Rickets Case Study
Fewer than a hundred cases are reported of those who suffer with Hypophosphatmeic (“Hereditary 1”). This type of rickets is the most common form that is genetic (“Hereditary” 2015). It is caused by a genetic main x-linked defect for the kidneys to maintain the amount of phosphate released into the urine. By the time the patient is a year old, there will be obvious signs and symptoms of hypohosphatemic (“Rickets Symptoms” 2). This condition reveals as a range of abnormalities such as: short stature, bowing legs, and bone pain. Tooth discoloration, bleeding gums, sensitivity, and loose teeth are some of the effects in the oral cavity due to hypophosphatemic (“Symptoms of Hypohosphatemic” 1). Hypophosphatemic is related to nutrition because with treatment one must consume nutritional supplements of phosphate and calcitriol which are activated forms of vitamin D. Treatment with calcitriol is started at 15 to 20 kg, dosage is soon increased over a number of months to 30 to 60 kg. Phosphate slats are also prescribed at 0.5 to 1g/24 in younger children and one to four every four hours at least five times a day in older children