Early vitamin K deficiency bleeding occurs in 1 and 60 newborns born in the first 0-24 hours after birth. Mothers who used certain medications during their pregnancy makes it a higher risk for early deficiency bleeding also making it more severe. Late vitamin K deficiency bleeding in 2-12 weeks after birth occurs in 1 in 14,000 infants. 30%-60% of these infants have bleeding within the brain and tend to occur in breastfed only babies who have not received the vitamin K shot. All pregnant women should be required to give their newborn the vitamin k shot if they plan to breast feed.
What is Vitamin K?
Vitamin K is a substance that our body needs to clot and to stop bleeding. Vitamin K is found in green leafy vegetables and oils, such as …show more content…
This is because a lack of vitamin reaching the fetus across the placenta, the low level of vitamin K in breast milk, and low colonic bacterial synthesis. Vitamin K deficiency is uncommon in otherwise healthy adults. Vitamin K deficiency bleeding or VKDB occurs when babies cannot stop bleeding because their blood does not have enough vitamin K to form a clot. The bleeding can occur anywhere on the inside or outside of the body. When the bleeding is on the inside the body it is more difficult to notice. Infants who do not receive the vitamin K deficiency shot at birth can develop vitamin K deficiency bleeding at any time up to six months of age. Commonly, a baby with VKDB will bleed into his or her intestines, or into the brain, which can lead to brain damage or even death. Vitamin K deficiency bleeding (VKDB) is now the preferred term for a hemorrhage disease of the newborn (HDN). There are three types of VKDB based on the age of the baby when the bleeding problems start. The first type is early VKDB presented within the first 24 hours of birth. Classic VKDB presented day 1 and day 7 of life is the second type of vitamin K deficiency bleeding. Lastly presented between week two and week twelve of life is late VKDB. Late Vitamin K deficiency bleeding can result in significant mortality due to intracranial hemorrhage and as resulted in most …show more content…
As like most other diseases when recognized early it is most likely to have a good prognosis and probability of being treated appropriately. However, severe bleeding can occur if left untreated. The medical therapy of VKD depends on the severity of the underlying disease state. The most effective approach to correcting the deficiency also depends on the nature of the bleeding and the risk of including a local hematoma at the VK injection site. All forms of VKDB are now far less common due to understanding of the etiology. Routine antenatal screening of all mothers has allowed for the early identification of babies who may be at risk of early VKDB. The greatest reduction has resulted from the routine administration of vitamin K in all newborn babies, usually at birth. This is given in the form of either an IM injection or a series of oral supplements, and as a consequence, VKDB is now rarely seen in the UK and other countries where this policy has been adopted. The IM routine is preferred. Classic VKDB is prevented by IM or oral administration of 1 mg vitamin K. In exclusively breast-fed infants, single IM administration at birth is also effective in preventing late VKDB but single oral administration is not. If given orally, prophylaxis should be continued by either weekly administration of 1 mg until 12 weeks or repeating 2 mg at weeks 1 and 4. The only infants not fully protected in this way are those with yet
What is Vitamin K?
Vitamin K is a substance that our body needs to clot and to stop bleeding. Vitamin K is found in green leafy vegetables and oils, such as …show more content…
This is because a lack of vitamin reaching the fetus across the placenta, the low level of vitamin K in breast milk, and low colonic bacterial synthesis. Vitamin K deficiency is uncommon in otherwise healthy adults. Vitamin K deficiency bleeding or VKDB occurs when babies cannot stop bleeding because their blood does not have enough vitamin K to form a clot. The bleeding can occur anywhere on the inside or outside of the body. When the bleeding is on the inside the body it is more difficult to notice. Infants who do not receive the vitamin K deficiency shot at birth can develop vitamin K deficiency bleeding at any time up to six months of age. Commonly, a baby with VKDB will bleed into his or her intestines, or into the brain, which can lead to brain damage or even death. Vitamin K deficiency bleeding (VKDB) is now the preferred term for a hemorrhage disease of the newborn (HDN). There are three types of VKDB based on the age of the baby when the bleeding problems start. The first type is early VKDB presented within the first 24 hours of birth. Classic VKDB presented day 1 and day 7 of life is the second type of vitamin K deficiency bleeding. Lastly presented between week two and week twelve of life is late VKDB. Late Vitamin K deficiency bleeding can result in significant mortality due to intracranial hemorrhage and as resulted in most …show more content…
As like most other diseases when recognized early it is most likely to have a good prognosis and probability of being treated appropriately. However, severe bleeding can occur if left untreated. The medical therapy of VKD depends on the severity of the underlying disease state. The most effective approach to correcting the deficiency also depends on the nature of the bleeding and the risk of including a local hematoma at the VK injection site. All forms of VKDB are now far less common due to understanding of the etiology. Routine antenatal screening of all mothers has allowed for the early identification of babies who may be at risk of early VKDB. The greatest reduction has resulted from the routine administration of vitamin K in all newborn babies, usually at birth. This is given in the form of either an IM injection or a series of oral supplements, and as a consequence, VKDB is now rarely seen in the UK and other countries where this policy has been adopted. The IM routine is preferred. Classic VKDB is prevented by IM or oral administration of 1 mg vitamin K. In exclusively breast-fed infants, single IM administration at birth is also effective in preventing late VKDB but single oral administration is not. If given orally, prophylaxis should be continued by either weekly administration of 1 mg until 12 weeks or repeating 2 mg at weeks 1 and 4. The only infants not fully protected in this way are those with yet