Henry Williams is a 74 year old African American male. Mr. Williams was admitted to the hospital with an acute exacerbation of chronic obstructive pulmonary disease (COPD). Mr. Williams has a past medical history of COPD, cardiovascular disease (CVD), asthma, hearing loss with use of hearing aids, hypertension (HTN), and hyperlipidemia. He smoked one pack per day for 50 years. He quit smoking about 6 years ago. Mr. Williams was sent to the emergency department (ED) by his primary care doctor, he suggested Mr. Williams go since he was unable to catch his breath.
Mr. Williams was brought to the ED by his neighbor. While here, Mr. Williams was more concerned about his wife, who has problems with her memory. Mr. Williams’ daughter, Betty, …show more content…
This type of hypertension has also been essential or idiopathic hypertension. Secondary hypertension is high BP secondary to a cause. These causes include narrowing of the renal artieries or renal artery stensois, renal disease hyperaldosteronism, medications, pregnancy, and coarctation of the aorta. The Joint National Committee on Prevention, Dectection, Evaluation, and Treatment of High BP of the National Institute of Health defines a BP of less than 120/80 mm Hg as normal, BP 120-139/80-89 mm Hg as prehypertension and BP of 140/90 mm Hg or higher as hypertension. Hypertension is further classified as stage 1 or stage 2. Stage 1 hypertension occurs when the systolic pressure is 140-159 mm Hg or greater and/or the diastolic pressure is 90-99 mm Hg or greater. Stage 2 hypertension occurs when the systolic pressure is 160 mm Hg or higher, or the diastolic pressure is 100 mm Hg or higher. If the systolic and diastolic pressure are in separate categories, the higher classification is …show more content…
These substances created blockages and narrow the coronary vessels in a way that reduces blood flow. Atherosclerosis involves a repetitious inflammatory response to injury to the artery wall. Atherosclerosis begins as fatty streaks of lipids that are deposited in the intima of the arterial wall. These lesions commonly begin early in life. Not all fatty streaks later develop into more advance lesions. Genetics and environmental factors influence the progression of these lesions. The continued development of atherosclerosis involves an inflammatory response, which beings with injury to the vascular endothelium. The injury may be initiated by smoking, hypertension, and other
Mr. Williams was brought to the ED by his neighbor. While here, Mr. Williams was more concerned about his wife, who has problems with her memory. Mr. Williams’ daughter, Betty, …show more content…
This type of hypertension has also been essential or idiopathic hypertension. Secondary hypertension is high BP secondary to a cause. These causes include narrowing of the renal artieries or renal artery stensois, renal disease hyperaldosteronism, medications, pregnancy, and coarctation of the aorta. The Joint National Committee on Prevention, Dectection, Evaluation, and Treatment of High BP of the National Institute of Health defines a BP of less than 120/80 mm Hg as normal, BP 120-139/80-89 mm Hg as prehypertension and BP of 140/90 mm Hg or higher as hypertension. Hypertension is further classified as stage 1 or stage 2. Stage 1 hypertension occurs when the systolic pressure is 140-159 mm Hg or greater and/or the diastolic pressure is 90-99 mm Hg or greater. Stage 2 hypertension occurs when the systolic pressure is 160 mm Hg or higher, or the diastolic pressure is 100 mm Hg or higher. If the systolic and diastolic pressure are in separate categories, the higher classification is …show more content…
These substances created blockages and narrow the coronary vessels in a way that reduces blood flow. Atherosclerosis involves a repetitious inflammatory response to injury to the artery wall. Atherosclerosis begins as fatty streaks of lipids that are deposited in the intima of the arterial wall. These lesions commonly begin early in life. Not all fatty streaks later develop into more advance lesions. Genetics and environmental factors influence the progression of these lesions. The continued development of atherosclerosis involves an inflammatory response, which beings with injury to the vascular endothelium. The injury may be initiated by smoking, hypertension, and other