Since cardiac output is calculated by multiplying SV by HR, a decrease in SV will in turn be can be associated with a decrease in CO. Thus, why CO had the same hypothesis, which was that SV and CO would decrease upon tilt and increase rapidly afterwards. However, SV did not follow the expected trend and nor did it follow the same trend that CO did. Instead of continuing to fall like SV (Figure 4), CO increased at 2-minute post-tilt (Figure 5). The increase in CO is causes by the large increase in HR (Figure 3), which climbed to 95 BPM, from a rate of 72BPM at tilt. The largest increase occurred at 30 second post-tilt, a 16. 67% …show more content…
TPR was hypothesized to decrease as having blood pool and needing to be returned, the body would want less resistance to make this less strenuous on the body. TPR is the resistance the vessels push back on the blood trying to flow through (Sherwood, 2010, p.358). A study by Goodman and LeSage (2002) illustrated exactly that, TPR decreasing upon being tilt. However, the results indicate that TPR increased during tilt (Figure 6). Since TPR is calculated by MAP/CO, and there being issues with CO, it is suspected a compounding issue when dealing with an indirect measurement is occurring. If, there were not the issues with CO, SV and HR and a similar result was seen, then alternatives could be possible