Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
23 Cards in this Set
- Front
- Back
Digestion
|
-the gastrointestinal process of breaking down food and absorbing its constituents into the body
|
|
How is energy delivered?
|
1)lipids
2)amino acids 3)glucose |
|
How is energy stored?
|
1)fat
2)protein 3)glycogen |
|
Phases of Energy Metabolism
|
1)cephalic phase
2)absorptive phase 3)fasting phase |
|
Cephalic Phase
|
“preparatory phase”
|
|
absorptive phase
|
“energy is absorbed into the bloodstream”; meets immediate energy needs
|
|
fasting phase
|
all the unstored energy as been used
|
|
Pancreatic Hormones
|
1)insulin
2)glucagon |
|
Insulin
|
promotes use of glucose as primary source of energy
-promotes conversion of blood-borne fuels to storable forms -promotes storage of glycogen in liver and muscle, fat in adipose tissue, protein in muscle -in cephalic phase, insulin lowers level of blood-borne fuels, and in absorptive phase it minimizes increasing levels of fuels by utilizing and storing them |
|
Gluconeogenesis
|
conversion of protein to glucose
|
|
“glucostatic theory”
|
glucose levels determine what we eat
|
|
“lipostatic theory”
|
fat stores determine the amount of consumption over long term
|
|
Problems with the Set Point theory
|
epidemic of obesity and overweight people should not occur
-inconsistent with basic eating-related evolutionary pressure -fail to recognize major influences such as sight, smell and taste |
|
Positive-Incentive Perspective
|
we are drawn by the pleasure of eating, evolved to crave food
-eating is influenced by many factors: 1-learned preference for flavors 2-past experiences 3-time since the last meal 4-social influences |
|
Factors that determine what/when/how much we eat
|
-Adaptive species-typical preferences
-sweet and fatty tastes: high energy, vitamins and minerals -salty: sodium-rich |
|
“appetizer effect”
|
small amounts of food may increase hunger
|
|
“satiety”
|
feeling full; food in gut and glucose in blood can induce satiety signals
|
|
Known as "feeding center"
|
- LH lesioned animals starve to death, LH became known as the “feeding center”
|
|
known as the "satiety center"
|
-VMH lesioned animals became fat, grew to be 4x their bod weight, VMH became known as the “satiety center”
|
|
lipogenesis
|
production of fat
|
|
lipolysis
|
breakdown of fat
|
|
Role of G.I Tract in Satiety
|
studies suggest that stomach contractions lead to hunger (Cannon and Washburn) but someone who had their stomach removed still reported hunger pangs
-Koopmans used second stomach preparation; rats eat less when 2nd stomach was loaded with food even though no innervation or nutrients absorbed |
|
Hunger and Satiety Peptides
|
gut peptides that decrease meal size: cholecystokinin (CCK), bombesin, glucagon, alpha-melanocyte-stimulating hormone, somatostatin
- peptides are usually synthesized in the hypothalamus: neuropeptide Y, galanin, orexin-A and ghrelin -hypothalamus plays role in eating behaviours |