All my life, I’ve always wondered, why? Why do I stammer? Is it something wrong with me? I know that I was born that way because unlike many people, stammering didn’t only occur when I was stressed or in an unconformable situation. I stuttered every time I tried to speak. No matter how hard I tried to control my speech flow, I couldn’t help it. It was natural. My relatives tried to help me, to cure me fof my speech disorder but they failed. They didn’t have patience with me. They believed that I was the one to blame for my speech disfluency. My parents told me that I should try hard not to stammer, I should put more efforts into avoiding stammering when I speak. I wish it was so easy. Slowly, I started to believe them, blaming myself for that
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Indeed, it explains how genetic and environmental influences shape certain behaviors. In this case, was I born with this speech disorder or did I develop it over time? Many studies agree that stuttering usually starts in early childhood. Children are more likely to recover from stuttering than teenagers/adults. As children reach adolescence the likelihood of such a recovery decreases (Yairi and Ambrose, 1999; Howell et al., 2008). According to Christina Kell (2009), stuttering is a disorder caused by anomalies in the left inferior frontal area of the brain. She believed that stuttering can appear in early childhood and disappear in the same time frame or the recovery can occur after many years of …show more content…
Researchers tested the writing ability of the subjects using the Boston Naming Test (BNT). The result of speech articulation task didn’t show any significant differences between the 3 groups. However, the results of oral reading tests differed significantly between the acquired stuttering group and the 2 other groups. The results of oral motor tasks didn’t show any differences between the 3 groups. Researchers also analyzed 25 regions of the brain related to speech motor control. They found out that of the 10 acquired stuttering subjects, 5 suffered from unilateral right lesions, 1 suffered from right and left lesions and 4 suffered from left sided lesions. They came to the conclusion that lesions in both the right and left hemisphere of the brain caused acquired stuttering. Furthermore, they noted that there was not a significant difference in the total volume of the lesion between the acquired stuttering group. Nevertheless, the frequency of lesions was higher in the acquired stuttering group than in the head injured group except in the supplementary motor area where head injured group had a higher frequency of
Indeed, it explains how genetic and environmental influences shape certain behaviors. In this case, was I born with this speech disorder or did I develop it over time? Many studies agree that stuttering usually starts in early childhood. Children are more likely to recover from stuttering than teenagers/adults. As children reach adolescence the likelihood of such a recovery decreases (Yairi and Ambrose, 1999; Howell et al., 2008). According to Christina Kell (2009), stuttering is a disorder caused by anomalies in the left inferior frontal area of the brain. She believed that stuttering can appear in early childhood and disappear in the same time frame or the recovery can occur after many years of …show more content…
Researchers tested the writing ability of the subjects using the Boston Naming Test (BNT). The result of speech articulation task didn’t show any significant differences between the 3 groups. However, the results of oral reading tests differed significantly between the acquired stuttering group and the 2 other groups. The results of oral motor tasks didn’t show any differences between the 3 groups. Researchers also analyzed 25 regions of the brain related to speech motor control. They found out that of the 10 acquired stuttering subjects, 5 suffered from unilateral right lesions, 1 suffered from right and left lesions and 4 suffered from left sided lesions. They came to the conclusion that lesions in both the right and left hemisphere of the brain caused acquired stuttering. Furthermore, they noted that there was not a significant difference in the total volume of the lesion between the acquired stuttering group. Nevertheless, the frequency of lesions was higher in the acquired stuttering group than in the head injured group except in the supplementary motor area where head injured group had a higher frequency of