Numerous studies conducted on the brains of deceased schizophrenia patients point out a high number of dopamine receptors (Seeman 2007). For that reason, researchers conclude that the previously mentioned result in hallucinations and paranoia found in patients with Schizophrenia. Moreover, the use of illegal substances, such as cocaine and amphetamines, amplify dopamine levels, enhancing the symptoms related to the disorder (Seeman 2007). This disease affects regions of the brain, specifically the thalamus and amygdala. The thalamus transmits the filtered sensory signals to the brain 's cortex, which handles alertness, consciousness, and sleep. In spite of this, a person who suffers from hallucinations endures irrepressible brain activity in the thalamus because of the disorder (Epstein et al., 1998). The amygdala integrates one 's motivation and emotional behavior. Subsequently, a person who suffers from paranoia has excessive brain activity in the amygdala (Job et al., 2006). Both affected regions of the brain contribute to the disorder, along with the individual’s prenatal
Numerous studies conducted on the brains of deceased schizophrenia patients point out a high number of dopamine receptors (Seeman 2007). For that reason, researchers conclude that the previously mentioned result in hallucinations and paranoia found in patients with Schizophrenia. Moreover, the use of illegal substances, such as cocaine and amphetamines, amplify dopamine levels, enhancing the symptoms related to the disorder (Seeman 2007). This disease affects regions of the brain, specifically the thalamus and amygdala. The thalamus transmits the filtered sensory signals to the brain 's cortex, which handles alertness, consciousness, and sleep. In spite of this, a person who suffers from hallucinations endures irrepressible brain activity in the thalamus because of the disorder (Epstein et al., 1998). The amygdala integrates one 's motivation and emotional behavior. Subsequently, a person who suffers from paranoia has excessive brain activity in the amygdala (Job et al., 2006). Both affected regions of the brain contribute to the disorder, along with the individual’s prenatal