Subtle differences were observed after comparing all body parts of mutant and a wild type D. Melanogaster. It was discovered that normal venation pattern was disrupted in mutant D. Melanogaster. The phenotype mutation (vein pattern) of D. Melanogaster was identified with a different degree of expressivity in mutant D. Melanogaster; this was achieved by differentiating the single line (longitudinal vein 5) on a wild-type wing which was disrupted. Additionally, few mutants had wings that did not fully extend to them, and also some mutants were unable to fly. Both male and female mutants did not show significant phenotypic differences. The adult flies and newly enclosed mutants …show more content…
After carefully conducting a thorough research via D. Melanogaster genome, five genes were found as the likely candidates for the disruption of normal venation pattern in D. Melanogaster mutants. Plexate (px) which is located at 2.107.2 is the first gene responsible for disruption of normal venation pattern observed in mutants. The biological processes the gene involved are not known. There is no phenotypic data available for this gene. However, it has seven reported alleles (Crozatier et al. 499). The second gene is Plexus (px) located at 2.100.5. This gene plays a role in biological process expounded with imaginal disc-derived wing vein morphogenesis. The gene has 61 reported alleles, and these alleles manifest themselves in wing vein, leg, wing, and eye (Kahsai et al. 2665). Therefore, it can lead to disruption of wing normal venation pattern which was observed in wild-type D. Melanogaster. The third gene is known as Blistered (bs) located at 2.107.3. Blistered (bs) is responsible for branching morphogenesis of an epithelial tube among other function. It has 62 reported alleles. The phenotypes of its alleles are manifested in the imaginal disc, one of the stages of development hence it is linked to