Intrinsic Cardiac Activity: The first peak of the electrocardiogram graph corresponded to the atrial contraction of the heart. The second peak of the electrocardiogram graph corresponded to the ventricular contraction of the heart. The third peak observed corresponded to the relaxation of the heart.
Frank-Starling Mechanism: There was no significant trend observed in ventricular contractile force in response to increasing imposed length of the heart (Fig 1a). A maximum ventricular contractile force of 0.0032 + 0.001039 N (N=3) was generated at an imposed length of 4 mm. A minimum ventricular contractile force of 0.00137 + 0.0000577 N (N=3) was generated at an imposed length of 0 mm.
There was no significant trend observed in heart rate in response to increasing imposed length of the heart (Fig 1b). A minimum heart rate of 31.7 + 0.577 beats/min (N=3) was generated at an imposed length of 7 mm. A maximum heart rate of 36.7 + 0.577 beats/min (N=3) was generated at an imposed length of 6 mm.
Autonomic Control of the Heart: A significant decrease in …show more content…
2b). A minimum heart rate of 33.7 + 0.577 beats/min (N=3) was observed with no additions. After the addition of ephinephrine, a significant increase in heart rate from 33.7 + 0.577 beats/min (N=3) to 37.7 + 0.577 beats/min (N=3) was observed. After the addition of eserine, no significant difference in heart rate was observed relative to the baseline value. Following the addition of atropine, a significant increase in heart rate from 33.7 + 0.577 beats/min (N=3) to 37.7 + 0.577 beats/min (N=3) was observed. No significant difference in heart rate was observed after the addition of acetylcholine relative to the baseline value. The maximum heart rate of 37.7 + 0.577 beats/min (N=3) was both observed after the addition of ephinephrine and
Frank-Starling Mechanism: There was no significant trend observed in ventricular contractile force in response to increasing imposed length of the heart (Fig 1a). A maximum ventricular contractile force of 0.0032 + 0.001039 N (N=3) was generated at an imposed length of 4 mm. A minimum ventricular contractile force of 0.00137 + 0.0000577 N (N=3) was generated at an imposed length of 0 mm.
There was no significant trend observed in heart rate in response to increasing imposed length of the heart (Fig 1b). A minimum heart rate of 31.7 + 0.577 beats/min (N=3) was generated at an imposed length of 7 mm. A maximum heart rate of 36.7 + 0.577 beats/min (N=3) was generated at an imposed length of 6 mm.
Autonomic Control of the Heart: A significant decrease in …show more content…
2b). A minimum heart rate of 33.7 + 0.577 beats/min (N=3) was observed with no additions. After the addition of ephinephrine, a significant increase in heart rate from 33.7 + 0.577 beats/min (N=3) to 37.7 + 0.577 beats/min (N=3) was observed. After the addition of eserine, no significant difference in heart rate was observed relative to the baseline value. Following the addition of atropine, a significant increase in heart rate from 33.7 + 0.577 beats/min (N=3) to 37.7 + 0.577 beats/min (N=3) was observed. No significant difference in heart rate was observed after the addition of acetylcholine relative to the baseline value. The maximum heart rate of 37.7 + 0.577 beats/min (N=3) was both observed after the addition of ephinephrine and