According to a researcher, in Celiac disease, when someone consumes food containing the protein gluten, the small intestine reacts by preventing vitamins and minerals to absorb (Niewinski M. M., Advances in Celiac Disease and Gluten-Free Diet, 2008). The compounds within gluten called peptides are perceived as foreigners by antibodies within the body. Therefore, these antibodies treat them as an “invading” threat, and begin to destroy the intestinal tissue and its wall called the epithelium. As the combat progresses, targeted destruction extends to the organ’s exterior. Consequently, complications outside the small intestine ensue which serve as the reason why Celiac disease (CD) victims experience symptoms excluding intestinal involvement. Some of the symptoms that affect additional organs and tissue, include iron-deficiency anemia, chronic fatigue, irritable bowel, dyspepsia, infertility, pubertal delay, arthralgia, aphthous stomatitis, folate/zinc deficiency, (Niewinski M. M., Advances in Celiac Disease and Gluten-Free Diet, 2008). But, these symptoms occur in a limited group of people. Furthermore, those susceptible to develop the disease include first degree relatives of diagnosed patient, Down Syndrome patients, and those diagnosed with other autoimmune diseases (Niewinski M. M., Advances in Celiac Disease and Gluten-Free Diet, 2008). Interestingly, CD symptoms can occur many years prior to the actual diagnosis. On average, patients suffer from symptoms for eleven to twelve years before the autoimmune disease is identified. Therefore, tests must be taken often to confirm that the disease resides in the body. To accurately validate the condition, an intestinal biopsy must be performed. Dubbed the gold standard diagnosis, it has proven to reveal precise results (Niewinski M. M., Advances in Celiac Disease and Gluten-Free Diet,
According to a researcher, in Celiac disease, when someone consumes food containing the protein gluten, the small intestine reacts by preventing vitamins and minerals to absorb (Niewinski M. M., Advances in Celiac Disease and Gluten-Free Diet, 2008). The compounds within gluten called peptides are perceived as foreigners by antibodies within the body. Therefore, these antibodies treat them as an “invading” threat, and begin to destroy the intestinal tissue and its wall called the epithelium. As the combat progresses, targeted destruction extends to the organ’s exterior. Consequently, complications outside the small intestine ensue which serve as the reason why Celiac disease (CD) victims experience symptoms excluding intestinal involvement. Some of the symptoms that affect additional organs and tissue, include iron-deficiency anemia, chronic fatigue, irritable bowel, dyspepsia, infertility, pubertal delay, arthralgia, aphthous stomatitis, folate/zinc deficiency, (Niewinski M. M., Advances in Celiac Disease and Gluten-Free Diet, 2008). But, these symptoms occur in a limited group of people. Furthermore, those susceptible to develop the disease include first degree relatives of diagnosed patient, Down Syndrome patients, and those diagnosed with other autoimmune diseases (Niewinski M. M., Advances in Celiac Disease and Gluten-Free Diet, 2008). Interestingly, CD symptoms can occur many years prior to the actual diagnosis. On average, patients suffer from symptoms for eleven to twelve years before the autoimmune disease is identified. Therefore, tests must be taken often to confirm that the disease resides in the body. To accurately validate the condition, an intestinal biopsy must be performed. Dubbed the gold standard diagnosis, it has proven to reveal precise results (Niewinski M. M., Advances in Celiac Disease and Gluten-Free Diet,