When Mr Jones was first admitted, there were several concerning cues that made him an at risk patient. Even before observations were taken Mr Jones’s age, weight and intense chest pain clearly indicated his need for immediate action to evaluation his condition because of the risk of heart complications. After observations Mr Jones had a total Q-ADDS score of 3 and a pain score of 7/10, most concerning was his O2 saturation at 92% and his severe crushing pain in his chest which had been going on for an hour and a half and had only been slightly relieved by 2 doses of GTN. There is evidence that abnormalities in the physical objective (BP, O2 sat) and subjective (pain score) observations are indicators of possible critical events (Australian Commission on Safety and Quality in Health Care, 2009).
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For his work he spends a lot of time sitting through travel and works from 7am to 8pm not giving him much time to rest, exorcise or east healthily during this time period. 18 months ago he was diagnosed with stable angina which has been progressively getting worse, in the past 6 months he has had daily chest pain leading up to his attack today. The most significant cue that Mr Jones is at risk of a critical event at 2130hrs is his 12 lead ECG showing a ST elevation myocardial infarction and pulmonary oedema. A myocardial infarction is highly life threatening and cause ischemia and necrosis of the heart tissue which will lead to cardiogenic shock if not treated at the right time (LeMone et al., 2014). Death from MI often occurs within three hours of the presentation of symptoms; therefor Mr Jones risk for a critical event is quite high unless he his appropriately stabilised and has the right cues to make him a patient at risk with need for immediate treatment due to his high
For his work he spends a lot of time sitting through travel and works from 7am to 8pm not giving him much time to rest, exorcise or east healthily during this time period. 18 months ago he was diagnosed with stable angina which has been progressively getting worse, in the past 6 months he has had daily chest pain leading up to his attack today. The most significant cue that Mr Jones is at risk of a critical event at 2130hrs is his 12 lead ECG showing a ST elevation myocardial infarction and pulmonary oedema. A myocardial infarction is highly life threatening and cause ischemia and necrosis of the heart tissue which will lead to cardiogenic shock if not treated at the right time (LeMone et al., 2014). Death from MI often occurs within three hours of the presentation of symptoms; therefor Mr Jones risk for a critical event is quite high unless he his appropriately stabilised and has the right cues to make him a patient at risk with need for immediate treatment due to his high