Upon examination, it was found that “six-month-old infants with postnatal tobacco exposure had less focused attention, lowered reactivity to basic sensory stimulation, and more distractibility compared to non-exposed infants (Wiebe, unpublished data), and the author concluded that PTE leads to deficits in information processing and self-regulation”( Cornelius and Day, 2009). It is evident that, even a miniscule amount of exposure can seriously affect the nervous system of a child. One type of essential neurotransmitter responsible for learning is dopamine. Nicotine and cotinine exposure causes change to dopamine’s signaling function by shortening the neurite length preventing proper neuro signaling when performing an activity such as reading. Even more, due to improper signaling, a child’s working memory is also affected. Working memory is part of the short term memory that helps with skills such as reading and writing. When dopamine’s function is altered these changes are permanent be it genetic or prenatal exposure. The outcome results in poor language development as well as affecting other neurobehavioral domains (Eicher et.al, 2015). These evidence support that when a child is exposed prenatally to nicotine and cotinine, there is an overall effect on the brains neurochemistry. Thus, society needs to become better aware of the consequences, because children are the future innovators and the next healthcare workers, so they need to be healthy and outlive their parents. Studies show that parents are more likely to outlive their children. In order for children to become great individuals they must be capable of comprehending and learning. However, if a majority of them end up being affected by passive smoking it impacts the
Upon examination, it was found that “six-month-old infants with postnatal tobacco exposure had less focused attention, lowered reactivity to basic sensory stimulation, and more distractibility compared to non-exposed infants (Wiebe, unpublished data), and the author concluded that PTE leads to deficits in information processing and self-regulation”( Cornelius and Day, 2009). It is evident that, even a miniscule amount of exposure can seriously affect the nervous system of a child. One type of essential neurotransmitter responsible for learning is dopamine. Nicotine and cotinine exposure causes change to dopamine’s signaling function by shortening the neurite length preventing proper neuro signaling when performing an activity such as reading. Even more, due to improper signaling, a child’s working memory is also affected. Working memory is part of the short term memory that helps with skills such as reading and writing. When dopamine’s function is altered these changes are permanent be it genetic or prenatal exposure. The outcome results in poor language development as well as affecting other neurobehavioral domains (Eicher et.al, 2015). These evidence support that when a child is exposed prenatally to nicotine and cotinine, there is an overall effect on the brains neurochemistry. Thus, society needs to become better aware of the consequences, because children are the future innovators and the next healthcare workers, so they need to be healthy and outlive their parents. Studies show that parents are more likely to outlive their children. In order for children to become great individuals they must be capable of comprehending and learning. However, if a majority of them end up being affected by passive smoking it impacts the