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34 Cards in this Set

  • Front
  • Back
Normal PH level
7.35 and 7.45
In ACIDOSIS, the___goes__ and the ___rate and ____ go___ in attempt to blow off acids. The carbonic acid created by the neutralizing action of HCO3 can be carried to the ____ where it is reduced to ____ and ___ and exhaled. Thus H+ ions are inactivated.
PH, down
respiratory rate, depth, up
lungs
carbon dioxide, water
In ALKALOSIS, the___goes ___and the ___and ___go down. Co2 is retained, and the carbonic acid concentration increases to neutralize and decrease the strength of excess HCO3.
PH, up
respiratory, depth, down
Carbonic Acid- HCO3 system ratio
20:1
Maintains a PH of 7.4 with a ratio of 20 parts HCO3 to 1 part carbonic acid
The carbonic acid concentration is controlled by excretion of __ by the __. The___and__of___change in response to CO2 levels.
Co2(carbon dioxide)
lungs
rate, depth, respirations
The HCO3 concentration is controlled by the___. Which selectively ___ or___HCO3 in response to the body needs.
kidneys
retain, secrete
Potassium acidosis
The body protects itself from the acid state by moving H+ ions into the cell, K moves out to make room for H+ ions. K level increases
Potassium alkalosis
The cells release H+ ions into the blood in attempt to increase the acidity of the blood. K moves into the cells, K level decreases.
Respiratory Acidosis
The total concentration of buffer base is lower than normal.
Increase in H+ concentration more H+ ions are circulating in the blood.
Any condition that causes an obstruction of the airway or depresses respiratory status can cause____.
Respiratory acidosis
respiratory acidosis causes
(H BBBAAC PERM)
Hypoventilation

Brain trauma
Bronchitis
Bronchiectasis
Asthma
Atelectasis
Central nervous system depression

Pneumonia
Pulmonary edema
Emphysema
Respiratory failure or depression
Medications
respiratory acidosis s/s
(CDDRR VHHM)
Cyanosis as hypoxia becomes more acute
Diaphoresis
Dysrhythmias leading to ven. fib.
Rapid, irregular pulse
Restlessness

Visual disturbances
Headache
Hyperkalemia
Mental status changes(drowsiness,and confusion)
respiratory acidosis interventions
(AATE PPMMS)
Adm. O2 (AP)
Avoid tranquilizers, opioids,sedative, hypnotics (further depress resp.)
Turn client to cough and deep breathe
Encourage hydration to this secretions unless excess fluid is contra

Place pt. in Semi Fowlers unless contraindicated
Prepare to adm. chest physiotherapy and postural drainage(AP)
Maintain patent airway
Monitor signs of resp. distress
Suction client prn
Conditions that causes overstimulation of the respiratory system.
respiratory alkalosis
respiratory alkalosis causes
(OPFAHHH)
Overventilation by mechanical ventilators
Pain
Fever
Anxiety
Hypoxia
Hyperventilation
Hysteria
respiratory alkalosis s/s
(PM LIHHT)
Paresthesias, such as tingling of the fingers and toes
Mental status changes

Lightheadedness, vertigo
Initially hyperventilation & resp. stimulation cause abnormal rapid and deep resp. in attempt to compensate, the resp rate and depth than decrease
Headache
Hypokalemia, hypocalcemia
The PH is <than>< than 35mm Hg
respiratory alkalosis interventions
(PPMM)
Provide cautious care with ventilator clients so that they are not forced to take deep breaths too deeply or rapidly
Prepare to assist with adm. calcium gluconate for tetany (AP)
Maintain patent airway
Monitor electrolyte values esp K+ and calcium.
The total concentration of buffer base is lower than normal. with relative increase in the H+ ion concentration, this occurs as a result of losing too much acid without sufficient base.
metabolic acidosis
metabolic acidosis causes
(IM GED LR HSS)
Insufficient metabolism or carbs
Malnutrition

Gastrointestinal fistulas
Excessive ingestion of aspirin
Diabetes mellitus or diabetic ketoacidosis

Lactic acidosis
Renal insufficiency or renal failure

High fat diet
Severe diarrhea
Starvation
metabolic acidosis s/s
(FINCHH)
Fruity smelling breath as a result of improper fat metabolism
In an attempt to blow off the extra CO2 and compensate for the acidosis, hyperpnea with Kussmaul resp(deep and fast) occurs.
Nausea and vomiting
CNS depression, mental dullness, drowsiness, stupor, coma
Hypotension
Hyperkalemia
metabolic acidosis interventions
Seizure precautions
Assess LOC for CNS depression
Monitor patent airway
Monitor I&O
Monitor serum K+ levels
A deficit loss of H+ or acids or an excess of base HCO3, results from the loss of acid without a comparable loss of bass in the body fluids.
metabolic alkalosis
metabolic alkalosis causes
(HINTDD)
Hypokalemia, hypocalcemia
In an attempt to compensate, RR and depth decrease to conserve CO2
Numbness and tingling in the extremities
Twitching in the extremities
Dizziness
Dysrythmia, tachycardia
metabolic acidosis interventions
(IMMPP)
Institute seizure precautions
Maintain patent airway
Monitor K and calcium levels
Prepare to replace K chloride(AP)
Prepare to adm. meds(AP) to promote the excretion of HCO3
ABG
(aterial blood gas) definition
Levels reflect the effectiveness of the lungs to exchange O2 & CO2

Kidneys to balancing the retention and elimination of HCO3

The effectiveness of the heart as a pump
ABG specimen- How to obtain
(Vicky asked is Peter eyes always really low)
Vicky- VS
Asked- Allen's test
Is- Identify factors that may affect accuracy
Peter- Prepare a heparinized syringe
Eyes- Emotional support to client
Always- Apply pressure stat to the puncture site for 5-10 min if pt is taking anticouagulants
Really- Record pt temp, type of supplemental O2
Low- Label the specimen, transport it to lab
What is Allen's test?
Apply pressure on pt. ulnar and radial arm same time,
While pressure is applied ask pt to open and close hand repeatedly. Hand should blanch
Release pressure from the ulnar artery, assess color of the extrem. distal to the press. point.
If pinkness fails to return within 6 secs, the ulnar artery is insufficient. The radial artery should not be used for obtaining a bld. specimen.
respiratory imbalance
PH is ____ of the PCO2.
opposite.

PH up PCO2 down
PH down PCO2 up
metabolic imbalance
PH is ___ as the HCO3.
same

PH up HCO3 up
PH down HCO3 down
acid base imbalances
fun fact
respiratory (opposite of pH)
metabolic (same)
PH level
7.35 to 7.45
PCO2 level
35 to 45 mm Hg
HCO3 level
22 to 27 mEg/L
PaO2 level
80 to 100 mm Hg