of LV failure = cardiogenic shock
*HY*
SEs of thiazide diuretics
hyperGLUC, hypoNaKal
MR:
1. type of murmur
2. most common symptoms
3. indication of more severe MR
4. causes
1. holosystolic murmur heard best @apex with radiation to axilla
2. fatigue and exertional dyspnea
3. dry cough <- pulmonary congestion and edema <- LV dysfnxn
4. rheumatic dz, infective endocarditis, trauma, ischemic heart dz, or HCOM
HCOM
1. murmur
2. cause of syncope
3. what is seen on echo?
1. systolic cresc-decresc on L. sternal border w/o radiation (helps differentiate from AS)
2. LV Hypertrophy
3. asymmetric septal hypertrophy
dilated cardiomyopathy
1. cause
2. how do you Dx
3. what will you see on #2
4.Tx
1. MC coxsackie B (others: parvo B19, HHV 6, adenovirus, enterovirus)
2. echocardio
3. dilated ventricles and diffuse hypokinesia -> systolic dysfnxn = low EF
4. supportive, management of CHF symptoms
tx of cocaine-related cardiac ischemia
benzos + ASA + nitrates (BAN)
cardiac tamponade
1. signs
2. ECG
3. Tx
1. hypoTN, tachycard, JVD
2. electrical alternans
3. massive vol resuscitation and emergent pericardiocentesis
pt. on bed rest develops a cold leg w/ no distal pulses and mottled in appearance.
what do you do?
Arterial occlusion:
-LMWH
-ECHO to dx thrombus
HTNsive pt. b/l upper abdom. masses are palpated.
-what is this?
- what is this condition assoc.d w/?
-Dx?
PKD.
also assoc.d w/ hematuria, 2ndary erythrocytosis, and RF.
Early Dx w/ abdo US
acute mngmt of STEMI
BMOAN
-reperfusion (thrombolysis or PCA)
- antiplatelet therapy
-morphine
-nitrates (contraindicated in certain circumstances see below*)
-beta blockers
*AS, recent PDE use, or RV infrxn (30% of inf wall MIs)
Presentation and Tx:
1. LV STEMI
2. RV STEMI
1. hypoTN and PE
BMOAN
2. hypoTN, JVD, clear lungs
IV fluid resuscitation to ? RV stroke vol. Nitrates are CONTRAINDICATED
acute MI w/ muffled S1 and S2 and presence of S3. basilar crackles that extend 1/2 way up lung fields b/l:
1. what is it?
2. Next step?
3. what do you do after #2
4. what do you do after #3
1. flash Pulm edema 2ndary to MI
2. furosemide = DOC
3. anticoag w/ hep
4. emergent PTCA or thrombolysis
1. in what 3 scenarios do you do CABG?
2.common complication post CABG
1. 3 vessels w/ @ least 70% occlusion, Left Main coronary artery occlusion, or 2 vessel dz in pt w/ DM
2.AF.
Jervell-Lange-Nielson syndrome
1. what is this
2. tell me about it
3. possible complication
4.Tx.
-congenital QT syndrome.
-Auto Recess + congenital deaf + QT prolong
-QT prolong -> torsades de pointes -> syncopal episode and sudden death
4. propanolol
most effective nonpharm intervention to < BP?
>ed fruit and vegies = DASH diet
chest pain at night , chronic cough and hoarseness.
GERD. Tx wih H2 antagonist or PPI
when do you see JVD
(4 Cs) CHF, constrictive pericarditis, cardiac tamonade, cor pulmonale
HTN is defined as
Normal pt: >140/ 90
DM or renal dz: >130/80
severe HTN: 160/100 = begin tx w/ 2 meds
kussmaul's sign
=
when do you see it
Most accurate test
rise of JVP on inhalation (from >ed venous return to heart on inhalation)
seen w/ constrictive pericarditis
CT or MRI -- look for thickened calcified pericardium
pulsus paradoxus
drop in syst. bp of >10 mmHG w/ inspiration
see it w/ cardiac tamponade, asthma, and emphysema
best initial therapy is bolus of fluid
when do you do an angio?
- cardiac stress test is abnormal
- prior to coronary surgery or angioplasty
-in ACS like unstable angina
(stenosis must be >70% to be significant)
stable angina
1. ECG
2. stable angina + HTN Tx
- why use this as Tx?
1. horizontal ST seg depression in stress test
2. beta blocker - will > threshold for anginal episod, control HTN, and is cardioprotective
paroxysmal nocturnal dyspnea + long standing HTN + Pulmonary edema (cardiogenic)
1. what kind of HF?
2. what kind of dysfnxn
3. what drug can relieve dyspnea?
4. how does #3 work?
5. what are other mainstays of therapy for decompensated heart failure ?
1. LVHF
2. diastolic
3. nitroglycerin (NTG)
4, rapidly reduces preload
5. loop diuretics (work by <ing total body volume)
Neurocardiogenic syncope (ie vasovagal syncope):
1. common symptoms
2. usually preceded by
3. dx
1. syncope precede by nausea, diaphoresis, tachycard, and pallor.
2. response to stress (medical needles), pain, or urination
3. tilt table
dihydropiridine
1. what are they and examples
2. side effects
1. CCB. any drug ending in -dipine. ex. nifedipine and amlodipine
2. peripheral edema and don't give if have kidney problems
drug that can cause of peripheral edema:
Dihydropyridines (CCB)
massive PE
1. PE complicated by ___ &/or _____
2. symptoms
3. how do you know you have Part 2 of #1
1. PE complicated by hypOTN &/or acute Right heart strain.
2. regular PE: dyspnea + pleuritic CP. Massive PE: Syncope
3. indic.s of right heart strain: JVD and RBBB
what causes LV dysfunction?
what improves it?
-tachycardia, neurohumoral activation, absence of atrial "kick" and atrial-ventricular desynchronization
-controlling rate and rhythm
Infective endocarditis (IE)
1. major criteria
2. minor criteria
3. how many majors and minors are necessary for DX
4. non-cardiac complications and what causes them?
5. what do you do if kid comes in and probably already experienced prior episode of rheumatic fever?
1. new murmur; >/= 2 positive blood Cx for org. (S. viridans, S. aureus); endocard involved on echo.
2. predisposing condition (prosthetic valve, IVDrugs); fever, vascular phenom (septic emboli); immuno phenom (glomerulonephritis); or 1 + blood Cx
3. 2 major; 1 major & 3 minor; or 5 minor
4. immune-complex deposition w/in tissues -> JONES (joint, heart,nodules, erythema, syndenham's )
5. give Abx prophylaxis of penicillin (if kid do it until pt. is 18 yo)
Pt. comes in w/ heart attack. Had PMHx of eczema and occasional dyspnea w/ coughing. hospitalized and on day 2 of treatment complains of SOB. PE shows b/l wheezing and prolonged expiration. what caused current resp. symptoms
Drug SE of metoprolol. PMHx of eczema and dyspnea shows possible asthma.
beta blockers can exacerbate Asthma and COPD
1. what is Dxic for malignant HTN
2. what is responsible for pathologic change in end-organ damage in malig HTN
1. BP >/= 200/140 mmHg + presence of papilledema
2. fibrinoid necrosis
best drug Tx for endocard w/ Strep viridans?
if it is sensitive for pen:
IV pen G or IV ceftriaxone (NEVER TX ENDOCARD W/ ORAL ABX!)
Acute Mitral regurg:
1. cause
2. Tx of #1
3. common sequela of MR
4. wht would PCWP show?
1. papillary muscle dysfnxn in acute MI
2. emergent Qx
3. PE (orthopnea and bibasilar crackles)
4. >ed P in LA
Causes of peripheral edema assoc.d w/
>ed cap hydrostatic P
1. HF (LV or cor pulmonale), primary renal Na retention (renal dz, drugs, pregs), venous obstrxn (cirrhosis, acute PE and venous insuff.)
Cause of peripheral edema from hypoalbuminemia (<ed oncotic P)
protein loss (nephrotic synd and GI tract losses)
<ed albumin synth (cirrhosis and manlnutrition)
causes of peripheral edema assoc.d w/ >ed capillary permeability
- burns, trauma, and sepsis
- allergic rxns
- ARDS
- Malignant ascites
Causes of peripheral edema assoc.d w/ lymphatic obstrxn/ <ed oncotic P
malignant ascites, hypothyroidism
How to tell btwn MI and gerd from presenting symptoms
MI will have diaphoresis and SOB, GERD won't
also S4 is a classic finding of MI
HTN + ^Ca (stones, bones, psych overtones)
hyperparathyroidism is an uncommon cause of 2ndary HTN
How does COPD -> pitting edema?
COPD (chronic hypoxemia) -> cnstrx of pulmonary arteries -> pulm HTN, RVH, and RVF (cor pulmonale)
Right heart failure:
1. ECG
2. how to confirm?
3. signs that point to RV failure
4. how does RV fail affect preload?
5. Tx
6. drugs to avoid
1. ST elevations in inf. leads = inf infarct = RV. and ST depression in leads I and AVL (left most leads) = STEMI affecting R side of heart
2. do a right sided ECG
3. JVD + Kussmaul's sign (>ed JVD w/ insp) + clear lung fields
4. <ed preload -> <ed CO and HYPOTN
5. IV fluids to maintain preload and BP
6. nitrates and diuretics
pt w/ hyperthyroid-related afib, what's DOC?
beta blocker
3 mechanical complications of MI. when they happen. what you see with each
1. Papillary muscle rupture : 5 days. -> MR (will hear holosytolic murmur loudest @ apex w/ rad to axilla)
2. LV free wall rupture : day 3-7. Majority are w/ Ant wall MI, Tamponade -> PEA is common of LV free wall rupture
3. interventricular septum rupture : 5 days. -> VSD (new onset holosystolic murmur. no PEA)
AAA:
-rad of choice for Dx
- " " "" follow up
Abdo US for both
MVA pt. driving car w/o seatbelt in (ie blunt chest trauma) w/ JVD, tachycard, and hypoTN despite fluid resusc.
1. DX
2. CXR findings
1. cardia tamponade
2. normal cardiac silhouette w/o tension pneumothorax
End stage renal dz Hemodialysis :
1. absolute indications
2. contraindications
1. (1) fluid overload not responsive to medical Tx (2) hyperkal not responsive to medical mngt (3) uremic pericarditis (4) refractory metab acidosis
2. severe irreversible dementia
1. MCC of 2ndary HTN?
murmur assoc.d w/ it?
1. Renal artery stenosis,
2. periumbilical
1. asymmetrically elevated BP in left arm suggestive of
2. Asymmetrically elevated BP in Right arm or arms>legs
1.subclavian Artery stenosis
2. coarctation of aorta
Dxing PAD in high risk or syptomatic pt.s
ABI using Doppler
kussmaul's (K) vs pulsus paradoxus (PP)
1. event
2. mechanism
3. Dz assoc.d w/
1. K = JVD w/ inspiration; PP = >ed SBP by > than 10 mmHg on inspiration
2. K = <ed capacitance of RV; PP = <ed capacitance of LV
3. K : constrictive pericard >> cardiac tamp
PP : cardiac tamp >> const pericard
Outpatient treatment for CHF
SAAB DL
spironolactone, ACEi, ASA, Beta block, Digox (maybe), loop (furos)
Acute Tx of exacerbations of CHF
LMNOPP
Loop, morphine, nitrate, O2, Pressors (dobutamine) and positioning of legs (legs down and pt sitting up)
1. LBBB causes
2. EKG dx of LBBB
3. RBBB causes
4. EKG of RBBB
1. Acute MI, dilated cardi, AS
2. WiLLiam (of william marrow)
V1 W and V6 M (or rabbit ears)
3. relatively benign when compared to LBBB
4. MaRRoW
V1 M and V6 W
PCI (angioplasty) vs thrombolytics
door to balloon time = 90 minutes
if > than 90 then use thrombolytics unless contraindication to them
absolute contraindications to thrombolytics
-Nonhemorrhagic stroke w/in the last 6 mo.s
- severe HTN >180/110
- Recent Qx w/in last 2 wks
- Major bleeding ex. CNS or GI
complications of acute MI:
1- assoc.d with bradyarrhythmia
2- assoc.d w/ tachy
1- sinus brady or 3rd heart block (cannon a waves)
2. (1) RV infarction (- new inferior wall MI: I, II, AVF and clear lungs; - flip the leads, - Tx: fluid replacement
(2) Tamponade/ free wall rupture
(3) VTach/ Vfib
(4) valve or septal rupture - new onset murmur and pulmonary congestion
systolic dysfnx (CHF)
dilated heart
<ed EF (< 60%)
massive pulmonary embolism:
-what will you see?
- what are changes in heart chamber pressures?
- complication
PE complicated by hypOTN &/or acute right heart strain.
- Syncope+JVD+RBBB
- RA Pressure > 10 (norm = 5), Pulmonary Artery pressure > 40 (norm = 25)
- shock (hypoTN & tachycardia)
What is most likely cause of 2ndary HTN?: Pt presents w/ agitation, headaches , gained 14 lbs in 3 mo.s, K level of 3.2, glucose of 205.
Adrenal cortical dz being Cushings in this scenario.
Dude w/ anterior wall MI one month ago comes in complaining of exertional dyspnea and fatigue. Exam shows pansystolic murmur. EKG shows Q waves and persistent ST segment elevation in anterior leads. Whats the cause?
What symptoms come with this?
How do you confirm Dx?
Ventricular aneurysm:
- symptoms of Vent an: CHF, vent. arrhythmias, mitral regurg and/or thrombus formation. Also persistent ST elevations are also often seen.
confirm Dx w/ echo: shows dyskinetic wall motion
what drugs do you want to avoid in STEMI?
dihydropyridine (CCB: nifedipine and amlodipine).
Bc. -> peripheral vasodil and reflex tachycard -> cardiac ischemia
32 yo woman, S3, enlarged cardiac silhouette (cardiomeg), b/l pleural effusions (pulm edema), enlarged liver. recent URI.
1. what is the Dx?
2. most likely cause of her symptoms
3. additional cause of Dx
4. complication
1. myocarditis
2. viral infxn (coxsackie)
3. Drug tox (doxorubicin)
4. CHF
how can you clinically determine if edema is cardiac in cause?
Positive Hepato-jugular reflex indicates that venous pressure is elevated and suggests that heart disease-related edema is present. If reflex is negative if the liver is the cause.
High-output (= high CO) cardiac failure:
1. causes
1. Arterio-venous fistula (AVF -> AV shunting -> >ed preload and CO and <ed SVR), thyrotoxicosis, Paget dz, anemia, and thiamine deficiency
situations that predispose to torsades do pointes
1. malnourished (ex ROHic)
2. Drugs: moxiflox, fluconazole, TCA, amiodarone
3. familial long QT: jervell -Lange-Nelisen = deaf; Romano ward = not deaf
Tx mag sulfate
(+ beta blocker for familial long QTs)
In tetralogy of fallot why does knee to chest position help?
When pt. is having a "tet spell" this position increases systemic vascular resistance -> >ed blood flow from the right ventricle to the pulmonary circulation. (morphine and IV fluid bolus can also be given to > pulmonary blood flow)
Endocarditis Tx:
1. 1st
2. Viridans or HACEK
3. Staph A (sensitive)
4. Fungal
5. S. Epi or S. Aureus (resistant)
6. Enterococci
1. 1st draw blood then empiric Tx of : Vanc + Gent
2. Ceftriaxone
3. Oxacillin, Nafcillin, or Cefazolin
4. Ampho + valve replacement
5. Vanco + Gent
6. Amp + Gent
Pericardial effusions:
1. Causes
2. appearance on CXR
3. when does this become tamponade?
1. MCC = idiopathic, viral, CA, post MI, uremia, autoimmune, hypothyroid
2. "water bottle" shape
3. when pericardial space can no longer stretch to accommodate the effusion
Post MI complication of interventricular wall rupture vs. ventricular free wall rupture:
1. when do they happen
2. what differentiates them
1. both approx. 5 days post MI
2. interventricular wall -> VSD (new onset holosys murmur)
ventri free wall rupture -> pericard tamponade (development of Pulseless Electrical Activity)
Post MI complication of recurrent ischemia
1. what will you see bc. of it
1. ventricular arrythmia (vtach or vfib; not PEA)
Post MI complic of papillary muscle rupture
1. when does it happen
2. what does it cause?
1. approx 5 days post MI
2. Mitral regurg (new onset holosys) -> hypOTN
Post MI ventricular aneurysm:
1. when
2. what does it present w/
1. days to months
2. akinesis of involved LV wall, vent arrhythmias, and systemic embolization
peripheral edema causes:
1. non-pitting
2. pitting
1. lymphatic obstrxn (lymphedema), or >ed interstitial accumulation of albumin and other proteins (ex myxedema assoc.d w/ hypOthyroid)
2. caused by >ed movement of fluid from vascular to interstitial space:
- >ed hydrostatic pressure (CHF, portal hyperTN)
- <ed plasma oncotic P (low albumin from malnutrition, nephrotic syndrom, cirrhosis)
- >ed capillary leak (burns, trauma, or infxn)
what can reverse heart failure in alcoholic cardiomyopathy
abstinence from ROH