Introduction
Chronic Confusion is a topic that is generally new to man. For example, we as humans know that if a concussion happens, we know there will be an altered consciousness and there will be visible symptoms. Symptoms from headache to blackout to mental confusion. The thing with a concussion is that we know it will pass with time and rest, but what about the patient that has confusion everyday for years and is irreversible and is getting worse with time? What do we truly know about symptoms like these, where memory, orientation, and behavior is affected due to a head injury or nerve tangles in the brain? Hopefully this paper will shed some light on the definitions, knowledge, and pathophysiology on the new and concerning threat that …show more content…
This is where Dorothea Orem also developed the theory of self-care deficit, in where there are requisites that Orem identifies that must be met to prove that a patient can self-care and care for dependents. These requisites are also known as Activities of Daily Living (ADL) and are as follows:
1. the maintenance of sufficient intake of air, food and water.
2. provision of care associated with elimination process
3. a balance between activities and rest, as well as between solitude and social interaction
4. the prevention of hazards to human life and well-being
5. the promotion of human …show more content…
The reasons chronic confusion happen could be because of “neuron degeneration, compression, atherosclerosis, and trauma. Genetic predisposition is associated with this neurodegenerative diseases, including Alzheimer’s and Huntington diseases” (PATHO BOOK). With dementia though, there is not too much information of how and why this occurs, because it is a broad, but if we look more in depth at a sub category of dementia we can see how this certain disease happens and why. Alzheimer disease, a subcategory of dementia, is the leading cause of dementia. While there are three stages of Alzheimer’s disease, we will be focusing on the most common form, which is 90% of all Alzheimer’s patients, which is late-onset AD. What happens in Alzheimer’s patients with late-onset AD is that their may be an involvement of chromosome 19 with the apolipoprotein E gene-allele 4.
“Pathological alterations in the brain include formation of neuritic plaques containing a core of amyloid-beta protein, creation of neurofibrillary tangles, and degeneration of basal forebrain cholinergic neurons with loss of acetylcholine” (PATHO
Chronic Confusion is a topic that is generally new to man. For example, we as humans know that if a concussion happens, we know there will be an altered consciousness and there will be visible symptoms. Symptoms from headache to blackout to mental confusion. The thing with a concussion is that we know it will pass with time and rest, but what about the patient that has confusion everyday for years and is irreversible and is getting worse with time? What do we truly know about symptoms like these, where memory, orientation, and behavior is affected due to a head injury or nerve tangles in the brain? Hopefully this paper will shed some light on the definitions, knowledge, and pathophysiology on the new and concerning threat that …show more content…
This is where Dorothea Orem also developed the theory of self-care deficit, in where there are requisites that Orem identifies that must be met to prove that a patient can self-care and care for dependents. These requisites are also known as Activities of Daily Living (ADL) and are as follows:
1. the maintenance of sufficient intake of air, food and water.
2. provision of care associated with elimination process
3. a balance between activities and rest, as well as between solitude and social interaction
4. the prevention of hazards to human life and well-being
5. the promotion of human …show more content…
The reasons chronic confusion happen could be because of “neuron degeneration, compression, atherosclerosis, and trauma. Genetic predisposition is associated with this neurodegenerative diseases, including Alzheimer’s and Huntington diseases” (PATHO BOOK). With dementia though, there is not too much information of how and why this occurs, because it is a broad, but if we look more in depth at a sub category of dementia we can see how this certain disease happens and why. Alzheimer disease, a subcategory of dementia, is the leading cause of dementia. While there are three stages of Alzheimer’s disease, we will be focusing on the most common form, which is 90% of all Alzheimer’s patients, which is late-onset AD. What happens in Alzheimer’s patients with late-onset AD is that their may be an involvement of chromosome 19 with the apolipoprotein E gene-allele 4.
“Pathological alterations in the brain include formation of neuritic plaques containing a core of amyloid-beta protein, creation of neurofibrillary tangles, and degeneration of basal forebrain cholinergic neurons with loss of acetylcholine” (PATHO