Alzheimer’s Disease is one of many forms of dementia, and is the most common form (Orimaye 1). What Alzheimer’s Disease does to a person is that they start to have progressive cognitive decline like losing memory and degeneration of reasoning ability (Cai 1). To be diagnosed with Alzheimer’s Disease, a person has to go through extensive neurophysical examinations by using a lot of cognitive tests that have a series of questions and images.
There is a maximum score of thirty for the test, and if one gets a score of twenty seven or higher, they don’t have Alzheimer’s Disease or any other dementia related disease. The tests aren’t always completely accurate though, because the experience and skill of the physician plays a big role in how the …show more content…
The disease starts to destroy the nerve cells that control cognitive, speech, and language processes. Most of the time, patients with alzheimer’s disease start to notice vocal utterances at a younger age. Many tests have been given to alzheimer’s disease patients to find verbal problems. For example, there was a test given to sixty elderly patients with alzheimer’s disease and sixty one healthy elderly patients. The patients were given word tests, and it was in conclusion that the patients with alzheimer’s disease had a lot harder of a time with the word tests than the healthy patients. (Orimaye …show more content…
There was a clinical trial named the Alzheimer’s Disease Exercise Program Trial. It was a twenty six week trial where people with early stage Alzheimer’s Disease did aerobic exercises and a non-aerobic stretching and toning program. It was concluded that the people who participated in this trial saw a decline in the progression of their Alzheimer’s Disease (Morris 3).
Another non-pharmaceutical treatment for Alzheimer’s Disease is the changing of the patient’s lifestyle. The changing of the lifestyle includes physical activity, mental challenges, energy restriction, and socialization (Precoma-Mendiola 8).
There are three mechanisms used in physical activity that treat Alzheimer’s Disease. The first one is the release of neurotrophic factors from neurons in synaptic activity, which stimulates neurogenesis and synaptic neural plasticity. The second is the reduction of free radicals in the hippocampus as well as the increase in superoxide dismutase and endothelial nitric oxide synthase. The third mechanism is that exercise releases peripheral signals that support the demand of active neuronal networks (Precoma-Mendiola
There is a maximum score of thirty for the test, and if one gets a score of twenty seven or higher, they don’t have Alzheimer’s Disease or any other dementia related disease. The tests aren’t always completely accurate though, because the experience and skill of the physician plays a big role in how the …show more content…
The disease starts to destroy the nerve cells that control cognitive, speech, and language processes. Most of the time, patients with alzheimer’s disease start to notice vocal utterances at a younger age. Many tests have been given to alzheimer’s disease patients to find verbal problems. For example, there was a test given to sixty elderly patients with alzheimer’s disease and sixty one healthy elderly patients. The patients were given word tests, and it was in conclusion that the patients with alzheimer’s disease had a lot harder of a time with the word tests than the healthy patients. (Orimaye …show more content…
There was a clinical trial named the Alzheimer’s Disease Exercise Program Trial. It was a twenty six week trial where people with early stage Alzheimer’s Disease did aerobic exercises and a non-aerobic stretching and toning program. It was concluded that the people who participated in this trial saw a decline in the progression of their Alzheimer’s Disease (Morris 3).
Another non-pharmaceutical treatment for Alzheimer’s Disease is the changing of the patient’s lifestyle. The changing of the lifestyle includes physical activity, mental challenges, energy restriction, and socialization (Precoma-Mendiola 8).
There are three mechanisms used in physical activity that treat Alzheimer’s Disease. The first one is the release of neurotrophic factors from neurons in synaptic activity, which stimulates neurogenesis and synaptic neural plasticity. The second is the reduction of free radicals in the hippocampus as well as the increase in superoxide dismutase and endothelial nitric oxide synthase. The third mechanism is that exercise releases peripheral signals that support the demand of active neuronal networks (Precoma-Mendiola