Myocardial Ischemia Physiology

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Main Question Post: Week 4: Cardiovascular Disorders

Myocardial Ischemia and Acute Coronary Syndrome
Cardiovascular disease (CVD) persists as the principle source of death amongst the men and women globally, and has claimed further breathes yearly in the United States (U.S) than “cancer, chronic lung disease, and accidents” collectively (Dunlay, & Roger, 2012). Myocardial ischemia and acute coronary syndrome originate with the coronary artery disease. According to Huether and McCance (2012), coronary artery disease (CAD) can reduce the myocardial bloodstocks until scarcity ruins myocardial metabolism sufficiently enough to instigate ischemia, an indigenous state whereby the cells are provisional denied of blood supply. A persistent ischemia or an absolute blockage of the coronary artery triggers the acute coronary syndrome. This continual obstruction advances into an infarction generally branded as a heart attack; the hallmark of averting a heart attack is contingent on avoiding the inception of coronary artery disease (Huether & McCance, 2012; Dunlay, & Roger, 2012).

The Pathophysiology of Myocardial Ischemia

The principle function of the coronary arteries is to resource blood flow to the myocardium as reckoned needed by the workload,
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These inexplicit symptoms might relate to dispersed and microvascular disease, the myocardial infarction symptoms for women varies. According to Dunlay, & Roger (2012), women have a tendency of grander high cholesterol and lessened physical activity level, which is a hallmark for cardiovascular disease risk and the risk element upsurges post menopause related to the reduced estrogen

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