Tropomyosin Essay

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Figure 1 Myosin and actin work together to generate motion within skeletal muscles, however, a vital means of communication, uniting motor neurons and muscles, must be present prior to movement. Tropomyosin and troponin maintain control over the contractions produced by myosin and actin and provide an essential outlet for dictating instructions. Tropomyosin, a two-stranded, alpha-helical coil protein, is positioned laterally along actin filaments and is responsible for “blocking” myosin heads from attaching and traveling along the actin. Troponin, a complex consisting of three regulatory proteins, secures the tropomyosin into place on the actin and can dictate where it is positioned (1. Khan).
Troponin and tropomyosin are involved in an area
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However, though the process may be the same, the level of difficulty one may experience with different motions may vary greatly. This distinction occurs due to the varying degrees of “traffic” that neural pathways receive. For instance, a habitual action such as walking, is a relatively easy and mindless task for most individuals because the neural pathways used to walk are well traveled. This act of strengthening, weakening, and reorganizing neural connections is referred to as neuroplasticity, and is the process by which all learning takes place (MedicineNet). Immediately after one learns a new fact, skill or movement, the brain begins to rewire itself. However, learning was not always perceived to occur in this way. Until the early 1960’s, the belief that the adult brain is a hardwired, unchangeable, psychologically static organ remained firmly in place. Since then, new research findings have pointed to a new, more progressive theory, that the brain is malleable and plasticity occurs throughout life. Without neuroplasticity, learning, prioritizing relevant skills, and even recovering from injury would be nearly impossible for those in adulthood

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