Conclusion
My hypothesis stated that Digitalis and Furosemide will both work upon the patient’s left side heart failure. However, the experiment concluded that only Digitalis was effective upon the patient’s condition. Administration of Furosemide led to a relatively quick death of the patient. I did not expect the patient to die while being administered Furosemide. In fact, I had hypothesized that while both drugs would work effectively upon the patient, that the Furosemide would work at a faster rate.
If I were to make an alternative hypothesis for the experiment, I would still say that Digitalis would accomplish its goal of helping the patient. But, I would hypothesize that Furosemide would help the symptoms, but not treat the actual …show more content…
In the untreated right side congestive heart failure experiment, the aldosterone levels briefly spiked within the first few hours, before it gradually declined.
In the untreated left side congestive heart failure, aldosterone levels rose with the first few hours, before starting to decline again. They raised and declined at a faster rate than the untreated right side congestive heart failure.
In the patient being treated with Digitalis for his left sided congestive heart failure, the levels started out mildly higher on the second day, started gradually declining, before slowly rising again during the fifth day. Digitalis helps a malfunction heart beat more efficiently, therefore helping the fluid move along within the body and aldosterone is not needed as much ("Digoxin", …show more content…
As the Digitalis helps the heart pump more efficiently, the the extra fluid within the cells is slowly lost, and the GFR becomes more efficient.
Lastly, the patient receiving Furosemide treatment starts at 125.9, rising sharply to 130.3, before succumbing to death. Because the Furosemide only treats water retention, the heart is still weakened, and therefore still not pumping as efficiently as it could be, leaving Furosemide ineffective ("Furosemide", 2014).
As Angiotensin II promotes sodium reabsorption, water follows the sodium, promoting water retention (Goodman, 1999). Due to the retention of water within the body, less leaves the body through the bladder. This causes a higher solute content within the available urine and therefore higher osmolarity.
ADH is normally used within the body as a mechanism to regulate extracellular fluid, and according to Goodman, “presence of ADH will cause reabsorption of water and a more concentrated urine,” (1999). “It may be that sympathetic and [RAAS] override the volume and low pressure cardiovascular receptors…and cause an increase in AVP secretion” (Klabunde,
My hypothesis stated that Digitalis and Furosemide will both work upon the patient’s left side heart failure. However, the experiment concluded that only Digitalis was effective upon the patient’s condition. Administration of Furosemide led to a relatively quick death of the patient. I did not expect the patient to die while being administered Furosemide. In fact, I had hypothesized that while both drugs would work effectively upon the patient, that the Furosemide would work at a faster rate.
If I were to make an alternative hypothesis for the experiment, I would still say that Digitalis would accomplish its goal of helping the patient. But, I would hypothesize that Furosemide would help the symptoms, but not treat the actual …show more content…
In the untreated right side congestive heart failure experiment, the aldosterone levels briefly spiked within the first few hours, before it gradually declined.
In the untreated left side congestive heart failure, aldosterone levels rose with the first few hours, before starting to decline again. They raised and declined at a faster rate than the untreated right side congestive heart failure.
In the patient being treated with Digitalis for his left sided congestive heart failure, the levels started out mildly higher on the second day, started gradually declining, before slowly rising again during the fifth day. Digitalis helps a malfunction heart beat more efficiently, therefore helping the fluid move along within the body and aldosterone is not needed as much ("Digoxin", …show more content…
As the Digitalis helps the heart pump more efficiently, the the extra fluid within the cells is slowly lost, and the GFR becomes more efficient.
Lastly, the patient receiving Furosemide treatment starts at 125.9, rising sharply to 130.3, before succumbing to death. Because the Furosemide only treats water retention, the heart is still weakened, and therefore still not pumping as efficiently as it could be, leaving Furosemide ineffective ("Furosemide", 2014).
As Angiotensin II promotes sodium reabsorption, water follows the sodium, promoting water retention (Goodman, 1999). Due to the retention of water within the body, less leaves the body through the bladder. This causes a higher solute content within the available urine and therefore higher osmolarity.
ADH is normally used within the body as a mechanism to regulate extracellular fluid, and according to Goodman, “presence of ADH will cause reabsorption of water and a more concentrated urine,” (1999). “It may be that sympathetic and [RAAS] override the volume and low pressure cardiovascular receptors…and cause an increase in AVP secretion” (Klabunde,