Causes Of Left Heart Failure

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Heart failure is a common killer of both men and women in the United States every year. It is a very serious issue that plagues this country and other developed countries like it. Heart failure is a syndrome with many risk factors, such as, age, obesity, congenital heart diseases, and many others. A client is considered to be in heart failure, when their heart is unable to profuse tissues in the body with oxygenated and nutrient rich blood for tissue to sufficiently function. A client when diagnosed with the disease is considered either in systolic or diastolic heart failure (McCance & Huether, 2014, p.1175). Left heart failure, also known as congestive heart failure, is a decrease in the cardiac output. This means less blood than is …show more content…
This system has direct control over the blood pressure, the volume of blood, and the fluid and electrolyte balance in the body. Angiotensin II controls vasoconstriction in the circulatory system, which increases blood pressure and can cause hypertension (Burchum and Rosenthal, 2016,p.471-473). Aldosterone is what controls the fluid and electrolyte balance in the body. It also is released from the adrenal cortex and activates retention of sodium, which increases the fluid volume, which consequently increases the blood pressure (Dhillon S., 2013, p.1452). During heart failure there is a decrease in the profusion to peripheral tissues. The RAAS thinks it is helping by increasing blood pressure, when actually it is making the situation …show more content…
ACE inhibiters are one of the only drugs that have actually been proven to prolong life and improve the status of clients affected by heart failure. ACE inhibitors actually prevent production of angiotensin II which when in over production is the major factors in both types of heart failure (Burchum and Rosenthal, 2016,p.520).
Cardiac remodeling is a very important and unique job of ACE inhibitors. This characteristic helps prolong life and decrease morbidity for patients with heart failure. The ACE inhibitor actually undoes what heart failure has already done. It diminishes the hypertrophied myocardium and fibrous tissue. Along with allowing the heart to work more rhythmically, it lowers the amount of oxygen required to nourish the heart and also increases contractility (O’Donovan, 2014,

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