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14 Cards in this Set
- Front
- Back
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Disposable Soma Theory
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major aim of the tissue system gives production to the reproduction cells--cont of species...once you reproduce don't have any purpose after that---reproduction cells cam divide infinatly...somatic cells have finite division-most complete division at birth-prone to appotosis-cellular suicide
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Programmed (Neuroendocrine) Theory
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this was the first theory proposed...factor that programs the aging system-chronometer (time keeper)-- this was not accepted...there are compounds and horomones-growth horomone (somatotropin)-1. high protein synthesis sarcopenia-loss of muscle mass 2. fate and carbs- allow cells to utilize it
national institute oh health 150 vol. elderly 6 mths injected w/ gh after 6 mths had to terminate the experiemnt- majority of individuals showed significant improvement |
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lentigo (lentigero)
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senial freckles- melanocyte
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achrochordon
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binine tumor- (cutaneous tags)- promarily made up of excessive dermal connective tissue- due to hormonal imbalance w/ age
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senite pruritis
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cracking of the skin- due to loss of sebacous glands (less oily) can be lethal because it allows bacteria to enter
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senile keratos (Actinic keratosis)
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localized red areas- this can be pre-cancerous- often on male scalps
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Herpes Zoster (shingles)
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vericella virus- smae virus that causes chicken pox-it affects free nerve endings-virus stays on the tip og the nerve endings causing severe pain
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Decubitus ulcer
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common in individuals who are wheelchair bound due to lack of motility
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Skin cancer
a. Basal cell carinoma |
affects the basal cells- rarely malignate- cure rate is 100% if treated- can rarely penetrate to the dermal layer
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Skin cancer
b. squamous cell carcinoma |
they form extensions into the dermal layer- can travel to other sites through vascular system
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Skin cancer
c. Melanoma |
maligante because they can readily metastasize
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Skin cancer
d. secondary skin cancer |
cancer develops somewhere else and then can spread to the skin
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Gene mutation theory
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as an individual ages the frequency of genetic mutations will increase- mutations will become lethal---increase is not enough to cause death
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Cross-linking theory
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not sure if it responsible for aging mechanism- proteins begin to cross-link themselves to other structures- there shape changes - configuration of proetin determines it's function....collagen undergoes crosslinking w/ aging (30%) -tendons and ligaments....sclerosed- hardening of the arteries- crosslinking of elastin...type II diabetis mellitus- cross liking of colagen- don't know if it the cause or the result
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