Pathogen infection leads to response induction as well as a change in antioxidant activity, chlorophyll content, chlorophyll fluorescence parameter and proteome of the host plant. (Swarbrick et al., 2006).Investigation of plant-pathogen interaction shows pathogens aim to overcome host defense responses while plants employ a battery of responses to limit pathogen growth and disease development. Consequently, plant induces defense mechanism which demands a reallocation of energy towards the defense response and this apportioning of energy for defense processes to necessitate the modification of primary metabolism (Bolton, 2009). Some observation showed that infection of citrus canker bacteria and other pathogen infection decreased the expression of sugar-regulated photosynthetic genes such as Rubisco and ATP synthase and also increase NADH dehydrogenase support the case for a reduction in photosynthetic
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The effect on photosynthesis can be analyzed by monitoring in vivo chlorophyll fluorescence. This method is based on measuring the fluorescence of chlorophyll in dark-adapted plants and after saturating light pulses(Schreiber et al., 1986). This fluorescence is a very sensitive marker for the efficiency of photosynthesis and also responds to the changes in energy conversion at photosystem II reaction centers and is also sensitive to any limitations in the dark enzymatic steps of the complex process of
The effect on photosynthesis can be analyzed by monitoring in vivo chlorophyll fluorescence. This method is based on measuring the fluorescence of chlorophyll in dark-adapted plants and after saturating light pulses(Schreiber et al., 1986). This fluorescence is a very sensitive marker for the efficiency of photosynthesis and also responds to the changes in energy conversion at photosystem II reaction centers and is also sensitive to any limitations in the dark enzymatic steps of the complex process of